ResearcherOregon Health & Science UniversityActive in the last 2 years
Isabel A. English
Matched on ORCID iD 0000-0002-0566-9585; OpenAlex author ID A5067240272. Sources: OpenAlex.
Their work
- Papers
- 6
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 34
2020: 1 paper · 2021: none · 2022: none · 2023: none · 2024: 1 paper · 2025: 4 papers
Papers 6
- Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in pancreatic cancer. Co-author DOI 10.1038/s43018-024-00881-3↗
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model Co-author DOI 10.1101/2025.07.14.664767↗
- The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3437↗
- A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK pathway Co-author DOI 10.64898/2025.12.12.693851↗
- KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. Co-author DOI 10.1038/s41388-024-03196-w↗
- Deconstructing Pancreatic Adenocarcinoma by Targeting the Conductor, MYC. Co-author DOI 10.1158/2159-8290.cd-20-0046↗
Closest collaborators
The 25 strongest of 34 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rosalie C. Sears · 6 shared papers, 2020–2025 · strength 1.06
- Paper Deconstructing Pancreatic Adenocarcinoma by Targeting the Conductor, MYC. DOI 10.1158/2159-8290.cd-20-0046↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. DOI 10.1038/s41388-024-03196-w↗
- Motoyuki Tsuda · 4 shared papers, 2025 · strength 0.65
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Brittany L. Allen-Petersen · 3 shared papers, 2024–2025 · strength 0.50
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. DOI 10.1038/s41388-024-03196-w↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Kevin Hawthorne · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Ellen M. Langer · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Lisa M. Coussens · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Patrick Joseph Worth · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Jonathan Robert Brody · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Jason M. Link · 3 shared papers, 2025 · strength 0.48
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Katherine R. Pelz · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Amy S. Farrell · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Terry K. Morgan · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Katie E. Blise · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Brett C. Sheppard · 2 shared papers, 2025 · strength 0.32
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Vidhi M. Shah · 2 shared papers, 2025 · strength 0.32
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Gordon Brent Mills · 2 shared papers, 2025 · strength 0.31
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Adel Kardosh · 2 shared papers, 2025 · strength 0.31
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Charles D. Lopez · 2 shared papers, 2025 · strength 0.31
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper A window trial in metastatic pancreatic ductal adenocarcinoma reveals resistance mechanisms to targeting the KRAS-MEK p… DOI 10.64898/2025.12.12.693851↗
- Meghan M. Joly · 1 shared paper, 2025 · strength 0.17
- Laura DeLong Wood · 1 shared paper, 2025 · strength 0.17
- Ashley L. Kiemen · 1 shared paper, 2025 · strength 0.17
- Luis Solorio · 1 shared paper, 2024 · strength 0.17
- Garima Baral · 1 shared paper, 2024 · strength 0.17
- Samantha L. Tinsley · 1 shared paper, 2024 · strength 0.17
- Marina Pasca di Magliano · 1 shared paper, 2024 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.