ResearcherOregon Health & Science UniversityActive in the last 2 years
Terry K. Morgan
Matched on ORCID iD 0000-0003-0742-5012; OpenAlex author ID A5009699749. Sources: OpenAlex.
Their work
- Papers
- 19 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 63
2012: 1 paper · 2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: 1 paper · 2018: none · 2019: 3 papers · 2020: none · 2021: 2 papers · 2022: none · 2023: 1 paper · 2024: 2 papers · 2025: 7 papers · 2026: 1 paper
Papers 19
- Liquid Biopsy Differentiation of Pancreatic Cancer From Non-Cancerous Pancreatic Disease Using Dielectrophoresis-Recovered Nanoparticles Carrying Cell-Free DNA… Co-author DOI 10.1002/smll.202502532↗
- Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. Co-author DOI 10.1016/j.xpro.2025.103672↗
- Cell free RNA detection of pancreatic cancer in pre diagnostic high risk and symptomatic patients. Co-author DOI 10.1038/s41467-025-62685-y↗
- Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in pancreatic cancer. Co-author DOI 10.1038/s43018-024-00881-3↗
- Self-amplifying NRF2-EZH2 epigenetic loop converts KRAS-initiated progenitors to invasive pancreatic cancer. Co-author DOI 10.1038/s43018-025-01003-3↗
- Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Co-author DOI 10.1038/s43018-025-01035-9↗
- Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate Conversion Co-author DOI 10.1101/2025.06.25.661419↗
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model Co-author DOI 10.1101/2025.07.14.664767↗
- Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. Co-author DOI 10.1016/j.heliyon.2024.e27221↗
- Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Co-author DOI 10.1038/s43018-023-00703-y↗
- Deletion of the mRNA stability factor ELAVL1 (HuR) in pancreatic cancer cells disrupts the tumor microenvironment integrity. Co-author DOI 10.1093/narcan/zcad016↗
- Longitudinal Analysis of Human Pancreatic Adenocarcinoma Development Reveals Transient Gene Expression Signatures. Co-author DOI 10.1158/1541-7786.mcr-21-0483↗
- Tumor-Infiltrating Leukocyte Phenotypes Distinguish Outcomes in Related Patients With Pancreatic Adenocarcinoma. Co-author DOI 10.1200/po.20.00287↗
- MyD88 signalling is critical in the development of pancreatic cancer cachexia. Co-author DOI 10.1002/jcsm.12377↗
- The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Co-author DOI 10.1038/s41467-019-12657-w↗
- Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Co-author DOI 10.1038/s41467-019-13151-z↗
- Establishment and characterization of a novel murine model of pancreatic cancer cachexia. Co-author DOI 10.1002/jcsm.12225↗
- Human pancreatic cancer fusion 2 (HPC2) 1-B3: a novel monoclonal antibody to screen for pancreatic ductal dysplasia. Co-author DOI 10.1002/cncy.21223↗
- The novel monoclonal antibody HPC2 and N-cadherin distinguish pancreatic ductal adenocarcinoma from cholangiocarcinoma. First author DOI 10.1016/j.humpath.2011.11.012↗
Closest collaborators
The 25 strongest of 63 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rosalie C. Sears · 10 shared papers, 2021–2026 · strength 1.80
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Liquid Biopsy Differentiation of Pancreatic Cancer From Non-Cancerous Pancreatic Disease Using Dielectrophoresis-Recove… DOI 10.1002/smll.202502532↗
- Paper Cell free RNA detection of pancreatic cancer in pre diagnostic high risk and symptomatic patients. DOI 10.1038/s41467-025-62685-y↗
- Jason M. Link · 5 shared papers, 2021–2025 · strength 0.88
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate Conversion DOI 10.1101/2025.06.25.661419↗
- Brett C. Sheppard · 6 shared papers, 2012–2025 · strength 0.83
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate Conversion DOI 10.1101/2025.06.25.661419↗
- Dmytro Grygoryev · 4 shared papers, 2021–2025 · strength 0.74
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate Conversion DOI 10.1101/2025.06.25.661419↗
- Jungsun Kim · 4 shared papers, 2021–2025 · strength 0.74
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Long Noncoding RNAs Preserve Pancreatic Cancer Identity and Resist Cell Fate Conversion DOI 10.1101/2025.06.25.661419↗
- Lisa M. Coussens · 5 shared papers, 2019–2025 · strength 0.60
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper Tumor-Infiltrating Leukocyte Phenotypes Distinguish Outcomes in Related Patients With Pancreatic Adenocarcinoma. DOI 10.1200/po.20.00287↗
- Brittany L. Allen-Petersen · 3 shared papers, 2024–2025 · strength 0.58
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Jonathan Robert Brody · 3 shared papers, 2023–2025 · strength 0.50
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Deletion of the mRNA stability factor ELAVL1 (HuR) in pancreatic cancer cells disrupts the tumor microenvironment integ… DOI 10.1093/narcan/zcad016↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Motoyuki Tsuda · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Paper Self-amplifying NRF2-EZH2 epigenetic loop converts KRAS-initiated progenitors to invasive pancreatic cancer. DOI 10.1038/s43018-025-01003-3↗
- Sohinee Bhattacharyya · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Holly Sandborg · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Mara H. Sherman · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Luis Francisco Diaz · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Katherine R. Pelz · 4 shared papers, 2019–2025 · strength 0.43
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Julien Tessier · 2 shared papers, 2025 · strength 0.41
- Gordon Brent Mills · 2 shared papers, 2025 · strength 0.37
- Thuy T. M. Ngo · 2 shared papers, 2021–2025 · strength 0.35
- Kevin G. Burfeind · 4 shared papers, 2017–2019 · strength 0.34
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Paper Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cance… DOI 10.1038/s41467-019-13151-z↗
- Katherine A. Michaelis · 4 shared papers, 2017–2019 · strength 0.34
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Paper Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cance… DOI 10.1038/s41467-019-13151-z↗
- Xinxia Zhu · 4 shared papers, 2017–2019 · strength 0.34
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Paper Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cance… DOI 10.1038/s41467-019-13151-z↗
- Daniel L. Marks · 4 shared papers, 2017–2019 · strength 0.34
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Paper Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cance… DOI 10.1038/s41467-019-13151-z↗
- Patrick Joseph Worth · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Isabel A. English · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Kevin Hawthorne · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Ellen M. Langer · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.