ResearcherOregon Health & Science UniversityActive in the last 2 years
Daniel L. Marks
Matched on ORCID iD 0000-0003-2675-7047; OpenAlex author ID A5000850327; NIH RePORTER profile ID 1929010. Sources: OpenAlex.
Their work
- Papers
- 13 (first author 0, last author 9)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 31
2017: 1 paper · 2018: none · 2019: 3 papers · 2020: 3 papers · 2021: 1 paper · 2022: 2 papers, 1 grant · 2023: none · 2024: 1 paper · 2025: none · 2026: 2 papers
Papers 13
- Pancreatic cancer cachexia promotes cardiac dysfunction through altered adrenergic signalling in the heart. Co-author DOI 10.1113/jp290616↗
- Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic cancer. Co-author DOI 10.1172/jci.insight.192814↗
- GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. Last author DOI 10.1016/j.tranon.2024.102129↗
- Validation of automated body composition analysis using diagnostic computed tomography imaging in patients with pancreatic cancer. Co-author DOI 10.1016/j.amjsurg.2022.03.025↗
- Critical changes in hypothalamic gene networks in response to pancreatic cancer as found by single-cell RNA sequencing. Last author DOI 10.1016/j.molmet.2022.101441↗
- Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. Last author DOI 10.1038/s41467-021-22361-3↗
- Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic cancer. Last author DOI 10.1002/glia.23796↗
- Multidisciplinary standards of care and recent progress in pancreatic ductal adenocarcinoma. Co-author DOI 10.3322/caac.21626↗
- Circulating myeloid cells invade the central nervous system to mediate cachexia during pancreatic cancer. Last author DOI 10.7554/elife.54095↗
- MyD88 signalling is critical in the development of pancreatic cancer cachexia. Last author DOI 10.1002/jcsm.12377↗
- The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Last author DOI 10.1038/s41467-019-12657-w↗
- Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Last author DOI 10.1038/s41467-019-13151-z↗
- Establishment and characterization of a novel murine model of pancreatic cancer cachexia. Last author DOI 10.1002/jcsm.12225↗
Grants 1
Closest collaborators
The 25 strongest of 31 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Xinxia Zhu · 9 shared papers, 2017–2026 · strength 1.11
- Paper Pancreatic cancer cachexia promotes cardiac dysfunction through altered adrenergic signalling in the heart. DOI 10.1113/jp290616↗
- Paper GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. DOI 10.1016/j.tranon.2024.102129↗
- Paper Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic ca… DOI 10.1002/glia.23796↗
- Brennan G. Olson · 8 shared papers, 2019–2024 · strength 0.95
- Paper GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. DOI 10.1016/j.tranon.2024.102129↗
- Paper Critical changes in hypothalamic gene networks in response to pancreatic cancer as found by single-cell RNA sequencing. DOI 10.1016/j.molmet.2022.101441↗
- Paper Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic ca… DOI 10.1002/glia.23796↗
- Aaron J. Grossberg · 5 shared papers, 2020–2026 · strength 0.83
- Paper Pancreatic cancer cachexia promotes cardiac dysfunction through altered adrenergic signalling in the heart. DOI 10.1113/jp290616↗
- Paper GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. DOI 10.1016/j.tranon.2024.102129↗
- Paper Validation of automated body composition analysis using diagnostic computed tomography imaging in patients with pancrea… DOI 10.1016/j.amjsurg.2022.03.025↗
- Parham Diba · 3 shared papers, 2024–2026 · strength 0.70
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper Pancreatic cancer cachexia promotes cardiac dysfunction through altered adrenergic signalling in the heart. DOI 10.1113/jp290616↗
- Paper GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. DOI 10.1016/j.tranon.2024.102129↗
- Ariana L. Sattler · 3 shared papers, 2024–2026 · strength 0.70
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper Pancreatic cancer cachexia promotes cardiac dysfunction through altered adrenergic signalling in the heart. DOI 10.1113/jp290616↗
- Paper GDF15 and LCN2 for early detection and prognosis of pancreatic cancer. DOI 10.1016/j.tranon.2024.102129↗
- Kevin G. Burfeind · 7 shared papers, 2017–2021 · strength 0.67
- Paper Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic ca… DOI 10.1002/glia.23796↗
- Paper Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. DOI 10.1038/s41467-021-22361-3↗
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Katherine A. Michaelis · 7 shared papers, 2017–2021 · strength 0.67
- Paper Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic ca… DOI 10.1002/glia.23796↗
- Paper Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. DOI 10.1038/s41467-021-22361-3↗
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Christian Huisman · 3 shared papers, 2020–2022 · strength 0.39
- Paper Critical changes in hypothalamic gene networks in response to pancreatic cancer as found by single-cell RNA sequencing. DOI 10.1016/j.molmet.2022.101441↗
- Paper Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic ca… DOI 10.1002/glia.23796↗
- Paper Circulating myeloid cells invade the central nervous system to mediate cachexia during pancreatic cancer. DOI 10.7554/elife.54095↗
- Katherine R. Pelz · 4 shared papers, 2019–2021 · strength 0.38
- Paper Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. DOI 10.1038/s41467-021-22361-3↗
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Terry K. Morgan · 4 shared papers, 2017–2019 · strength 0.34
- Paper MyD88 signalling is critical in the development of pancreatic cancer cachexia. DOI 10.1002/jcsm.12377↗
- Paper The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. DOI 10.1038/s41467-019-12657-w↗
- Paper Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cance… DOI 10.1038/s41467-019-13151-z↗
- Christopher R. Deig · 2 shared papers, 2020–2022 · strength 0.28
- Kevin Hawthorne · 1 shared paper, 2026 · strength 0.26
- Sebnem Ece Eksi · 1 shared paper, 2026 · strength 0.26
- Mara H. Sherman · 1 shared paper, 2026 · strength 0.26
- Rosalie C. Sears · 1 shared paper, 2026 · strength 0.26
- Teresa A Zimmers · 1 shared paper, 2026 · strength 0.26
- Paige C. Arneson‐Wissink · 1 shared paper, 2026 · strength 0.26
- Brett C. Sheppard · 1 shared paper, 2022 · strength 0.17
- Ali Noel Gunesch · 1 shared paper, 2022 · strength 0.17
- Thomas Lee Sutton · 1 shared paper, 2022 · strength 0.17
- Lisa M. Coussens · 2 shared papers, 2019 · strength 0.16
- H. Carlo Maurer · 2 shared papers, 2019 · strength 0.16
- Shannon M. Liudahl · 2 shared papers, 2019 · strength 0.16
- Kenneth P. Olive · 2 shared papers, 2019 · strength 0.16
- William L. Hwang · 1 shared paper, 2020 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.