ResearcherMemorial Sloan Kettering Cancer CenterActive in the last 2 years
Sohinee Bhattacharyya
Matched on ORCID iD 0000-0001-6180-8415; OpenAlex author ID A5074718111. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 22
2020: 1 paper · 2021: none · 2022: 1 paper · 2023: none · 2024: 1 paper · 2025: 1 paper · 2026: 1 paper
Papers 5
- Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. Co-author DOI 10.1158/0008-5472.can-25-1969↗
- Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Co-author DOI 10.1038/s43018-025-01035-9↗
- Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Co-author DOI 10.1038/s43018-023-00703-y↗
- Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer-Associated Fibroblasts. Co-author DOI 10.1158/2159-8290.cd-21-0601↗
- Acidic fibroblast growth factor underlies microenvironmental regulation of MYC in pancreatic cancer. Co-author DOI 10.1084/jem.20191805↗
Closest collaborators
The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mara H. Sherman · 5 shared papers, 2020–2026 · strength 0.93
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. DOI 10.1158/0008-5472.can-25-1969↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Terry K. Morgan · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Holly Sandborg · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Luis Francisco Diaz · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Dmitri Petrov · 1 shared paper, 2026 · strength 0.23
- Sofia Ferreira · 1 shared paper, 2026 · strength 0.23
- Brittany M. Flowers · 1 shared paper, 2026 · strength 0.23
- Ghmkin Hassan · 1 shared paper, 2026 · strength 0.23
- Laura DeLong Wood · 1 shared paper, 2026 · strength 0.23
- Andrew J. Aguirre · 1 shared paper, 2026 · strength 0.23
- Valerie Marie Weaver · 1 shared paper, 2026 · strength 0.23
- Laura D Attardi · 1 shared paper, 2026 · strength 0.23
- Monte Meier Winslow · 1 shared paper, 2026 · strength 0.23
- Jason M. Link · 1 shared paper, 2020 · strength 0.13
- Rosalie C. Sears · 1 shared paper, 2020 · strength 0.13
- Jennifer M. Finan · 1 shared paper, 2022 · strength 0.13
- Ariana L. Sattler · 1 shared paper, 2022 · strength 0.13
- Zheng Xia · 1 shared paper, 2022 · strength 0.13
- Christopher C. DuFort · 1 shared paper, 2022 · strength 0.13
- Erin J. Helms · 1 shared paper, 2022 · strength 0.13
- David W. Dawson · 1 shared paper, 2022 · strength 0.13
- Sunil R. Hingorani · 1 shared paper, 2022 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.