ResearcherBeth Israel Deaconess Medical CenterActive in the last 2 years
Valerie Marie Weaver
Matched on ORCID iD 0000-0003-4786-6752; OpenAlex author ID A5022114303; NIH RePORTER profile ID 6896322. Sources: funder_awards.
Their work
- Papers
- 8 (first author 0, last author 2)
- Trials
- 0
- Grants
- 3
- Labs led
- 1
- Collaborators
- 43
2013: 1 grant · 2014: 1 grant · 2015: none · 2016: 1 paper · 2017: none · 2018: 1 paper · 2019: none · 2020: 1 paper · 2021: none · 2022: none · 2023: none · 2024: 2 papers · 2025: 2 papers · 2026: 1 paper, 1 grant
Papers 8
- Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. Co-author DOI 10.1158/0008-5472.can-25-1969↗
- A STAT3/integrin axis accelerates pancreatic cancer initiation and progression. Co-author DOI 10.1016/j.celrep.2025.116010↗
- Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplastic Reaction. Co-author DOI 10.1021/acs.biochem.5c00125↗
- Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Last author DOI 10.1038/s41591-023-02694-w↗
- nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer Co-author DOI 10.1101/2024.09.23.614610↗
- Pancreatic ductal adenocarcinoma progression is restrained by stromal matrix. Co-author DOI 10.1172/jci136760↗
- Compartment resolved proteomics reveals a dynamic matrisome in a biomechanically driven model of pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.regen.2018.03.002↗
- Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Last author DOI 10.1038/nm.4082↗
Grants 3
Closest collaborators
The 25 strongest of 43 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Sarah Monica Knox · 1 shared grant, 2026 · strength 1.26
- Kirk Charles Hansen · 4 shared papers, 2016–2025 · strength 0.55
- Paper Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplasti… DOI 10.1021/acs.biochem.5c00125↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Compartment resolved proteomics reveals a dynamic matrisome in a biomechanically driven model of pancreatic ductal aden… DOI 10.1016/j.regen.2018.03.002↗
- Alex Barrett · 3 shared papers, 2016–2025 · strength 0.45
- Paper Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplasti… DOI 10.1021/acs.biochem.5c00125↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Laura DeLong Wood · 3 shared papers, 2016–2026 · strength 0.43
- Paper Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. DOI 10.1158/0008-5472.can-25-1969↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Grace Kim · 3 shared papers, 2016–2024 · strength 0.35
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer DOI 10.1101/2024.09.23.614610↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Matthias Hebrok · 3 shared papers, 2016–2024 · strength 0.35
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer DOI 10.1101/2024.09.23.614610↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Raghu Kalluri · 2 shared papers, 1 shared grant, 2014–2024 · strength 0.35
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Grant Role of Fibroblasts, Myeloid Cells and Matrix in PDAC U01CA151925↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Eric Andrew Collisson · 3 shared papers, 2016–2024 · strength 0.29
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Pancreatic ductal adenocarcinoma progression is restrained by stromal matrix. DOI 10.1172/jci136760↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Todd M. Pitts · 1 shared paper, 2025 · strength 0.25
- Wells A. Messersmith · 1 shared paper, 2025 · strength 0.25
- Jen Jen Yeh · 2 shared papers, 2020–2024 · strength 0.23
- Dmitri Petrov · 1 shared paper, 2026 · strength 0.23
- Sohinee Bhattacharyya · 1 shared paper, 2026 · strength 0.23
- Sofia Ferreira · 1 shared paper, 2026 · strength 0.23
- Brittany M. Flowers · 1 shared paper, 2026 · strength 0.23
- Ghmkin Hassan · 1 shared paper, 2026 · strength 0.23
- Mara H. Sherman · 1 shared paper, 2026 · strength 0.23
- Andrew J. Aguirre · 1 shared paper, 2026 · strength 0.23
- Laura D Attardi · 1 shared paper, 2026 · strength 0.23
- Monte Meier Winslow · 1 shared paper, 2026 · strength 0.23
- Hanane Laklai · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Nilotpal Roy · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Harold L. Moses · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Julia Sidenius Johansen · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Christine A. Iacobuzio–Donahue · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.