ResearcherThomas Jefferson UniversityActive in the last 2 years
Matthias Hebrok
Matched on ORCID iD 0000-0002-3833-8862; OpenAlex author ID A5002156552; NIH RePORTER profile ID 6796101. Sources: funder_awards.
Their work
- Papers
- 13 (first author 1, last author 6)
- Trials
- 0
- Grants
- 4
- Labs led
- 1
- Collaborators
- 64
2008: 1 grant · 2009: none · 2010: none · 2011: 1 paper, 1 grant · 2012: 1 paper · 2013: 1 paper · 2014: 2 papers, 1 grant · 2015: 2 papers · 2016: 2 papers · 2017: none · 2018: 1 grant · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: 3 papers · 2025: none · 2026: 1 paper
Papers 13
- KDM6 Histone Demethylases Tune Pancreatic Cancer Plasticity. Last author DOI 10.1158/0008-5472.can-25-3126↗
- Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Co-author DOI 10.1038/s41591-023-02694-w↗
- KRAS degradation averts PDAC chemoresistance First author DOI 10.1038/s43018-023-00708-7↗
- nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer Last author DOI 10.1101/2024.09.23.614610↗
- Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Co-author DOI 10.1038/nm.4082↗
- PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. Co-author DOI 10.1101/gad.291021.116↗
- Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. Last author DOI 10.1101/gad.256628.114↗
- Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. Co-author DOI 10.15252/embj.201489574↗
- The chromatin regulator Brg1 suppresses formation of intraductal papillary mucinous neoplasm and pancreatic ductal adenocarcinoma. Last author DOI 10.1038/ncb2916↗
- Dicer regulates differentiation and viability during mouse pancreatic cancer initiation. Last author DOI 10.1371/journal.pone.0095486↗
- Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. Co-author DOI 10.1158/0008-5472.can-12-2067↗
- Identification of Sox9-dependent acinar-to-ductal reprogramming as the principal mechanism for initiation of pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.ccr.2012.10.025↗
- Stat3 and MMP7 contribute to pancreatic ductal adenocarcinoma initiation and progression. Last author DOI 10.1016/j.ccr.2011.03.002↗
Grants 4
- Epigenetic regulation of pancreatic cancer R01CA172045↗
- Embryonic signaling pathways in pancreatic cancer R01CA112537↗
- Role of miRNAs in Pancreatic Adenocarcinoma AWARD pancan:2011-matthias-hebrok-phd-role-of-mirnas-in-pancreatic-adenocarcin
- NF-kB Signaling in PanIN Formation AWARD pancan:2008-matthias-hebrok-phd-nf-kb-signaling-in-panin-formation
Closest collaborators
The 25 strongest of 64 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Laura Leonhardt · 3 shared papers, 2016–2026 · strength 0.70
- Paper KRAS degradation averts PDAC chemoresistance DOI 10.1038/s43018-023-00708-7↗
- Paper KDM6 Histone Demethylases Tune Pancreatic Cancer Plasticity. DOI 10.1158/0008-5472.can-25-3126↗
- Paper PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. DOI 10.1101/gad.291021.116↗
- Grace Kim · 7 shared papers, 2011–2026 · strength 0.65
- Paper KDM6 Histone Demethylases Tune Pancreatic Cancer Plasticity. DOI 10.1158/0008-5472.can-25-3126↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer DOI 10.1101/2024.09.23.614610↗
- David W. Dawson · 5 shared papers, 2014–2026 · strength 0.48
- Paper KDM6 Histone Demethylases Tune Pancreatic Cancer Plasticity. DOI 10.1158/0008-5472.can-25-3126↗
- Paper nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer DOI 10.1101/2024.09.23.614610↗
- Paper Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. DOI 10.1101/gad.256628.114↗
- Valerie Marie Weaver · 3 shared papers, 2016–2024 · strength 0.35
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper nSMase2-mediated exosome secretion shapes the tumor microenvironment to immunologically support pancreatic cancer DOI 10.1101/2024.09.23.614610↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Audrey M. Hendley · 2 shared papers, 2024–2026 · strength 0.35
- Nilotpal Roy · 5 shared papers, 2014–2024 · strength 0.33
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. DOI 10.1101/gad.256628.114↗
- Paper PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. DOI 10.1101/gad.291021.116↗
- Eric Andrew Collisson · 3 shared papers, 2015–2024 · strength 0.25
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. DOI 10.1101/gad.256628.114↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Alex Barrett · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Hanane Laklai · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Laura DeLong Wood · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Raghu Kalluri · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Harold L. Moses · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Julia Sidenius Johansen · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Christine A. Iacobuzio–Donahue · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Kirk Charles Hansen · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Giuseppe Malleo · 1 shared paper, 2026 · strength 0.20
- Jin Peng He · 1 shared paper, 2026 · strength 0.20
- Rondell Patrell Graham · 1 shared paper, 2026 · strength 0.20
- Elie M. Ghabi · 1 shared paper, 2026 · strength 0.20
- Claudio Luchini · 1 shared paper, 2026 · strength 0.20
- David Kuang-Fu Chang · 2 shared papers, 2016–2024 · strength 0.19
- Andrew Victor Biankin · 2 shared papers, 2016–2024 · strength 0.19
- John P. Morris · 5 shared papers, 2011–2014 · strength 0.16
- Paper Dicer regulates differentiation and viability during mouse pancreatic cancer initiation. DOI 10.1371/journal.pone.0095486↗
- Paper The chromatin regulator Brg1 suppresses formation of intraductal papillary mucinous neoplasm and pancreatic ductal aden… DOI 10.1038/ncb2916↗
- Paper Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. DOI 10.1158/0008-5472.can-12-2067↗
- Guido von Figura · 4 shared papers, 2012–2015 · strength 0.15
- Paper Brg1 promotes both tumor-suppressive and oncogenic activities at distinct stages of pancreatic cancer formation. DOI 10.1101/gad.256628.114↗
- Paper Dicer regulates differentiation and viability during mouse pancreatic cancer initiation. DOI 10.1371/journal.pone.0095486↗
- Paper The chromatin regulator Brg1 suppresses formation of intraductal papillary mucinous neoplasm and pancreatic ductal aden… DOI 10.1038/ncb2916↗
- Grainne Mary O'Kane · 1 shared paper, 2024 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.