ResearcherUniversity of Colorado DenverActive in the last 2 years
Alex Barrett
Matched on OpenAlex author ID A5062139399. Sources: OpenAlex.
Their work
- Papers
- 3
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 14
2016: 1 paper · 2017: none · 2018: none · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: 1 paper · 2025: 1 paper
Papers 3
- Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplastic Reaction. Co-author DOI 10.1021/acs.biochem.5c00125↗
- Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Co-author DOI 10.1038/s41591-023-02694-w↗
- Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Co-author DOI 10.1038/nm.4082↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Valerie Marie Weaver · 3 shared papers, 2016–2025 · strength 0.45
- Paper Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplasti… DOI 10.1021/acs.biochem.5c00125↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Kirk Charles Hansen · 3 shared papers, 2016–2025 · strength 0.45
- Paper Pancreatic Cancer Progression Involves Increased Lysyl Oxidase-Mediated Collagen Cross-Links as Part of the Desmoplasti… DOI 10.1021/acs.biochem.5c00125↗
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Todd M. Pitts · 1 shared paper, 2025 · strength 0.25
- Wells A. Messersmith · 1 shared paper, 2025 · strength 0.25
- Grace Kim · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Hanane Laklai · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Nilotpal Roy · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Matthias Hebrok · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Laura DeLong Wood · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Eric Andrew Collisson · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Raghu Kalluri · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Harold L. Moses · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Julia Sidenius Johansen · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
- Christine A. Iacobuzio–Donahue · 2 shared papers, 2016–2024 · strength 0.20
- Paper Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and … DOI 10.1038/s41591-023-02694-w↗
- Paper Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. DOI 10.1038/nm.4082↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.