ResearcherPurdue University West LafayetteActive in the last 2 years
Brittany L. Allen-Petersen
Matched on ORCID iD 0000-0002-7436-9412; OpenAlex author ID A5052134233; NIH RePORTER profile ID 11725379. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 1, last author 3)
- Trials
- 0
- Grants
- 4
- Labs led
- 1
- Collaborators
- 47
2014: 1 paper · 2015: none · 2016: none · 2017: 1 paper, 1 grant · 2018: none · 2019: 1 paper · 2020: none · 2021: none · 2022: 1 paper, 2 grants · 2023: none · 2024: 2 papers · 2025: 5 papers · 2026: 1 paper, 1 grant
Papers 12
- Activation of PP2A-B56a leads to aberrant EGFR signaling and proliferative phenotypes in PDAC. Last author DOI 10.1158/1535-7163.mct-25-0970↗
- Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. Co-author DOI 10.1016/j.devcel.2024.12.003↗
- Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. Co-author DOI 10.1016/j.xpro.2025.103672↗
- PP2A activation drives aberrant macropinocytosis and cell death in pancreatic ductal adenocarcinoma Last author DOI 10.1101/2025.07.14.664742↗
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model Co-author DOI 10.1101/2025.07.14.664767↗
- The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3437↗
- Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. Co-author DOI 10.1016/j.heliyon.2024.e27221↗
- KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. Last author DOI 10.1038/s41388-024-03196-w↗
- Microfluidics Formulated Liposomes of Hypoxia Activated Prodrug for Treatment of Pancreatic Cancer. Co-author DOI 10.3390/pharmaceutics14040713↗
- Activation of PP2A and Inhibition of mTOR Synergistically Reduce MYC Signaling and Decrease Tumor Growth in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-18-0717↗
- MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome and chemoresistance. Co-author DOI 10.1038/s41467-017-01967-6↗
- Targeting inhibitors of the tumor suppressor PP2A for the treatment of pancreatic cancer. First author DOI 10.1158/1541-7786.mcr-13-0542↗
Grants 4
- Posttranslational regulation of macropinocytosis in pancreatic cancer R01CA295946↗
- The Role of PP2A in Regulating Pancreatic Cancer Macropinocytosis AWARD pancan:2022-brittany-allen-petersen-phd-the-role-of-pp2a-in-regulating-pancreati
- The role of PP2A B56a in pancreatic tumorigenesis K22CA237620↗
- MYC is a critical downstream effector in KRAS-driven pancreatic cancer F32CA192769↗
Closest collaborators
The 25 strongest of 47 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rosalie C. Sears · 10 shared papers, 2014–2025 · strength 1.44
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Brett C. Sheppard · 7 shared papers, 2014–2025 · strength 0.89
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Jason M. Link · 4 shared papers, 2017–2025 · strength 0.62
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Garima Baral · 3 shared papers, 2024–2026 · strength 0.61
- Paper PP2A activation drives aberrant macropinocytosis and cell death in pancreatic ductal adenocarcinoma DOI 10.1101/2025.07.14.664742↗
- Paper Activation of PP2A-B56a leads to aberrant EGFR signaling and proliferative phenotypes in PDAC. DOI 10.1158/1535-7163.mct-25-0970↗
- Paper KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. DOI 10.1038/s41388-024-03196-w↗
- Terry K. Morgan · 3 shared papers, 2024–2025 · strength 0.58
- Paper Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts. DOI 10.1016/j.xpro.2025.103672↗
- Paper Sendai virus is robust and consistent in delivering genes into human pancreatic cancer cells. DOI 10.1016/j.heliyon.2024.e27221↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Isabel A. English · 3 shared papers, 2024–2025 · strength 0.50
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper KRAS-mediated upregulation of CIP2A promotes suppression of PP2A-B56α to initiate pancreatic cancer development. DOI 10.1038/s41388-024-03196-w↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Claire M. Pfeffer · 2 shared papers, 2025–2026 · strength 0.44
- Amy S. Farrell · 4 shared papers, 2014–2025 · strength 0.42
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Katherine R. Pelz · 3 shared papers, 2019–2025 · strength 0.41
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Activation of PP2A and Inhibition of mTOR Synergistically Reduce MYC Signaling and Decrease Tumor Growth in Pancreatic … DOI 10.1158/0008-5472.can-18-0717↗
- Dmytro Grygoryev · 2 shared papers, 2024–2025 · strength 0.41
- Jungsun Kim · 2 shared papers, 2024–2025 · strength 0.41
- Jennifer P. Morton · 3 shared papers, 2017–2026 · strength 0.35
- Paper Activation of PP2A-B56a leads to aberrant EGFR signaling and proliferative phenotypes in PDAC. DOI 10.1158/1535-7163.mct-25-0970↗
- Paper Activation of PP2A and Inhibition of mTOR Synergistically Reduce MYC Signaling and Decrease Tumor Growth in Pancreatic … DOI 10.1158/0008-5472.can-18-0717↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Kevin Hawthorne · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Motoyuki Tsuda · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Ellen M. Langer · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Vidhi M. Shah · 2 shared papers, 2022–2025 · strength 0.31
- Megan T. Hoffman · 2 shared papers, 2017–2025 · strength 0.26
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Howard C. Crawford · 2 shared papers, 2017–2025 · strength 0.26
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Meghan M. Joly · 2 shared papers, 2017–2025 · strength 0.22
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Patrick Joseph Worth · 2 shared papers, 2017–2025 · strength 0.22
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Julien Tessier · 1 shared paper, 2025 · strength 0.22
- Kathleen E. DelGiorno · 1 shared paper, 2025 · strength 0.21
- Nina G. Steele · 1 shared paper, 2025 · strength 0.21
- Daniel James Salas-Escabillas · 1 shared paper, 2025 · strength 0.21
- Simone Benitz · 1 shared paper, 2025 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.