ResearcherUniversity of MichiganActive in the last 2 years
Megan T. Hoffman
Matched on ORCID iD 0000-0003-0907-9977; OpenAlex author ID A5035111732. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 4, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 83
2016: 1 paper · 2017: 1 paper · 2018: none · 2019: none · 2020: none · 2021: 2 papers · 2022: 1 paper · 2023: 2 papers · 2024: 1 paper · 2025: 1 paper · 2026: 2 papers
Papers 11
- CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. First author DOI 10.1016/j.cell.2026.08.045↗
- Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockade-responsive pancreatic cancer. Co-author DOI 10.1093/immadv/ltag013↗
- Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. First author DOI 10.1016/j.devcel.2024.12.003↗
- Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer. First author DOI 10.1158/2159-8290.cd-24-0177↗
- Ornithine aminotransferase supports polyamine synthesis in pancreatic cancer. Co-author DOI 10.1038/s41586-023-05891-2↗
- Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. First author DOI 10.1053/j.gastro.2023.05.038↗
- Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. Co-author DOI 10.7554/elife.73245↗
- The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. Co-author DOI 10.1016/j.jcmgh.2020.08.011↗
- Inhibition of Hedgehog Signaling Alters Fibroblast Composition in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-20-3715↗
- MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome and chemoresistance. Co-author DOI 10.1038/s41467-017-01967-6↗
- PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. Co-author DOI 10.1101/gad.291021.116↗
Closest collaborators
The 25 strongest of 83 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Stephanie K. Dougan · 4 shared papers, 2023–2026 · strength 0.83
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Michael Lawrence Dougan · 3 shared papers, 2023–2026 · strength 0.69
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Andrew J. Aguirre · 3 shared papers, 2023–2026 · strength 0.69
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Howard C. Crawford · 6 shared papers, 2016–2025 · strength 0.61
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. DOI 10.1016/j.jcmgh.2020.08.011↗
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Brendan D. Parent · 2 shared papers, 2026 · strength 0.54
- Nina G. Steele · 4 shared papers, 2021–2025 · strength 0.51
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. DOI 10.1016/j.jcmgh.2020.08.011↗
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Daniel James Salas-Escabillas · 2 shared papers, 2021–2025 · strength 0.32
- Simone Benitz · 2 shared papers, 2021–2025 · strength 0.32
- Samantha B. Kemp · 3 shared papers, 2021–2022 · strength 0.30
- Paper The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. DOI 10.1016/j.jcmgh.2020.08.011↗
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Paper Inhibition of Hedgehog Signaling Alters Fibroblast Composition in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3715↗
- Yaqing Zhang · 3 shared papers, 2021–2022 · strength 0.30
- Paper The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. DOI 10.1016/j.jcmgh.2020.08.011↗
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Paper Inhibition of Hedgehog Signaling Alters Fibroblast Composition in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3715↗
- Marina Pasca di Magliano · 3 shared papers, 2021–2022 · strength 0.30
- Paper The Gustatory Sensory G-Protein GNAT3 Suppresses Pancreatic Cancer Progression in Mice. DOI 10.1016/j.jcmgh.2020.08.011↗
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Paper Inhibition of Hedgehog Signaling Alters Fibroblast Composition in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3715↗
- Rosalie C. Sears · 2 shared papers, 2017–2025 · strength 0.26
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Brittany L. Allen-Petersen · 2 shared papers, 2017–2025 · strength 0.26
- Paper Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. DOI 10.1016/j.devcel.2024.12.003↗
- Paper MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome… DOI 10.1038/s41467-017-01967-6↗
- Christopher J. Halbrook · 3 shared papers, 2016–2022 · strength 0.24
- Paper Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context. DOI 10.7554/elife.73245↗
- Paper Inhibition of Hedgehog Signaling Alters Fibroblast Composition in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3715↗
- Paper PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. DOI 10.1101/gad.291021.116↗
- Kathleen E. DelGiorno · 1 shared paper, 2025 · strength 0.21
- Filip Bednar · 2 shared papers, 2021 · strength 0.20
- Zeribe C. Nwosu · 2 shared papers, 2021–2022 · strength 0.19
- Costas Andreas Lyssiotis · 2 shared papers, 2021–2022 · strength 0.19
- Harshabad Singh · 1 shared paper, 2026 · strength 0.19
- Courtney T.S. Kureshi · 1 shared paper, 2026 · strength 0.19
- Chong Zuo · 1 shared paper, 2026 · strength 0.19
- George Molina · 1 shared paper, 2026 · strength 0.19
- Thomas E. Clancy · 1 shared paper, 2026 · strength 0.19
- Qiang Li · 1 shared paper, 2026 · strength 0.19
- Douglas Adam Rubinson · 1 shared paper, 2026 · strength 0.19
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.