ResearcherHarvard UniversityActive in the last 2 years
Thomas E. Clancy
Matched on ORCID iD 0000-0002-0949-4874; OpenAlex author ID A5087484173. Sources: OpenAlex.
Their work
- Papers
- 30 (first author 2, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 122
2015: 2 papers · 2016: none · 2017: 4 papers · 2018: 2 papers · 2019: 1 paper · 2020: 2 papers · 2021: 6 papers · 2022: 3 papers · 2023: 3 papers · 2024: 3 papers · 2025: 1 paper · 2026: 3 papers
Papers 30
- Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. Co-author DOI 10.1016/j.ccell.2026.04.005↗
- CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. Co-author DOI 10.1016/j.cell.2026.08.045↗
- Quantitative Assessments of Shared Decision-Making for Pancreatic Cancer Surgery: A Scoping Review. Co-author DOI 10.1016/j.jss.2026.03.060↗
- Retraction: Prostaglandin E2 Enhances Pancreatic Cancer Invasiveness through an Ets-1-Dependent Induction of Matrix Metalloproteinase-2. Co-author DOI 10.1158/0008-5472.can-25-0887↗
- Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. Co-author DOI 10.1136/gutjnl-2023-331074↗
- PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-23-1444↗
- Impact of Tumor Response After Neoadjuvant Treatment on Overall Survival Among Patients With Pancreatic Ductal Adenocarcinoma: National Cancer Database Analysi… Co-author DOI 10.7759/cureus.73524↗
- A blood-based metabolomic signature predictive of risk for pancreatic cancer. Co-author DOI 10.1016/j.xcrm.2023.101194↗
- Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and Extensive Intratumoral Heterogeneity. Co-author DOI 10.1158/0008-5472.can-22-3050↗
- Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-22-3930↗
- Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment in Resected Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-22-1125↗
- Predicting Surgical Margins in Patients With Borderline Resectable and Locally Advanced Pancreatic Cancer Undergoing Resection. Last author DOI 10.1177/00031348211011129↗
- Prognostic Factors in Patients With Borderline Resectable Pancreatic Ductal Adenocarcinoma Undergoing Resection. Last author DOI 10.1177/0003134821991962↗
- Microenvironment drives cell state, plasticity, and drug response in pancreatic cancer. Co-author DOI 10.1016/j.cell.2021.11.017↗
- Temporal Assessment of Prognostic Factors in Patients With Pancreatic Ductal Adenocarcinoma Undergoing Neoadjuvant Treatment and Resection. Last author DOI 10.1016/j.jss.2020.07.073↗
- Lead-Time Trajectory of CA19-9 as an Anchor Marker for Pancreatic Cancer Early Detection. Co-author DOI 10.1053/j.gastro.2020.11.052↗
- Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-20-3141↗
- Leukocyte Heterogeneity in Pancreatic Ductal Adenocarcinoma: Phenotypic and Spatial Features Associated with Clinical Outcome. Co-author DOI 10.1158/2159-8290.cd-20-0841↗
- Racial Disparity in Pancreatoduodenectomy for Borderline Resectable Pancreatic Adenocarcinoma. First author DOI 10.1245/s10434-020-08717-x↗
- Insulin-Like Growth Factor-1 Receptor Expression and Disease Recurrence and Survival in Patients with Resected Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1055-9965.epi-19-1315↗
- Neoadjuvant Therapy is Associated with Improved Survival in Borderline-Resectable Pancreatic Cancer. Co-author DOI 10.1245/s10434-019-08087-z↗
- Germline cancer susceptibility gene variants, somatic second hits, and survival outcomes in patients with resected pancreatic cancer. Co-author DOI 10.1038/s41436-018-0009-5↗
- Association of Alterations in Main Driver Genes With Outcomes of Patients With Resected Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1001/jamaoncol.2017.3420↗
- Real-time Genomic Characterization of Advanced Pancreatic Cancer to Enable Precision Medicine. Co-author DOI 10.1158/2159-8290.cd-18-0275↗
- Lymph node metastases in resected pancreatic ductal adenocarcinoma: predictors of disease recurrence and survival. Co-author DOI 10.1038/bjc.2017.349↗
- Borderline resectable pancreatic cancer: conceptual evolution and current approach to image-based classification. Co-author DOI 10.1093/annonc/mdx180↗
- Reply to the letter to the editor 'Borderline resectable pancreatic cancer: an evolving concept' by Petrucciani et al. Co-author DOI 10.1093/annonc/mdx273↗
- When, What, and Why of Perioperative Treatment of Potentially Curable Pancreatic Adenocarcinoma. First author DOI 10.1200/jco.2016.70.2134↗
- Surgery for Pancreatic Cancer. Co-author DOI 10.1016/j.hoc.2015.04.001↗
- Survival among patients with pancreatic cancer and long-standing or recent-onset diabetes mellitus. Co-author DOI 10.1200/jco.2014.57.5688↗
Closest collaborators
The 25 strongest of 122 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Brian Matthew Wolpin · 21 shared papers, 2015–2026 · strength 2.12
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Jonathan Andrew Nowak · 14 shared papers, 2017–2026 · strength 1.51
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Douglas Adam Rubinson · 14 shared papers, 2015–2026 · strength 1.39
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Stanley W. Ashley · 5 shared papers, 2021–2025 · strength 1.14
- Paper Retraction: Prostaglandin E2 Enhances Pancreatic Cancer Invasiveness through an Ets-1-Dependent Induction of Matrix Met… DOI 10.1158/0008-5472.can-25-0887↗
- Paper Predicting Surgical Margins in Patients With Borderline Resectable and Locally Advanced Pancreatic Cancer Undergoing Re… DOI 10.1177/00031348211011129↗
- Paper Prognostic Factors in Patients With Borderline Resectable Pancreatic Ductal Adenocarcinoma Undergoing Resection. DOI 10.1177/0003134821991962↗
- Kimberly J. Perez · 9 shared papers, 2017–2026 · strength 1.03
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- Andrew J. Aguirre · 10 shared papers, 2018–2026 · strength 1.02
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- Joseph Douglas Mancias · 7 shared papers, 2017–2026 · strength 0.97
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Chen Yuan · 11 shared papers, 2015–2024 · strength 0.93
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- George Molina · 5 shared papers, 2020–2026 · strength 0.89
- Paper Impact of Tumor Response After Neoadjuvant Treatment on Overall Survival Among Patients With Pancreatic Ductal Adenocar… DOI 10.7759/cureus.73524↗
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Kimmie Ng · 10 shared papers, 2015–2024 · strength 0.87
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Paper A blood-based metabolomic signature predictive of risk for pancreatic cancer. DOI 10.1016/j.xcrm.2023.101194↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- James M. Cleary · 6 shared papers, 2021–2026 · strength 0.80
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- Jiping Wang · 6 shared papers, 2020–2024 · strength 0.76
- Paper Racial Disparity in Pancreatoduodenectomy for Borderline Resectable Pancreatic Adenocarcinoma. DOI 10.1245/s10434-020-08717-x↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Neoadjuvant Therapy is Associated with Improved Survival in Borderline-Resectable Pancreatic Cancer. DOI 10.1245/s10434-019-08087-z↗
- Harshabad Singh · 5 shared papers, 2022–2026 · strength 0.72
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Oncogenic Drivers and Therapeutic Vulnerabilities in KRAS Wild-Type Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-22-3930↗
- Andressa Dias Costa · 7 shared papers, 2020–2024 · strength 0.72
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Stephanie K. Dougan · 4 shared papers, 2022–2026 · strength 0.65
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Weizheng Ren · 3 shared papers, 2021–2022 · strength 0.64
- Paper Predicting Surgical Margins in Patients With Borderline Resectable and Locally Advanced Pancreatic Cancer Undergoing Re… DOI 10.1177/00031348211011129↗
- Paper Prognostic Factors in Patients With Borderline Resectable Pancreatic Ductal Adenocarcinoma Undergoing Resection. DOI 10.1177/0003134821991962↗
- Paper Temporal Assessment of Prognostic Factors in Patients With Pancreatic Ductal Adenocarcinoma Undergoing Neoadjuvant Trea… DOI 10.1016/j.jss.2020.07.073↗
- Vicente Morales‐Oyarvide · 8 shared papers, 2017–2023 · strength 0.63
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Albert C. Koong · 7 shared papers, 2017–2023 · strength 0.55
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Richard Francis Dunne · 7 shared papers, 2017–2023 · strength 0.55
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Margaret M. Kozak · 7 shared papers, 2017–2023 · strength 0.55
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Daniel Tandel Chang · 7 shared papers, 2017–2023 · strength 0.55
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Aram F. Hezel · 7 shared papers, 2017–2023 · strength 0.55
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
- Ana Babić · 6 shared papers, 2017–2024 · strength 0.53
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Paper A blood-based metabolomic signature predictive of risk for pancreatic cancer. DOI 10.1016/j.xcrm.2023.101194↗
- Paper Lead-Time Trajectory of CA19-9 as an Anchor Marker for Pancreatic Cancer Early Detection. DOI 10.1053/j.gastro.2020.11.052↗
- Andrea J. Bullock · 6 shared papers, 2017–2024 · strength 0.51
- Paper Protein biomarkers and alternatively methylated cell-free DNA detect early stage pancreatic cancer. DOI 10.1136/gutjnl-2023-331074↗
- Paper A blood-based metabolomic signature predictive of risk for pancreatic cancer. DOI 10.1016/j.xcrm.2023.101194↗
- Paper Lead-Time Trajectory of CA19-9 as an Anchor Marker for Pancreatic Cancer Early Detection. DOI 10.1053/j.gastro.2020.11.052↗
- Sara A. Väyrynen · 5 shared papers, 2021–2023 · strength 0.48
- Paper Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and … DOI 10.1158/0008-5472.can-22-3050↗
- Paper Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment i… DOI 10.1158/1078-0432.ccr-22-1125↗
- Paper Composition, Spatial Characteristics, and Prognostic Significance of Myeloid Cell Infiltration in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-20-3141↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.