ResearcherHarvard UniversityActive in the last 2 years
Stephanie K. Dougan
Matched on ORCID iD 0000-0002-2263-363X; OpenAlex author ID A5001285437; NIH RePORTER profile ID 9406399. Sources: OpenAlex.
Their work
- Papers
- 21 (first author 3, last author 8)
- Trials
- 0
- Grants
- 2
- Labs led
- 1
- Collaborators
- 87
2017: 1 paper · 2018: 2 papers · 2019: 1 paper · 2020: none · 2021: 2 papers · 2022: 3 papers · 2023: 3 papers · 2024: 4 papers · 2025: 1 paper · 2026: 4 papers, 2 grants
Papers 21
- Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. Last author DOI 10.1016/j.ccell.2026.04.005↗
- CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. Last author DOI 10.1016/j.cell.2026.08.045↗
- Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockade-responsive pancreatic cancer. Last author DOI 10.1093/immadv/ltag013↗
- Autophagy Inhibition Reprograms the Tumor Microenvironment of Pancreatic Cancer to Promote Macrophage Phagocytosis of Tumor Cells. Co-author DOI 10.1158/0008-5472.can-25-2451↗
- De novo pyrimidine biosynthesis inhibition synergizes with BCL-XL targeting in pancreatic cancer. Co-author DOI 10.1038/s41467-025-61242-x↗
- PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer. Last author DOI 10.1158/1078-0432.ccr-23-1444↗
- Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-24-1164↗
- Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-24-0177↗
- Blockade of IL1β and PD1 with Combination Chemotherapy Reduces Systemic Myeloid Suppression in Metastatic Pancreatic Cancer with Heterogeneous Effects in the T… Last author DOI 10.1158/2326-6066.cir-23-1073↗
- Ornithine aminotransferase supports polyamine synthesis in pancreatic cancer. Co-author DOI 10.1038/s41586-023-05891-2↗
- Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. Last author DOI 10.1053/j.gastro.2023.05.038↗
- Multicenter randomized controlled trial of neoadjuvant chemoradiotherapy alone or in combination with pembrolizumab in patients with resectable or borderline r… Co-author DOI 10.1136/jitc-2023-007586↗
- Translational advances in pancreatic ductal adenocarcinoma therapy. First author DOI 10.1038/s43018-022-00349-2↗
- Bicarbonate transport as a vulnerability in pancreatic cancer. First author DOI 10.1038/s43018-022-00492-w↗
- Neoadjuvant Chemotherapy Is Associated with Altered Immune Cell Infiltration and an Anti-Tumorigenic Microenvironment in Resected Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-22-1125↗
- Radiation combines with immune checkpoint blockade to enhance T cell priming in a murine model of poorly immunogenic pancreatic cancer. Last author DOI 10.1098/rsob.210245↗
- Opportunities for Utilization of DNA Repair Inhibitors in Homologous Recombination Repair-Deficient and Proficient Pancreatic Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-21-1367↗
- Broadening the Impact of Immunotherapy to Pancreatic Cancer: Challenges and Opportunities. Last author DOI 10.1053/j.gastro.2018.12.038↗
- Unmasking Pancreatic Cancer: Epitope Spreading After Single Antigen Chimeric Antigen Receptor T-Cell Therapy in a Human Phase I Trial. First author DOI 10.1053/j.gastro.2018.06.023↗
- Radiation and Local Anti-CD40 Generate an Effective in situ Vaccine in Preclinical Models of Pancreatic Cancer. Co-author DOI 10.3389/fimmu.2018.02030↗
- The Pancreatic Cancer Microenvironment. Co-author DOI 10.1097/ppo.0000000000000288↗
Grants 2
Closest collaborators
The 25 strongest of 87 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Andrew J. Aguirre · 8 shared papers, 1 shared grant, 2021–2026 · strength 2.50
- Grant Stromal modulation of pancreatic cancer malignant cell state and therapeutic sensitivity U01CA274276↗
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper Translational advances in pancreatic ductal adenocarcinoma therapy. DOI 10.1038/s43018-022-00349-2↗
- Brian Matthew Wolpin · 8 shared papers, 2021–2026 · strength 1.20
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Jonathan Andrew Nowak · 8 shared papers, 2021–2026 · strength 1.20
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Michael Lawrence Dougan · 5 shared papers, 2023–2026 · strength 1.01
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- William C. Hahn · 1 shared grant, 2026 · strength 1.00
- Harshabad Singh · 6 shared papers, 2021–2026 · strength 0.90
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- James M. Cleary · 6 shared papers, 2021–2026 · strength 0.90
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- Joseph Douglas Mancias · 5 shared papers, 2022–2026 · strength 0.85
- Paper De novo pyrimidine biosynthesis inhibition synergizes with BCL-XL targeting in pancreatic cancer. DOI 10.1038/s41467-025-61242-x↗
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Megan T. Hoffman · 4 shared papers, 2023–2026 · strength 0.83
- Paper Growth factor supportive care for chemotherapy-induced neutropenia suppresses antitumour immunity in checkpoint blockad… DOI 10.1093/immadv/ltag013↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Brandon M. Huffman · 5 shared papers, 2021–2026 · strength 0.79
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- Victoire Cardot‐Ruffino · 5 shared papers, 2023–2026 · strength 0.77
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Kimberly J. Perez · 5 shared papers, 2022–2026 · strength 0.77
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- Andressa Dias Costa · 5 shared papers, 2022–2024 · strength 0.70
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- Paper Multicenter randomized controlled trial of neoadjuvant chemoradiotherapy alone or in combination with pembrolizumab in … DOI 10.1136/jitc-2023-007586↗
- Thomas E. Clancy · 4 shared papers, 2022–2026 · strength 0.65
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Lestat R. Ali · 4 shared papers, 2023–2026 · strength 0.63
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Douglas Adam Rubinson · 4 shared papers, 2022–2026 · strength 0.62
- Paper Dendritic cell redundancy enables priming of anti-tumor CD4+ T cells in pancreatic cancer. DOI 10.1016/j.ccell.2026.04.005↗
- Paper CD4 T cells convert transient responses to KRAS inhibition to durable remissions in pancreatic cancer. DOI 10.1016/j.cell.2026.08.045↗
- Paper Blockade of IL1β and PD1 with Combination Chemotherapy Reduces Systemic Myeloid Suppression in Metastatic Pancreatic Ca… DOI 10.1158/2326-6066.cir-23-1073↗
- Brendan D. Parent · 2 shared papers, 2026 · strength 0.54
- Qiang Li · 2 shared papers, 2022–2026 · strength 0.52
- Osama E. Rahma · 3 shared papers, 2023–2024 · strength 0.44
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Multicenter randomized controlled trial of neoadjuvant chemoradiotherapy alone or in combination with pembrolizumab in … DOI 10.1136/jitc-2023-007586↗
- Paper Blockade of IL1β and PD1 with Combination Chemotherapy Reduces Systemic Myeloid Suppression in Metastatic Pancreatic Ca… DOI 10.1158/2326-6066.cir-23-1073↗
- Srivatsan Raghavan · 3 shared papers, 2021–2024 · strength 0.42
- Paper Clinical and Genomic Features of Classical and Basal Transcriptional Subtypes in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-24-1164↗
- Paper Transforming Growth Factor-β Blockade in Pancreatic Cancer Enhances Sensitivity to Combination Chemotherapy. DOI 10.1053/j.gastro.2023.05.038↗
- Paper Opportunities for Utilization of DNA Repair Inhibitors in Homologous Recombination Repair-Deficient and Proficient Panc… DOI 10.1158/1078-0432.ccr-21-1367↗
- Courtney T.S. Kureshi · 2 shared papers, 2026 · strength 0.38
- Chong Zuo · 2 shared papers, 2026 · strength 0.38
- George Molina · 2 shared papers, 2026 · strength 0.38
- Craig Lee Slingluff · 2 shared papers, 2023–2024 · strength 0.30
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Multicenter randomized controlled trial of neoadjuvant chemoradiotherapy alone or in combination with pembrolizumab in … DOI 10.1136/jitc-2023-007586↗
- Matthew H. G. Katz · 2 shared papers, 2023–2024 · strength 0.30
- Paper PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with … DOI 10.1158/1078-0432.ccr-23-1444↗
- Paper Multicenter randomized controlled trial of neoadjuvant chemoradiotherapy alone or in combination with pembrolizumab in … DOI 10.1136/jitc-2023-007586↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.