ResearcherFred Hutch Cancer CenterActive in the last 2 years
Kathleen E. DelGiorno
Matched on ORCID iD 0000-0002-5749-8394; OpenAlex author ID A5078093187; NIH RePORTER profile ID 10626873. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 3, last author 0)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 56
2015: 1 paper · 2016: 2 papers · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: none · 2021: 1 paper · 2022: none · 2023: 2 papers · 2024: 1 paper · 2025: 2 papers · 2026: 1 paper, 1 grant
Papers 12
- Oncogenic KRAS-MAPK signaling sustains a CD73 dependent therapeutic vulnerability in pancreatic cancer Co-author DOI 10.64898/2026.10.20.760559↗
- Tuft cells transdifferentiate to neural-like progenitor cells in the progression of pancreatic cancer. Co-author DOI 10.1016/j.devcel.2024.12.003↗
- Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer Co-author DOI 10.64898/2025.12.04.691935↗
- Editorial: Emerging anti-cancer compounds and immunomodulators for pancreatic cancer treatment. Co-author DOI 10.3389/fonc.2024.1411836↗
- Bone Morphogenetic Protein (BMP) signaling upregulates expression of ID1 and ID3 in pancreatitis and pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2023.09.01.555987↗
- CD73 Is a Critical Immune Checkpoint in a Molecular Subtype of Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-23-0268↗
- Author Correction: Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. Co-author DOI 10.1038/s41586-021-04176-w↗
- Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. Co-author DOI 10.1038/s41586-019-1130-6↗
- Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapy. First author DOI 10.1371/journal.pone.0189051↗
- Interstitial Pressure in Pancreatic Ductal Adenocarcinoma Is Dominated by a Gel-Fluid Phase. First author DOI 10.1016/j.bpj.2016.03.040↗
- Mounting Pressure in the Microenvironment: Fluids, Solids, and Cells in Pancreatic Ductal Adenocarcinoma. First author DOI 10.1053/j.gastro.2016.03.040↗
- RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1016/j.cell.2015.04.048↗
Grants 1
Closest collaborators
The 25 strongest of 56 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jennifer M. Bailey · 2 shared papers, 2024–2026 · strength 0.62
- Stephen H Safe · 1 shared paper, 2024 · strength 0.36
- Urvinder Kaur Sardarni · 1 shared paper, 2026 · strength 0.26
- Khadija Shahab Turabi · 1 shared paper, 2026 · strength 0.26
- Marina Pasca di Magliano · 1 shared paper, 2026 · strength 0.26
- Thomas B. McFall · 1 shared paper, 2026 · strength 0.26
- Nina G. Steele · 1 shared paper, 2025 · strength 0.21
- Daniel James Salas-Escabillas · 1 shared paper, 2025 · strength 0.21
- Megan T. Hoffman · 1 shared paper, 2025 · strength 0.21
- Simone Benitz · 1 shared paper, 2025 · strength 0.21
- Rosalie C. Sears · 1 shared paper, 2025 · strength 0.21
- Brittany L. Allen-Petersen · 1 shared paper, 2025 · strength 0.21
- Howard C. Crawford · 1 shared paper, 2025 · strength 0.21
- Geoffrey Myles Wahl · 3 shared papers, 2017–2021 · strength 0.20
- Paper Author Correction: Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-021-04176-w↗
- Paper Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-019-1130-6↗
- Paper Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapy. DOI 10.1371/journal.pone.0189051↗
- Maya Ridinger-Saison · 3 shared papers, 2017–2021 · strength 0.20
- Paper Author Correction: Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-021-04176-w↗
- Paper Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-019-1130-6↗
- Paper Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapy. DOI 10.1371/journal.pone.0189051↗
- Andrew M. Lowy · 3 shared papers, 2017–2021 · strength 0.20
- Paper Author Correction: Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-021-04176-w↗
- Paper Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-019-1130-6↗
- Paper Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapy. DOI 10.1371/journal.pone.0189051↗
- Ronald M. Evans · 3 shared papers, 2017–2021 · strength 0.20
- Paper Author Correction: Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-021-04176-w↗
- Paper Targeting LIF-mediated paracrine interaction for pancreatic cancer therapy and monitoring. DOI 10.1038/s41586-019-1130-6↗
- Paper Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapy. DOI 10.1371/journal.pone.0189051↗
- H. Carlo Maurer · 1 shared paper, 2023 · strength 0.19
- Megha Raghunathan · 1 shared paper, 2023 · strength 0.19
- Pamela Itkin‐Ansari · 1 shared paper, 2023 · strength 0.19
- Sunil R. Hingorani · 3 shared papers, 2015–2016 · strength 0.19
- Paper Mounting Pressure in the Microenvironment: Fluids, Solids, and Cells in Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2016.03.040↗
- Paper Interstitial Pressure in Pancreatic Ductal Adenocarcinoma Is Dominated by a Gel-Fluid Phase. DOI 10.1016/j.bpj.2016.03.040↗
- Paper RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma. DOI 10.1016/j.cell.2015.04.048↗
- Oliver Gene McDonald · 1 shared paper, 2025 · strength 0.19
- Austin R. Dosch · 1 shared paper, 2025 · strength 0.19
- Vineet Kumar Gupta · 1 shared paper, 2025 · strength 0.19
- Jashodeep Datta · 1 shared paper, 2025 · strength 0.19
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.