ResearcherUniversity of MiamiActive in the last 2 years
Austin R. Dosch
Matched on ORCID iD 0000-0001-9071-1591; OpenAlex author ID A5089529776. Sources: OpenAlex.
Their work
- Papers
- 14 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 30
2018: 1 paper · 2019: 3 papers · 2020: 1 paper · 2021: 2 papers · 2022: 2 papers · 2023: 2 papers · 2024: 1 paper · 2025: 2 papers
Papers 14
- CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Alcoholic Pancreatitis. Co-author DOI 10.1016/j.jcmgh.2025.101606↗
- Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer Co-author DOI 10.64898/2025.12.04.691935↗
- Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancreatic Cancer. First author DOI 10.1158/0008-5472.can-23-1200↗
- Cell-Autonomous Cxcl1 Sustains Tolerogenic Circuitries and Stromal Inflammation via Neutrophil-Derived TNF in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-22-1046↗
- Remodeling of Stromal Immune Microenvironment by Urolithin A Improves Survival with Immune Checkpoint Blockade in Pancreatic Cancer. Co-author DOI 10.1158/2767-9764.crc-22-0329↗
- Distinct mechanisms of innate and adaptive immune regulation underlie poor oncologic outcomes associated with KRAS-TP53 co-alteration in pancreatic cancer. Co-author DOI 10.1038/s41388-022-02368-w↗
- Combined MEK and STAT3 Inhibition Uncovers Stromal Plasticity by Enriching for Cancer-Associated Fibroblasts With Mesenchymal Stem Cell-Like Features to Overco… Co-author DOI 10.1053/j.gastro.2022.07.076↗
- Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1535-7163.mct-19-1043↗
- Targeting Tumor-Stromal IL6/STAT3 Signaling through IL1 Receptor Inhibition in Pancreatic Cancer. Co-author DOI 10.1158/1535-7163.mct-21-0083↗
- Combined Src/EGFR Inhibition Targets STAT3 Signaling and Induces Stromal Remodeling to Improve Survival in Pancreatic Cancer. Co-author DOI 10.1158/1541-7786.mcr-19-0741↗
- Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. Co-author DOI 10.1158/1535-7163.mct-18-0464↗
- Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.26621↗
- New mechanistic insights into modifiable risk factors that promote pancreatic cancer. First author DOI 10.18632/oncotarget.26813↗
- Tobacco Carcinogen-Induced Production of GM-CSF Activates CREB to Promote Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-18-0579↗
Closest collaborators
The 25 strongest of 30 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Nagaraj S. Nagathihalli · 14 shared papers, 2018–2025 · strength 1.84
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Nipun B. Merchant · 13 shared papers, 2018–2025 · strength 1.68
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Siddharth Mehra · 10 shared papers, 2020–2025 · strength 1.43
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Supriya Srinivasan · 9 shared papers, 2018–2025 · strength 1.19
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper New mechanistic insights into modifiable risk factors that promote pancreatic cancer. DOI 10.18632/oncotarget.26813↗
- Iago De Castro Silva · 7 shared papers, 2021–2025 · strength 1.02
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Paper Remodeling of Stromal Immune Microenvironment by Urolithin A Improves Survival with Immune Checkpoint Blockade in Pancr… DOI 10.1158/2767-9764.crc-22-0329↗
- Samara P. Singh · 7 shared papers, 2021–2025 · strength 1.02
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Paper Remodeling of Stromal Immune Microenvironment by Urolithin A Improves Survival with Immune Checkpoint Blockade in Pancr… DOI 10.1158/2767-9764.crc-22-0329↗
- Jashodeep Datta · 5 shared papers, 2022–2024 · strength 0.71
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Paper Remodeling of Stromal Immune Microenvironment by Urolithin A Improves Survival with Immune Checkpoint Blockade in Pancr… DOI 10.1158/2767-9764.crc-22-0329↗
- Paper Combined MEK and STAT3 Inhibition Uncovers Stromal Plasticity by Enriching for Cancer-Associated Fibroblasts With Mesen… DOI 10.1053/j.gastro.2022.07.076↗
- Haleh Amirian · 4 shared papers, 2023–2025 · strength 0.69
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Michael Nathan VanSaun · 5 shared papers, 2018–2025 · strength 0.57
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. DOI 10.18632/oncotarget.26621↗
- Anna Bianchi · 3 shared papers, 2023–2025 · strength 0.50
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Tumor Cell-Intrinsic p38 MAPK Signaling Promotes IL1α-Mediated Stromal Inflammation and Therapeutic Resistance in Pancr… DOI 10.1158/0008-5472.can-23-1200↗
- Paper Cell-Autonomous Cxcl1 Sustains Tolerogenic Circuitries and Stromal Inflammation via Neutrophil-Derived TNF in Pancreati… DOI 10.1158/2159-8290.cd-22-1046↗
- Jashodeep Datta · 3 shared papers, 2021–2025 · strength 0.50
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Targeting CREB remodels the immune microenvironment to enhance immunotherapy responses in pancreatic cancer DOI 10.64898/2025.12.04.691935↗
- Paper Targeting Tumor-Stromal IL6/STAT3 Signaling through IL1 Receptor Inhibition in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-21-0083↗
- Jason A. Castellanos · 4 shared papers, 2018–2021 · strength 0.37
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. DOI 10.18632/oncotarget.26621↗
- Paper Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-18-0464↗
- Tulasigeri M. Totiger · 3 shared papers, 2018–2025 · strength 0.35
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-18-0464↗
- Paper Tobacco Carcinogen-Induced Production of GM-CSF Activates CREB to Promote Pancreatic Cancer. DOI 10.1158/0008-5472.can-18-0579↗
- Peter J. Hosein · 2 shared papers, 2022 · strength 0.24
- Paper Combined MEK and STAT3 Inhibition Uncovers Stromal Plasticity by Enriching for Cancer-Associated Fibroblasts With Mesen… DOI 10.1053/j.gastro.2022.07.076↗
- Paper Distinct mechanisms of innate and adaptive immune regulation underlie poor oncologic outcomes associated with KRAS-TP53… DOI 10.1038/s41388-022-02368-w↗
- Christine Isabelle Rafie · 2 shared papers, 2022–2023 · strength 0.24
- Paper Cell-Autonomous Cxcl1 Sustains Tolerogenic Circuitries and Stromal Inflammation via Neutrophil-Derived TNF in Pancreati… DOI 10.1158/2159-8290.cd-22-1046↗
- Paper Distinct mechanisms of innate and adaptive immune regulation underlie poor oncologic outcomes associated with KRAS-TP53… DOI 10.1038/s41388-022-02368-w↗
- Nilesh Deshpande · 2 shared papers, 2022–2023 · strength 0.24
- Paper Cell-Autonomous Cxcl1 Sustains Tolerogenic Circuitries and Stromal Inflammation via Neutrophil-Derived TNF in Pancreati… DOI 10.1158/2159-8290.cd-22-1046↗
- Paper Distinct mechanisms of innate and adaptive immune regulation underlie poor oncologic outcomes associated with KRAS-TP53… DOI 10.1038/s41388-022-02368-w↗
- Brent A. Willobee · 2 shared papers, 2020–2021 · strength 0.23
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Combined Src/EGFR Inhibition Targets STAT3 Signaling and Induces Stromal Remodeling to Improve Survival in Pancreatic C… DOI 10.1158/1541-7786.mcr-19-0741↗
- Kathleen E. DelGiorno · 1 shared paper, 2025 · strength 0.19
- Oliver Gene McDonald · 1 shared paper, 2025 · strength 0.19
- Vineet Kumar Gupta · 1 shared paper, 2025 · strength 0.19
- Chanjuan Shi · 2 shared papers, 2018–2019 · strength 0.17
- Eli Gilboa · 1 shared paper, 2023 · strength 0.16
- William L. Hwang · 1 shared paper, 2023 · strength 0.12
- Ifeanyichukwu Chinedu Ogobuiro · 1 shared paper, 2023 · strength 0.12
- Carina Shiau · 1 shared paper, 2023 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.