ResearcherUniversity of MiamiActive in the last 2 years
Michael Nathan VanSaun
Matched on ORCID iD 0000-0002-2538-8107; OpenAlex author ID A5036458814; NIH RePORTER profile ID 9770145. Sources: OpenAlex.
Their work
- Papers
- 15 (first author 0, last author 6)
- Trials
- 0
- Grants
- 2
- Labs led
- 2
- Collaborators
- 36
2015: 1 paper · 2016: none · 2017: none · 2018: 2 papers · 2019: 2 papers · 2020: none · 2021: 1 paper · 2022: 2 papers · 2023: 2 papers, 1 grant · 2024: none · 2025: 5 papers, 1 grant
Papers 15
- CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Alcoholic Pancreatitis. Co-author DOI 10.1016/j.jcmgh.2025.101606↗
- DUSP6 is upregulated in metastasis and influences migration and metabolism in pancreatic cancer cells. Co-author DOI 10.1038/s41598-025-12967-8↗
- Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancreatic cancer. Last author DOI 10.1136/jitc-2024-010057↗
- Editor's Note: Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-25-1304↗
- Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. Last author DOI 10.3390/cancers17071152↗
- SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. Last author DOI 10.1007/s12079-022-00691-1↗
- Anticancer Activity of Novel Difluorinated Curcumin Analog and Its Inclusion Complex with 2-Hydroxypropyl-β-Cyclodextrin against Pancreatic Cancer. Co-author DOI 10.3390/ijms24076336↗
- Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. Last author DOI 10.1038/s41419-022-04572-8↗
- Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. Last author DOI 10.1038/s41419-022-04615-0↗
- Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1535-7163.mct-19-1043↗
- Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. Co-author DOI 10.1158/1535-7163.mct-18-0464↗
- Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.26621↗
- Tobacco Carcinogen-Induced Production of GM-CSF Activates CREB to Promote Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-18-0579↗
- Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-18-0634↗
- Modulation of the leptin receptor mediates tumor growth and migration of pancreatic cancer cells. Last author DOI 10.1371/journal.pone.0126686↗
Grants 2
Closest collaborators
The 25 strongest of 36 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Bailey A. Bye · 6 shared papers, 2022–2025 · strength 1.21
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper DUSP6 is upregulated in metastasis and influences migration and metabolism in pancreatic cancer cells. DOI 10.1038/s41598-025-12967-8↗
- R. McKinnon Walsh · 5 shared papers, 2022–2025 · strength 1.03
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper DUSP6 is upregulated in metastasis and influences migration and metabolism in pancreatic cancer cells. DOI 10.1038/s41598-025-12967-8↗
- Appolinaire A. Olou · 5 shared papers, 2022–2025 · strength 0.98
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. DOI 10.1007/s12079-022-00691-1↗
- Mariana T. Ruckert · 5 shared papers, 2022–2025 · strength 0.94
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper DUSP6 is upregulated in metastasis and influences migration and metabolism in pancreatic cancer cells. DOI 10.1038/s41598-025-12967-8↗
- Paper SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. DOI 10.1007/s12079-022-00691-1↗
- Nipun B. Merchant · 7 shared papers, 2018–2025 · strength 0.90
- Paper Editor's Note: Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreat… DOI 10.1158/0008-5472.can-25-1304↗
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Nagaraj S. Nagathihalli · 7 shared papers, 2018–2025 · strength 0.90
- Paper Editor's Note: Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreat… DOI 10.1158/0008-5472.can-25-1304↗
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Jason A. Castellanos · 6 shared papers, 2018–2025 · strength 0.70
- Paper Editor's Note: Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreat… DOI 10.1158/0008-5472.can-25-1304↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. DOI 10.18632/oncotarget.26621↗
- Austin R. Dosch · 5 shared papers, 2018–2025 · strength 0.57
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. DOI 10.18632/oncotarget.26621↗
- Chanjuan Shi · 4 shared papers, 2018–2025 · strength 0.50
- Paper Editor's Note: Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreat… DOI 10.1158/0008-5472.can-25-1304↗
- Paper Src kinase inhibition restores E-cadherin expression in dasatinib-sensitive pancreatic cancer cells. DOI 10.18632/oncotarget.26621↗
- Paper Inverse Correlation of STAT3 and MEK Signaling Mediates Resistance to RAS Pathway Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-18-0634↗
- Supriya Srinivasan · 4 shared papers, 2018–2025 · strength 0.47
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Paper Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-18-0464↗
- Shrikant Anant · 3 shared papers, 2022–2023 · strength 0.45
- Paper Anticancer Activity of Novel Difluorinated Curcumin Analog and Its Inclusion Complex with 2-Hydroxypropyl-β-Cyclodextri… DOI 10.3390/ijms24076336↗
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- Paper Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04615-0↗
- Tulasigeri M. Totiger · 3 shared papers, 2018–2025 · strength 0.35
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Urolithin A, a Novel Natural Compound to Target PI3K/AKT/mTOR Pathway in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-18-0464↗
- Paper Tobacco Carcinogen-Induced Production of GM-CSF Activates CREB to Promote Pancreatic Cancer. DOI 10.1158/0008-5472.can-18-0579↗
- Prasad R. Dandawate · 2 shared papers, 2023 · strength 0.35
- Siddharth Mehra · 2 shared papers, 2021–2025 · strength 0.32
- Paper CREB Drives Acinar to Ductal Cells Reprogramming and Promotes Pancreatic Cancer Progression in Preclinical Models of Al… DOI 10.1016/j.jcmgh.2025.101606↗
- Paper Combined Blockade of MEK and CDK4/6 Pathways Induces Senescence to Improve Survival in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1535-7163.mct-19-1043↗
- Sharon Manley · 2 shared papers, 2022 · strength 0.29
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- Paper Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04615-0↗
- Xi Chen · 1 shared paper, 2025 · strength 0.25
- Emily L. Lasse Opsahl · 1 shared paper, 2025 · strength 0.23
- Marina Pasca di Magliano · 1 shared paper, 2025 · strength 0.23
- Jiaqi Shi · 1 shared paper, 2025 · strength 0.23
- Vanessa S. Silveira · 1 shared paper, 2025 · strength 0.23
- Costas Andreas Lyssiotis · 1 shared paper, 2025 · strength 0.23
- Filip Bednar · 1 shared paper, 2025 · strength 0.23
- Anna Bianchi · 1 shared paper, 2025 · strength 0.20
- Iago De Castro Silva · 1 shared paper, 2025 · strength 0.20
- Jashodeep Datta · 1 shared paper, 2025 · strength 0.20
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.