ResearcherUniversity of MiamiActive in the last 2 years
Appolinaire A. Olou
Matched on ORCID iD 0000-0002-6754-2969; OpenAlex author ID A5057693072. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 9
2022: 2 papers · 2023: 1 paper · 2024: none · 2025: 3 papers · 2026: 1 paper
Papers 7
- Comprehensive genome sequencing associates elevated mitochondrial DNA and homoplasmy with early stage I of pancreatic cancer. Co-author DOI 10.1016/j.isci.2026.115453↗
- EV DNA from pancreatic cancer patient-derived cells harbors molecular, coding, non-coding signatures and mutational hotspots. Co-author DOI 10.1038/s42003-025-07567-1↗
- Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancreatic cancer. Co-author DOI 10.1136/jitc-2024-010057↗
- Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. Co-author DOI 10.3390/cancers17071152↗
- SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. Co-author DOI 10.1007/s12079-022-00691-1↗
- Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. First author DOI 10.1038/s41419-022-04572-8↗
- Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. First author DOI 10.1038/s41419-022-04615-0↗
Closest collaborators
The 9 strongest of 9 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Bailey A. Bye · 5 shared papers, 2022–2025 · strength 0.98
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. DOI 10.1007/s12079-022-00691-1↗
- Michael Nathan VanSaun · 5 shared papers, 2022–2025 · strength 0.98
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. DOI 10.1007/s12079-022-00691-1↗
- R. McKinnon Walsh · 4 shared papers, 2022–2025 · strength 0.80
- Paper Combined Omipalisib and MAPK Inhibition Suppress PDAC Growth. DOI 10.3390/cancers17071152↗
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- M. Rohan Fernando · 2 shared papers, 2025–2026 · strength 0.75
- Paper Comprehensive genome sequencing associates elevated mitochondrial DNA and homoplasmy with early stage I of pancreatic c… DOI 10.1016/j.isci.2026.115453↗
- Paper EV DNA from pancreatic cancer patient-derived cells harbors molecular, coding, non-coding signatures and mutational hot… DOI 10.1038/s42003-025-07567-1↗
- Mariana T. Ruckert · 4 shared papers, 2022–2025 · strength 0.71
- Paper Depletion of tumor-derived CXCL5 improves T cell infiltration and anti-PD-1 therapy response in an obese model of pancr… DOI 10.1136/jitc-2024-010057↗
- Paper SHP2 regulates adipose maintenance and adipocyte-pancreatic cancer cell crosstalk via PDHA1. DOI 10.1007/s12079-022-00691-1↗
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- Sharon Manley · 2 shared papers, 2022 · strength 0.29
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- Paper Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04615-0↗
- Shrikant Anant · 2 shared papers, 2022 · strength 0.29
- Paper Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04572-8↗
- Paper Correction: Synthetic adiponectin-receptor agonist, AdipoRon, induces glycolytic dependence in pancreatic cancer cells. DOI 10.1038/s41419-022-04615-0↗
- Danny R. Welch · 1 shared paper, 2025 · strength 0.25
- Prasad R. Dandawate · 1 shared paper, 2023 · strength 0.18
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.