ResearcherMedical College of WisconsinActive in the last 2 years
Thomas B. McFall
Matched on ORCID iD 0000-0002-5284-6994; OpenAlex author ID A5066121818. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 37
2024: 2 papers · 2025: none · 2026: 6 papers
Papers 8
- MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with KRAS alterations. Co-author DOI 10.1093/oncolo/oyag040↗
- Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenoca… Last author DOI 10.1158/0008-5472.can-25-0018↗
- KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvironment. Co-author DOI 10.1158/0008-5472.can-25-2630↗
- Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. Last author DOI 10.1158/0008-5472.can-26-1181↗
- The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed Patient Selection. Co-author DOI 10.3390/cancers18203376↗
- Oncogenic KRAS-MAPK signaling sustains a CD73 dependent therapeutic vulnerability in pancreatic cancer Co-author DOI 10.64898/2026.10.20.760559↗
- Transcriptomic-Based Microenvironment Classification Reveals Precision Medicine Strategies for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1053/j.gastro.2024.01.028↗
- Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, a case series. Co-author DOI 10.1177/17588359241253113↗
Closest collaborators
The 25 strongest of 37 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mandana Kamgar · 6 shared papers, 2024–2026 · strength 1.37
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Douglas B. Evans · 5 shared papers, 2024–2026 · strength 1.10
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Razelle Kurzrock · 5 shared papers, 2024–2026 · strength 1.01
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- William Adrian Hall · 4 shared papers, 2024–2026 · strength 0.89
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Yongwoo David Seo · 3 shared papers, 2026 · strength 0.77
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Susan Tsai · 4 shared papers, 2024–2026 · strength 0.74
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Mohammed Aldakkak · 3 shared papers, 2026 · strength 0.68
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- G. Aaron Hobbs · 3 shared papers, 2026 · strength 0.62
- Paper KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvir… DOI 10.1158/0008-5472.can-25-2630↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Kathleen K. Christians · 3 shared papers, 2024–2026 · strength 0.54
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Paper Transcriptomic-Based Microenvironment Classification Reveals Precision Medicine Strategies for Pancreatic Ductal Adenoc… DOI 10.1053/j.gastro.2024.01.028↗
- Sam Z. Thalji · 2 shared papers, 2026 · strength 0.48
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Rachel A. Burge · 2 shared papers, 2026 · strength 0.42
- Paper KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvir… DOI 10.1158/0008-5472.can-25-2630↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Channing J. Der · 2 shared papers, 2026 · strength 0.42
- Paper KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvir… DOI 10.1158/0008-5472.can-25-2630↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Christabelle Rajesh · 2 shared papers, 2026 · strength 0.41
- Paper KRASG12R-Mutant Pancreatic Cancer Features Limited ERK/MAPK Transcriptional Activity and a Distinctive Tumor Microenvir… DOI 10.1158/0008-5472.can-25-2630↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Nikki K. Lytle · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Daniel Dorbin · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Ben George · 2 shared papers, 2024–2026 · strength 0.38
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Beth E. Erickson · 2 shared papers, 2024–2026 · strength 0.38
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Kshitij S. Gaur · 1 shared paper, 2026 · strength 0.36
- Elizabeth Diane Auckley · 1 shared paper, 2026 · strength 0.27
- Kathleen E. DelGiorno · 1 shared paper, 2026 · strength 0.26
- Urvinder Kaur Sardarni · 1 shared paper, 2026 · strength 0.26
- Khadija Shahab Turabi · 1 shared paper, 2026 · strength 0.26
- Marina Pasca di Magliano · 1 shared paper, 2026 · strength 0.26
- Jennifer M. Bailey · 1 shared paper, 2026 · strength 0.26
- Lu Han · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.