ResearcherUniversity of Nebraska–LincolnActive in the last 2 years
Razelle Kurzrock
Matched on ORCID iD 0000-0003-4110-1214; OpenAlex author ID A5003304495, A5053481267. Sources: ClinicalTrials.gov.
Their work
- Papers
- 15 (first author 2, last author 5)
- Trials
- 3 (principal investigator on 2)
- Grants
- 0
- Labs led
- 1
- Collaborators
- 58
2011: 1 paper · 2012: none · 2013: none · 2014: none · 2015: 1 paper · 2016: 1 paper · 2017: none · 2018: none · 2019: 1 paper · 2020: none · 2021: 2 papers, 1 trial · 2022: 1 trial · 2023: 2 papers · 2024: 3 papers, 1 trial · 2025: none · 2026: 4 papers
Papers 15
- Advancing precision medicine in pancreatic cancer using novel biomarkers and clinical targets. Co-author DOI 10.1016/j.mam.2026.101476↗
- MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with KRAS alterations. Co-author DOI 10.1093/oncolo/oyag040↗
- Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenoca… Co-author DOI 10.1158/0008-5472.can-25-0018↗
- Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-26-1181↗
- Pan-cancer analysis of TIM-3 transcriptomic expression reveals high levels in pancreatic cancer and interpatient heterogeneity. First author DOI 10.1002/cam4.6844↗
- Transcriptomic-Based Microenvironment Classification Reveals Precision Medicine Strategies for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1053/j.gastro.2024.01.028↗
- Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, a case series. Co-author DOI 10.1177/17588359241253113↗
- Personalized matched targeted therapy in advanced pancreatic cancer: a pilot cohort analysis. Last author DOI 10.1038/s41525-022-00346-5↗
- Development of a blood-based extracellular vesicle classifier for detection of early-stage pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s43856-023-00351-4↗
- SWI/SNF complex alterations as a biomarker of immunotherapy efficacy in pancreatic cancer. Last author DOI 10.1172/jci.insight.150453↗
- Immune profiling and immunotherapeutic targets in pancreatic cancer. Co-author DOI 10.21037/atm-20-1076↗
- Clinical correlates of blood-derived circulating tumor DNA in pancreatic cancer. Last author DOI 10.1186/s13045-019-0824-4↗
- The effects of curcumin (diferuloylmethane) on body composition of patients with advanced pancreatic cancer. Last author DOI 10.18632/oncotarget.7773↗
- Molecular landscape of pancreatic cancer: implications for current clinical trials. First author DOI 10.18632/oncotarget.2972↗
- Phase 1 clinical trials in 83 patients with pancreatic cancer: The M. D. Anderson Cancer Center experience. Last author DOI 10.1002/cncr.25346↗
Trials 3
- Molecular Profile-related Individualized Targeted Therapy in Resected Pancreatic Cancer With High-Risk of Cancer Recurrence Principal investigator NCT06228599↗
- Investigation of Profile-related Evidence Determining Individualized Cancer Therapy for Patients With Aggressive Malignancies and Poor Prognoses Principal investigator NCT05674825↗
- The Registry of Oncology Outcomes Associated With Testing and Treatment Study chair NCT04028479↗
Closest collaborators
The 25 strongest of 58 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mandana Kamgar · 5 shared papers, 1 shared trial, 2024–2026 · strength 1.55
- Trial Molecular Profile-related Individualized Targeted Therapy in Resected Pancreatic Cancer With High-Risk of Cancer Recurr… NCT06228599↗
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Thomas B. McFall · 5 shared papers, 2024–2026 · strength 1.01
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Shumei Kato · 5 shared papers, 2019–2024 · strength 0.76
- Paper Pan-cancer analysis of TIM-3 transcriptomic expression reveals high levels in pancreatic cancer and interpatient hetero… DOI 10.1002/cam4.6844↗
- Paper Personalized matched targeted therapy in advanced pancreatic cancer: a pilot cohort analysis. DOI 10.1038/s41525-022-00346-5↗
- Paper SWI/SNF complex alterations as a biomarker of immunotherapy efficacy in pancreatic cancer. DOI 10.1172/jci.insight.150453↗
- Douglas B. Evans · 4 shared papers, 2024–2026 · strength 0.74
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Susan Tsai · 4 shared papers, 2024–2026 · strength 0.74
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Mohammed Aldakkak · 3 shared papers, 2026 · strength 0.68
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Douglas B. Evans · 1 shared trial, 2024 · strength 0.54
- William Adrian Hall · 3 shared papers, 2024–2026 · strength 0.54
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Paper Transcriptomic-Based Microenvironment Classification Reveals Precision Medicine Strategies for Pancreatic Ductal Adenoc… DOI 10.1053/j.gastro.2024.01.028↗
- Kathleen K. Christians · 3 shared papers, 2024–2026 · strength 0.54
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Paper Transcriptomic-Based Microenvironment Classification Reveals Precision Medicine Strategies for Pancreatic Ductal Adenoc… DOI 10.1053/j.gastro.2024.01.028↗
- Hui-Zi Chen · 1 shared trial, 2022 · strength 0.49
- Sam Z. Thalji · 2 shared papers, 2026 · strength 0.48
- Paper MEK inhibitor-based genomically matched combinatorial targeted therapies in metastatic pancreatic adenocarcinoma with K… DOI 10.1093/oncolo/oyag040↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Nikki K. Lytle · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Yongwoo David Seo · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Daniel Dorbin · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- G. Aaron Hobbs · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Ben George · 2 shared papers, 2024–2026 · strength 0.38
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Beth E. Erickson · 2 shared papers, 2024–2026 · strength 0.38
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Chemotherapy-free treatment targeting fusions and driver mutations in KRAS wild-type pancreatic ductal adenocarcinoma, … DOI 10.1177/17588359241253113↗
- Gregory P. Botta · 2 shared papers, 2021–2023 · strength 0.34
- Hitendra Patel · 2 shared papers, 2019–2023 · strength 0.29
- Jennifer Johnson · 1 shared trial, 2021 · strength 0.29
- Vivek Subbiah · 1 shared trial, 2021 · strength 0.29
- Raymond Bergan · 1 shared trial, 2021 · strength 0.29
- Elizabeth Diane Auckley · 1 shared paper, 2026 · strength 0.27
- Andrew Karam · 1 shared paper, 2026 · strength 0.26
- Divya Murthy · 1 shared paper, 2026 · strength 0.26
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.