ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Yongwoo David Seo
Matched on ORCID iD 0000-0001-6837-5018; OpenAlex author ID A5073658575. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 2, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 44
2017: 2 papers · 2018: none · 2019: 1 paper · 2020: none · 2021: none · 2022: 1 paper · 2023: 2 papers · 2024: 1 paper · 2025: 1 paper · 2026: 3 papers
Papers 11
- Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenoca… Co-author DOI 10.1158/0008-5472.can-25-0018↗
- Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-26-1181↗
- The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed Patient Selection. Last author DOI 10.3390/cancers18203376↗
- The Landmark Series: The Future of Pancreatic Cancer Clinical Trials. Co-author DOI 10.1245/s10434-024-16840-2↗
- Intratumoral T-cell receptor repertoire composition predicts overall survival in patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.1080/2162402x.2024.2320411↗
- Quality of superior mesenteric and hepatic artery dissection in robotic pancreatoduodenectomy for pancreatic cancer. First author DOI 10.1002/jhbp.1218↗
- From bugs to drugs: Bacterial 3-IAA enhances efficacy of chemotherapy in pancreatic cancer. Co-author DOI 10.1016/j.xcrm.2023.101039↗
- Preoperative therapy for pancreatic adenocarcinoma-precision beyond anatomy. Co-author DOI 10.1002/cncr.34273↗
- Mobilization of CD8+ T Cells via CXCR4 Blockade Facilitates PD-1 Checkpoint Therapy in Human Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-19-0081↗
- T-cell programming in pancreatic adenocarcinoma: a review. Co-author DOI 10.1038/cgt.2016.66↗
- Long-lived pancreatic ductal adenocarcinoma slice cultures enable precise study of the immune microenvironment. First author DOI 10.1080/2162402x.2017.1333210↗
Closest collaborators
The 25 strongest of 44 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Matthew H. G. Katz · 3 shared papers, 2022–2025 · strength 0.93
- Paper The Landmark Series: The Future of Pancreatic Cancer Clinical Trials. DOI 10.1245/s10434-024-16840-2↗
- Paper Preoperative therapy for pancreatic adenocarcinoma-precision beyond anatomy. DOI 10.1002/cncr.34273↗
- Paper Quality of superior mesenteric and hepatic artery dissection in robotic pancreatoduodenectomy for pancreatic cancer. DOI 10.1002/jhbp.1218↗
- Douglas B. Evans · 3 shared papers, 2026 · strength 0.77
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Thomas B. McFall · 3 shared papers, 2026 · strength 0.77
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Mandana Kamgar · 3 shared papers, 2026 · strength 0.77
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- William Adrian Hall · 2 shared papers, 2026 · strength 0.56
- Paper The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed P… DOI 10.3390/cancers18203376↗
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Venu Gopal Pillarisetty · 4 shared papers, 2017–2024 · strength 0.49
- Paper Intratumoral T-cell receptor repertoire composition predicts overall survival in patients with pancreatic ductal adenoc… DOI 10.1080/2162402x.2024.2320411↗
- Paper T-cell programming in pancreatic adenocarcinoma: a review. DOI 10.1038/cgt.2016.66↗
- Paper Mobilization of CD8+ T Cells via CXCR4 Blockade Facilitates PD-1 Checkpoint Therapy in Human Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-0081↗
- Rebecca Anne Snyder · 1 shared paper, 2025 · strength 0.42
- Nikki K. Lytle · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Daniel Dorbin · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Mohammed Aldakkak · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Susan Tsai · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- G. Aaron Hobbs · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Razelle Kurzrock · 2 shared papers, 2026 · strength 0.41
- Paper Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapi… DOI 10.1158/0008-5472.can-25-0018↗
- Paper Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer. DOI 10.1158/0008-5472.can-26-1181↗
- Jennifer Ann Wargo · 1 shared paper, 2023 · strength 0.39
- Kshitij S. Gaur · 1 shared paper, 2026 · strength 0.36
- Rachel A. Burge · 1 shared paper, 2026 · strength 0.21
- Sam Z. Thalji · 1 shared paper, 2026 · strength 0.21
- Ben George · 1 shared paper, 2026 · strength 0.21
- Beth E. Erickson · 1 shared paper, 2026 · strength 0.21
- Channing J. Der · 1 shared paper, 2026 · strength 0.21
- Kathleen K. Christians · 1 shared paper, 2026 · strength 0.21
- Brian F Volkman · 1 shared paper, 2026 · strength 0.20
- Christabelle Rajesh · 1 shared paper, 2026 · strength 0.20
- Graham D. Hogg · 1 shared paper, 2024 · strength 0.20
- William G. Hawkins · 1 shared paper, 2024 · strength 0.20
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.