ResearcherBaylor College of MedicineActive in the last 2 years
Divya Murthy
Matched on ORCID iD 0000-0001-6492-1894; OpenAlex author ID A5082370633. Sources: OpenAlex.
Their work
- Papers
- 12
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 46
2017: 2 papers · 2018: 2 papers · 2019: none · 2020: 1 paper · 2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers · 2025: none · 2026: 1 paper
Papers 12
- Advancing precision medicine in pancreatic cancer using novel biomarkers and clinical targets. Co-author DOI 10.1016/j.mam.2026.101476↗
- Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 axis. Co-author DOI 10.1038/s41556-024-01372-4↗
- Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 … Co-author DOI 10.1038/s41556-024-01417-8↗
- The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. Co-author DOI 10.1073/pnas.2315509121↗
- PTGES Expression Is Associated with Metabolic and Immune Reprogramming in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/ijms24087304↗
- CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. Co-author DOI 10.1038/s41388-021-02132-6↗
- Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. Co-author DOI 10.1053/j.gastro.2021.06.045↗
- JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.07.025↗
- Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. Co-author DOI 10.1007/978-1-4939-7665-2_8↗
- Phosphoinositide 3-Kinase Signaling Pathway in Pancreatic Ductal Adenocarcinoma Progression, Pathogenesis, and Therapeutics. Co-author DOI 10.3389/fphys.2018.00335↗
- GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. Co-author DOI 10.1016/j.canlet.2017.04.029↗
- MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.06.004↗
Closest collaborators
The 25 strongest of 46 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kuldeep Singh Attri · 9 shared papers, 2017–2024 · strength 1.36
- Paper PTGES Expression Is Associated with Metabolic and Immune Reprogramming in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/ijms24087304↗
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Pankaj Kumar Singh · 10 shared papers, 2017–2024 · strength 1.14
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Surendra Kumar Shukla · 8 shared papers, 2017–2024 · strength 0.90
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ryan J. King · 7 shared papers, 2017–2024 · strength 0.71
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Nina V. Chaika · 6 shared papers, 2017–2024 · strength 0.71
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ravi Kumar Thakur · 5 shared papers, 2021–2024 · strength 0.67
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Aneesha Dasgupta · 6 shared papers, 2017–2024 · strength 0.63
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Michael Anthony Hollingsworth · 5 shared papers, 2017–2024 · strength 0.63
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Scott E. Mulder · 5 shared papers, 2020–2024 · strength 0.60
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Kamiya Mehla · 5 shared papers, 2017–2024 · strength 0.59
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Chunbo He · 4 shared papers, 2021–2024 · strength 0.48
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Jaime Abrego · 5 shared papers, 2017–2022 · strength 0.46
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. DOI 10.1007/978-1-4939-7665-2_8↗
- Paul M. Grandgenett · 4 shared papers, 2017–2024 · strength 0.44
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Enza Vernucci · 5 shared papers, 2017–2022 · strength 0.43
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. DOI 10.1007/978-1-4939-7665-2_8↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Tuo Hu · 3 shared papers, 2021–2024 · strength 0.36
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Robert Powers · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Teklab Gebregiworgis · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Rosa F. Hwang · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ajay Jain · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Vikrant Rai · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Joshua J. Souchek · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Sai Sundeep Kollala · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Barish H. Edil · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kathryn Elaine Wellen · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Amarnath (Amar) Natarajan · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.