ResearcherUniversity of Nebraska Medical CenterActive in the last 2 years
Ryan J. King
Matched on ORCID iD 0000-0003-2081-3327; OpenAlex author ID A5029009402. Sources: OpenAlex.
Their work
- Papers
- 13
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 44
2015: 1 paper · 2016: none · 2017: 4 papers · 2018: none · 2019: 1 paper · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper · 2023: none · 2024: 2 papers · 2025: none · 2026: 1 paper
Papers 13
- Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. Co-author DOI 10.1186/s12943-025-02340-8↗
- Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 axis. Co-author DOI 10.1038/s41556-024-01372-4↗
- Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 … Co-author DOI 10.1038/s41556-024-01417-8↗
- CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. Co-author DOI 10.1038/s41388-021-02132-6↗
- Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. Co-author DOI 10.1053/j.gastro.2021.06.045↗
- Macrophages potentiate STAT3 signaling in skeletal muscles and regulate pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.04.017↗
- JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.07.025↗
- Evaluating the Metabolic Alterations in Pancreatic Cancer. Co-author DOI 10.1007/978-1-4939-8879-2_20↗
- GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. Co-author DOI 10.1016/j.canlet.2017.04.029↗
- MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.06.004↗
- De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-16-3062↗
- MUC1-Mediated Metabolic Alterations Regulate Response to Radiotherapy in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-17-1151↗
- Silibinin-mediated metabolic reprogramming attenuates pancreatic cancer-induced cachexia and tumor growth. Co-author DOI 10.18632/oncotarget.5843↗
Closest collaborators
The 25 strongest of 44 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Surendra Kumar Shukla · 13 shared papers, 2015–2026 · strength 1.33
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Pankaj Kumar Singh · 12 shared papers, 2015–2024 · strength 1.11
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper Evaluating the Metabolic Alterations in Pancreatic Cancer. DOI 10.1007/978-1-4939-8879-2_20↗
- Aneesha Dasgupta · 10 shared papers, 2015–2024 · strength 0.96
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper Evaluating the Metabolic Alterations in Pancreatic Cancer. DOI 10.1007/978-1-4939-8879-2_20↗
- Kamiya Mehla · 7 shared papers, 2015–2026 · strength 0.78
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Michael Anthony Hollingsworth · 6 shared papers, 2017–2026 · strength 0.77
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kuldeep Singh Attri · 7 shared papers, 2017–2024 · strength 0.75
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Divya Murthy · 7 shared papers, 2017–2024 · strength 0.71
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Chunbo He · 5 shared papers, 2021–2026 · strength 0.71
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ravi Kumar Thakur · 5 shared papers, 2021–2026 · strength 0.71
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Nina V. Chaika · 8 shared papers, 2015–2024 · strength 0.69
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Scott E. Mulder · 5 shared papers, 2020–2024 · strength 0.60
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Enza Vernucci · 8 shared papers, 2015–2022 · strength 0.59
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Macrophages potentiate STAT3 signaling in skeletal muscles and regulate pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.04.017↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Tuo Hu · 4 shared papers, 2021–2026 · strength 0.59
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paul M. Grandgenett · 5 shared papers, 2017–2024 · strength 0.55
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Venugopal Gunda · 7 shared papers, 2015–2022 · strength 0.52
- Paper Evaluating the Metabolic Alterations in Pancreatic Cancer. DOI 10.1007/978-1-4939-8879-2_20↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. DOI 10.1016/j.canlet.2017.04.029↗
- Sai Sundeep Kollala · 3 shared papers, 2024–2026 · strength 0.50
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Jaime Abrego · 6 shared papers, 2017–2022 · strength 0.47
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. DOI 10.1016/j.canlet.2017.04.029↗
- Joshua J. Souchek · 4 shared papers, 2015–2024 · strength 0.38
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1-Mediated Metabolic Alterations Regulate Response to Radiotherapy in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-17-1151↗
- Spas Dimitrov Markov · 2 shared papers, 2020–2026 · strength 0.33
- Robert Powers · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Teklab Gebregiworgis · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Rosa F. Hwang · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ajay Jain · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Vikrant Rai · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Barish H. Edil · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.