ResearcherUniversity of Oklahoma Health Sciences CenterActive in the last 2 years
Ravi Kumar Thakur
Matched on ORCID iD 0000-0003-2505-9757; OpenAlex author ID A5072304509, A5140260118. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 38
2021: 2 papers · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers · 2025: 1 paper · 2026: 2 papers
Papers 10
- KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-1984↗
- Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. Co-author DOI 10.1186/s12943-025-02340-8↗
- Tumor-stromal metabolic crosstalk in pancreatic cancer. Co-author DOI 10.1016/j.tcb.2025.04.007↗
- Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 axis. Co-author DOI 10.1038/s41556-024-01372-4↗
- Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 … Co-author DOI 10.1038/s41556-024-01417-8↗
- The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. Co-author DOI 10.1073/pnas.2315509121↗
- ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. First author DOI 10.1016/j.canlet.2022.215981↗
- CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. Co-author DOI 10.1038/s41388-021-02132-6↗
- Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. Co-author DOI 10.1053/j.gastro.2021.06.045↗
- Molecular Subtypes of Pancreatic Cancer: A Proteomics Approach. Co-author DOI 10.1158/1078-0432.ccr-21-0640↗
Closest collaborators
The 25 strongest of 38 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Pankaj Kumar Singh · 9 shared papers, 2021–2026 · strength 1.70
- Paper Tumor-stromal metabolic crosstalk in pancreatic cancer. DOI 10.1016/j.tcb.2025.04.007↗
- Paper Molecular Subtypes of Pancreatic Cancer: A Proteomics Approach. DOI 10.1158/1078-0432.ccr-21-0640↗
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Kamiya Mehla · 7 shared papers, 2021–2026 · strength 1.36
- Paper Tumor-stromal metabolic crosstalk in pancreatic cancer. DOI 10.1016/j.tcb.2025.04.007↗
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Surendra Kumar Shukla · 8 shared papers, 2021–2026 · strength 1.28
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Chunbo He · 7 shared papers, 2021–2026 · strength 1.09
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. DOI 10.1016/j.canlet.2022.215981↗
- Nina V. Chaika · 6 shared papers, 2021–2024 · strength 0.84
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. DOI 10.1016/j.canlet.2022.215981↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Tuo Hu · 5 shared papers, 2021–2026 · strength 0.81
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Michael Anthony Hollingsworth · 5 shared papers, 2022–2026 · strength 0.80
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Ryan J. King · 5 shared papers, 2021–2026 · strength 0.71
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Divya Murthy · 5 shared papers, 2021–2024 · strength 0.67
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kuldeep Singh Attri · 5 shared papers, 2021–2024 · strength 0.67
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Sai Sundeep Kollala · 4 shared papers, 2023–2026 · strength 0.66
- Paper Systemic immunosuppression limits NK cell therapy efficacy in pancreatic cancer. DOI 10.1186/s12943-025-02340-8↗
- Paper ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. DOI 10.1016/j.canlet.2022.215981↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Scott E. Mulder · 5 shared papers, 2021–2024 · strength 0.65
- Paper ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. DOI 10.1016/j.canlet.2022.215981↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Nicholas James Mullen · 3 shared papers, 2021–2025 · strength 0.62
- Paper Tumor-stromal metabolic crosstalk in pancreatic cancer. DOI 10.1016/j.tcb.2025.04.007↗
- Paper ENT1 blockade by CNX-774 overcomes resistance to DHODH inhibition in pancreatic cancer. DOI 10.1016/j.canlet.2022.215981↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Susanna Varkey Ulahannan · 3 shared papers, 2024–2026 · strength 0.50
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Voddu Suresh · 2 shared papers, 2024–2026 · strength 0.41
- Paper KRAS Signaling Inhibition Induces a Targetable Metabolic Dependency on Lipophagy-Dependent Fatty Acid Oxidation in Panc… DOI 10.1158/0008-5472.can-24-1984↗
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Aneesha Dasgupta · 3 shared papers, 2022–2024 · strength 0.40
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paul M. Grandgenett · 3 shared papers, 2022–2024 · strength 0.40
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Rosa F. Hwang · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ajay Jain · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Vikrant Rai · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Robert Powers · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Teklab Gebregiworgis · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Joshua J. Souchek · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Barish H. Edil · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kathryn Elaine Wellen · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.