ResearcherBaylor College of MedicineActive in the last 2 years
Kuldeep Singh Attri
Matched on ORCID iD 0000-0002-0668-5245; OpenAlex author ID A5014183306. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 5, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 41
2017: 1 paper · 2018: 2 papers · 2019: none · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers
Papers 11
- Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 axis. First author DOI 10.1038/s41556-024-01372-4↗
- Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2-SP1-SAT1 … First author DOI 10.1038/s41556-024-01417-8↗
- The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. First author DOI 10.1073/pnas.2315509121↗
- PTGES Expression Is Associated with Metabolic and Immune Reprogramming in Pancreatic Ductal Adenocarcinoma. First author DOI 10.3390/ijms24087304↗
- CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. Co-author DOI 10.1038/s41388-021-02132-6↗
- Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. Co-author DOI 10.1053/j.gastro.2021.06.045↗
- Macrophages potentiate STAT3 signaling in skeletal muscles and regulate pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.04.017↗
- JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.07.025↗
- Evaluation of Macrophage Polarization in Pancreatic Cancer Microenvironment Under Hypoxia. Co-author DOI 10.1007/978-1-4939-7665-2_23↗
- Phosphoinositide 3-Kinase Signaling Pathway in Pancreatic Ductal Adenocarcinoma Progression, Pathogenesis, and Therapeutics. First author DOI 10.3389/fphys.2018.00335↗
- MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.06.004↗
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Divya Murthy · 9 shared papers, 2017–2024 · strength 1.36
- Paper PTGES Expression Is Associated with Metabolic and Immune Reprogramming in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/ijms24087304↗
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Pankaj Kumar Singh · 10 shared papers, 2017–2024 · strength 1.21
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Surendra Kumar Shukla · 8 shared papers, 2017–2024 · strength 0.94
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kamiya Mehla · 7 shared papers, 2017–2024 · strength 0.83
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ryan J. King · 7 shared papers, 2017–2024 · strength 0.75
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Michael Anthony Hollingsworth · 6 shared papers, 2017–2024 · strength 0.73
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Nina V. Chaika · 6 shared papers, 2017–2024 · strength 0.71
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ravi Kumar Thakur · 5 shared papers, 2021–2024 · strength 0.67
- Paper The MUC1-HIF-1α signaling axis regulates pancreatic cancer pathogenesis through polyamine metabolism remodeling. DOI 10.1073/pnas.2315509121↗
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Aneesha Dasgupta · 6 shared papers, 2017–2024 · strength 0.66
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Scott E. Mulder · 5 shared papers, 2020–2024 · strength 0.60
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paul M. Grandgenett · 5 shared papers, 2017–2024 · strength 0.55
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Chunbo He · 4 shared papers, 2021–2024 · strength 0.48
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Tuo Hu · 3 shared papers, 2021–2024 · strength 0.36
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Enza Vernucci · 4 shared papers, 2017–2022 · strength 0.36
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Macrophages potentiate STAT3 signaling in skeletal muscles and regulate pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.04.017↗
- Paper Metabolic Rewiring by Loss of Sirt5 Promotes Kras-Induced Pancreatic Cancer Progression. DOI 10.1053/j.gastro.2021.06.045↗
- Robert Powers · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Teklab Gebregiworgis · 3 shared papers, 2017–2024 · strength 0.32
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Jaime Abrego · 3 shared papers, 2017–2022 · strength 0.28
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Rosa F. Hwang · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Ajay Jain · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Vikrant Rai · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Joshua J. Souchek · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Sai Sundeep Kollala · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Barish H. Edil · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Kathryn Elaine Wellen · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
- Amarnath (Amar) Natarajan · 2 shared papers, 2024 · strength 0.28
- Paper Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism vi… DOI 10.1038/s41556-024-01372-4↗
- Paper Author Correction: Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering poly… DOI 10.1038/s41556-024-01417-8↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.