ResearcherUniversity of Nebraska–LincolnNo recent work listed
Jaime Abrego
Matched on ORCID iD 0000-0002-8569-2741; OpenAlex author ID A5086355202. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 32
2015: 1 paper · 2016: none · 2017: 5 papers · 2018: 1 paper · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper
Papers 10
- CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. Co-author DOI 10.1038/s41388-021-02132-6↗
- JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. Co-author DOI 10.1016/j.canlet.2020.07.025↗
- Metabolic Alterations in Pancreatic Cancer Progression. First author DOI 10.3390/cancers12010002↗
- Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. Co-author DOI 10.1007/978-1-4939-7665-2_8↗
- GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. Co-author DOI 10.1016/j.canlet.2017.04.029↗
- MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.06.004↗
- Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. Co-author DOI 10.1021/acs.jproteome.7b00246↗
- De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-16-3062↗
- MUC1-Mediated Metabolic Alterations Regulate Response to Radiotherapy in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-17-1151↗
- MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism. Co-author DOI 10.18632/oncotarget.4078↗
Closest collaborators
The 25 strongest of 32 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Pankaj Kumar Singh · 10 shared papers, 2015–2022 · strength 0.77
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. DOI 10.1007/978-1-4939-7665-2_8↗
- Surendra Kumar Shukla · 9 shared papers, 2015–2022 · strength 0.67
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Metabolic Alterations in Pancreatic Cancer Progression. DOI 10.3390/cancers12010002↗
- Venugopal Gunda · 8 shared papers, 2015–2022 · strength 0.55
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Metabolic Alterations in Pancreatic Cancer Progression. DOI 10.3390/cancers12010002↗
- Paper GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. DOI 10.1016/j.canlet.2017.04.029↗
- Enza Vernucci · 7 shared papers, 2017–2022 · strength 0.54
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. DOI 10.1007/978-1-4939-7665-2_8↗
- Paper Metabolic Alterations in Pancreatic Cancer Progression. DOI 10.3390/cancers12010002↗
- Aneesha Dasgupta · 6 shared papers, 2017–2022 · strength 0.49
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Metabolic Alterations in Pancreatic Cancer Progression. DOI 10.3390/cancers12010002↗
- Nina V. Chaika · 7 shared papers, 2015–2022 · strength 0.48
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper Metabolic Alterations in Pancreatic Cancer Progression. DOI 10.3390/cancers12010002↗
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Ryan J. King · 6 shared papers, 2017–2022 · strength 0.47
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper GOT1-mediated anaplerotic glutamine metabolism regulates chronic acidosis stress in pancreatic cancer cells. DOI 10.1016/j.canlet.2017.04.029↗
- Divya Murthy · 5 shared papers, 2017–2022 · strength 0.46
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Evaluating the Metabolic Impact of Hypoxia on Pancreatic Cancer Cells. DOI 10.1007/978-1-4939-7665-2_8↗
- Scott E. Mulder · 3 shared papers, 2017–2022 · strength 0.31
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Kuldeep Singh Attri · 3 shared papers, 2017–2022 · strength 0.28
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper JNK signaling contributes to skeletal muscle wasting and protein turnover in pancreatic cancer cachexia. DOI 10.1016/j.canlet.2020.07.025↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Michael Anthony Hollingsworth · 2 shared papers, 2017–2022 · strength 0.16
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paul M. Grandgenett · 2 shared papers, 2017–2022 · strength 0.16
- Paper CD73 induces GM-CSF/MDSC-mediated suppression of T cells to accelerate pancreatic cancer pathogenesis. DOI 10.1038/s41388-021-02132-6↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Saber Tadros · 2 shared papers, 2017 · strength 0.13
- Chunbo He · 1 shared paper, 2022 · strength 0.12
- Ravi Kumar Thakur · 1 shared paper, 2022 · strength 0.12
- Vinee Purohit · 2 shared papers, 2017 · strength 0.11
- Robert Powers · 2 shared papers, 2017 · strength 0.11
- Teklab Gebregiworgis · 2 shared papers, 2017 · strength 0.11
- Jean L. Grem · 2 shared papers, 2017 · strength 0.10
- Paper De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer. DOI 10.1158/0008-5472.can-16-3062↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Lyudmyla Derby Berim · 2 shared papers, 2017 · strength 0.10
- Paper De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer. DOI 10.1158/0008-5472.can-16-3062↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Joshua J. Souchek · 2 shared papers, 2015–2017 · strength 0.10
- Vikrant Rai · 1 shared paper, 2019 · strength 0.08
- Shuo Wang · 1 shared paper, 2017 · strength 0.06
- Chi H. Lin · 1 shared paper, 2017 · strength 0.06
- Dhanya Haridas · 1 shared paper, 2015 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.