ResearcherNew York UniversityNo recent work listed
Vinee Purohit
Matched on ORCID iD 0000-0001-8093-5540; OpenAlex author ID A5042580120. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 43
2012: 3 papers · 2013: none · 2014: 1 paper · 2015: 1 paper · 2016: none · 2017: 2 papers · 2018: none · 2019: none · 2020: none · 2021: 1 paper
Papers 8
- ATDC binds to KEAP1 to drive NRF2-mediated tumorigenesis and chemoresistance in pancreatic cancer. Co-author DOI 10.1101/gad.344184.120↗
- MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. First author DOI 10.1016/j.ccell.2017.06.004↗
- Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. First author DOI 10.1021/acs.jproteome.7b00246↗
- Active YAP promotes pancreatic cancer cell motility, invasion and tumorigenesis in a mitotic phosphorylation-dependent manner through LPAR3. Co-author DOI 10.18632/oncotarget.5935↗
- Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. Co-author DOI 10.1186/2049-3002-2-18↗
- Graviola: a novel promising natural-derived drug that inhibits tumorigenicity and metastasis of pancreatic cancer cells in vitro and in vivo through altering c… Co-author DOI 10.1016/j.canlet.2012.03.031↗
- MUC1 mucin stabilizes and activates hypoxia-inducible factor 1 alpha to regulate metabolism in pancreatic cancer. Co-author DOI 10.1073/pnas.1203339109↗
- Differential expression of metabolic genes in tumor and stromal components of primary and metastatic loci in pancreatic adenocarcinoma. Co-author DOI 10.1371/journal.pone.0032996↗
Closest collaborators
The 25 strongest of 43 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Pankaj Kumar Singh · 7 shared papers, 2012–2017 · strength 0.28
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper Active YAP promotes pancreatic cancer cell motility, invasion and tumorigenesis in a mitotic phosphorylation-dependent … DOI 10.18632/oncotarget.5935↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Nina V. Chaika · 5 shared papers, 2012–2017 · strength 0.21
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. DOI 10.1186/2049-3002-2-18↗
- Surendra Kumar Shukla · 4 shared papers, 2014–2017 · strength 0.20
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper Active YAP promotes pancreatic cancer cell motility, invasion and tumorigenesis in a mitotic phosphorylation-dependent … DOI 10.18632/oncotarget.5935↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Robert Powers · 4 shared papers, 2012–2017 · strength 0.18
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. DOI 10.1186/2049-3002-2-18↗
- Teklab Gebregiworgis · 4 shared papers, 2012–2017 · strength 0.18
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. DOI 10.1186/2049-3002-2-18↗
- Venugopal Gunda · 3 shared papers, 2014–2017 · strength 0.15
- Paper Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells. DOI 10.1021/acs.jproteome.7b00246↗
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. DOI 10.1186/2049-3002-2-18↗
- Costas Andreas Lyssiotis · 2 shared papers, 2017–2021 · strength 0.15
- Kamiya Mehla · 4 shared papers, 2012–2017 · strength 0.14
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. DOI 10.1186/2049-3002-2-18↗
- Paper Differential expression of metabolic genes in tumor and stromal components of primary and metastatic loci in pancreatic… DOI 10.1371/journal.pone.0032996↗
- Jaime Abrego · 2 shared papers, 2017 · strength 0.11
- Thales Y. Papagiannakopoulos · 1 shared paper, 2021 · strength 0.10
- Lidong Wang · 1 shared paper, 2021 · strength 0.10
- Marina Pasca di Magliano · 1 shared paper, 2021 · strength 0.10
- Diane M. Simeone · 1 shared paper, 2021 · strength 0.10
- Howard C. Crawford · 1 shared paper, 2021 · strength 0.10
- Ethan V. Abel · 1 shared paper, 2021 · strength 0.10
- Michael Anthony Hollingsworth · 3 shared papers, 2012–2017 · strength 0.10
- Paper MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancrea… DOI 10.1016/j.ccell.2017.06.004↗
- Paper Differential expression of metabolic genes in tumor and stromal components of primary and metastatic loci in pancreatic… DOI 10.1371/journal.pone.0032996↗
- Paper MUC1 mucin stabilizes and activates hypoxia-inducible factor 1 alpha to regulate metabolism in pancreatic cancer. DOI 10.1073/pnas.1203339109↗
- Saber Tadros · 1 shared paper, 2017 · strength 0.07
- Scott E. Mulder · 1 shared paper, 2017 · strength 0.07
- Prakash Radhakrishnan · 2 shared papers, 2012–2014 · strength 0.07
- Keith R Johnson · 2 shared papers, 2012 · strength 0.05
- Paper Differential expression of metabolic genes in tumor and stromal components of primary and metastatic loci in pancreatic… DOI 10.1371/journal.pone.0032996↗
- Paper MUC1 mucin stabilizes and activates hypoxia-inducible factor 1 alpha to regulate metabolism in pancreatic cancer. DOI 10.1073/pnas.1203339109↗
- Jixin Dong · 1 shared paper, 2015 · strength 0.05
- Shuping Yang · 1 shared paper, 2015 · strength 0.05
- Joyce C. Solheim · 1 shared paper, 2015 · strength 0.05
- Divya Murthy · 1 shared paper, 2017 · strength 0.04
- Aneesha Dasgupta · 1 shared paper, 2017 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.