Pancreatic Cancer Research

ResearcherJiangsu UniversityActive in the last 2 years

Rongkun Li

Matched on OpenAlex author ID A5001403312. Sources: OpenAlex.

Their work

Papers
11 (first author 1, last author 1)
Trials
0
Grants
0
Labs led
0
Collaborators
33
Papers, trials and grants by year

2014: 1 paper · 2015: 1 paper · 2016: none · 2017: none · 2018: none · 2019: 1 paper · 2020: 1 paper · 2021: 3 papers · 2022: none · 2023: 3 papers · 2024: 1 paper

Papers 11

  1. Ubiquitin-specific protease 7 maintains c-Myc stability to support pancreatic cancer glycolysis and tumor growth. Co-author DOI 10.1186/s12967-024-05962-6↗
  2. The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. First author DOI 10.1007/s11427-022-2146-5↗
  3. METTL3 suppresses pancreatic ductal adenocarcinoma progression through activating endogenous dsRNA-induced anti-tumor immunity. Co-author DOI 10.1007/s13402-023-00829-2↗
  4. PYGL-mediated glucose metabolism reprogramming promotes EMT phenotype and metastasis of pancreatic cancer. Last author DOI 10.7150/ijbs.76756↗
  5. Reciprocal regulation of LOXL2 and HIF1α drives the Warburg effect to support pancreatic cancer aggressiveness. Co-author DOI 10.1038/s41419-021-04391-3↗
  6. TRIM50 Suppresses Pancreatic Cancer Progression and Reverses the Epithelial-Mesenchymal Transition via Facilitating the Ubiquitous Degradation of Snail1. Co-author DOI 10.3389/fonc.2021.695740↗
  7. Deciphering the genomic and lncRNA landscapes of aerobic glycolysis identifies potential therapeutic targets in pancreatic cancer. Co-author DOI 10.7150/ijbs.49243↗
  8. TRIM59 predicts poor prognosis and promotes pancreatic cancer progression via the PI3K/AKT/mTOR-glycolysis signaling axis. Co-author DOI 10.1002/jcb.29433↗
  9. GABRP regulates chemokine signalling, macrophage recruitment and tumour progression in pancreatic cancer through tuning KCNN4-mediated Ca2+ signalling in a GAB… Co-author DOI 10.1136/gutjnl-2018-317479↗
  10. Trophoblast glycoprotein promotes pancreatic ductal adenocarcinoma cell metastasis through Wnt/planar cell polarity signaling. Co-author DOI 10.3892/mmr.2015.3412↗
  11. PNMA1 promotes cell growth in human pancreatic ductal adenocarcinoma. Co-author PMID 25120759↗

Closest collaborators

The 25 strongest of 33 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Shu‐Heng Jiang · 8 shared papers, 2014–2023 · strength 0.86
  2. Zhigang Zhang · 7 shared papers, 2014–2023 · strength 0.75
  3. Lili Zhu · 5 shared papers, 2020–2023 · strength 0.69
  4. Li-Peng Hu · 5 shared papers, 2019–2023 · strength 0.60
  5. Hengchao Li · 3 shared papers, 2021–2024 · strength 0.51
  6. Xu Wang · 3 shared papers, 2021–2023 · strength 0.39
  7. Yongwei Sun · 3 shared papers, 2019–2023 · strength 0.34
  8. Qin Yang · 3 shared papers, 2019–2023 · strength 0.34
  9. Dejun Liu · 2 shared papers, 2021–2023 · strength 0.32
  10. Xueli Zhang · 2 shared papers, 2019–2023 · strength 0.26
  11. Qing Li · 2 shared papers, 2020–2021 · strength 0.23
  12. Jichun Gu · 1 shared paper, 2024 · strength 0.23
  13. Yishen Mao · 1 shared paper, 2024 · strength 0.23
  14. Chen Jin · 1 shared paper, 2024 · strength 0.23
  15. Xi Xiao · 1 shared paper, 2024 · strength 0.23
  16. Deliang Fu · 1 shared paper, 2024 · strength 0.23
  17. Rong Hua · 2 shared papers, 2019–2023 · strength 0.23
  18. Botai Li · 1 shared paper, 2023 · strength 0.19
  19. Yongsheng Jiang · 2 shared papers, 2019–2021 · strength 0.18
  20. Xi Pu · 1 shared paper, 2023 · strength 0.17
  21. Qian Ji · 1 shared paper, 2023 · strength 0.17
  22. Chongyi Jiang · 1 shared paper, 2023 · strength 0.17
  23. Shan Zhang · 1 shared paper, 2023 · strength 0.16
  24. Junfeng Zhang · 1 shared paper, 2023 · strength 0.16
  25. Xin Xing · 1 shared paper, 2021 · strength 0.11

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.