ResearcherShanghai Jiao Tong UniversityActive in the last 2 years
Rong Hua
Matched on ORCID iD 0000-0003-2477-3764; OpenAlex author ID A5101582267. Sources: OpenAlex.
Their work
- Papers
- 32 (first author 2, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 66
2013: 1 paper · 2014: 3 papers · 2015: 5 papers · 2016: 3 papers · 2017: 2 papers · 2018: 1 paper · 2019: 4 papers · 2020: 4 papers · 2021: 4 papers · 2022: 2 papers · 2023: 1 paper · 2024: 2 papers
Papers 32
- Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41419-024-07110-w↗
- The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling pathway. Co-author DOI 10.1186/s12935-024-03440-z↗
- The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s11427-022-2146-5↗
- IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. Co-author DOI 10.1007/s13402-022-00670-z↗
- Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. Co-author DOI 10.1111/cpr.13237↗
- SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. Co-author DOI 10.1002/1878-0261.12970↗
- CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2021.03.016↗
- ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. Co-author DOI 10.1155/2021/5553425↗
- Integrated genomic and transcriptomic analysis reveals unique characteristics of hepatic metastases and pro-metastatic role of complement C1q in pancreatic duc… Co-author DOI 10.1186/s13059-020-02222-w↗
- Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-19-2689↗
- Lactate-Modulated Immunosuppression of Myeloid-Derived Suppressor Cells Contributes to the Radioresistance of Pancreatic Cancer. Co-author DOI 10.1158/2326-6066.cir-20-0111↗
- Molecular markers associated with perineural invasion in pancreatic ductal adenocarcinoma. Co-author DOI 10.3892/ol.2020.11866↗
- Endoplasmic Reticulum stress-dependent expression of ERO1L promotes aerobic glycolysis in Pancreatic Cancer. Last author DOI 10.7150/thno.45124↗
- Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via transcription of PGK1. Co-author DOI 10.1038/s41419-019-2072-5↗
- GABRP regulates chemokine signalling, macrophage recruitment and tumour progression in pancreatic cancer through tuning KCNN4-mediated Ca2+ signalling in a GAB… Co-author DOI 10.1136/gutjnl-2018-317479↗
- DKK2 Impairs Tumor Immunity Infiltration and Correlates with Poor Prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1155/2019/8656282↗
- SFRP4 is a prognostic marker and correlated with Treg cell infiltration in pancreatic ductal adenocarcinoma. Co-author PMID 30906634↗
- Decreased expression of USP9X is associated with poor prognosis in Chinese pancreatic ductal adenocarcinoma patients. Co-author DOI 10.3892/ol.2018.8452↗
- The role of Dickkopf-1 as a potential prognostic marker in pancreatic ductal adenocarcinoma. Co-author DOI 10.1080/15384101.2017.1356510↗
- High Expression of FAM83B Predicts Poor Prognosis in Patients with Pancreatic Ductal Adenocarcinoma and Correlates with Cell Cycle and Cell Proliferation. Co-author DOI 10.7150/jca.20086↗
- Analysis of long non-coding RNA expression profiles in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/srep33535↗
- Overexpressed EDIL3 predicts poor prognosis and promotes anchorage-independent tumor growth in human pancreatic cancer. Co-author DOI 10.18632/oncotarget.6772↗
- Elevated expression of CTHRC1 predicts unfavorable prognosis in patients with pancreatic ductal adenocarcinoma. Co-author PMID 27648368↗
- Decreased LKB1 predicts poor prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1038/srep10575↗
- Corrigendum: Decreased LKB1 predicts poor prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1038/srep15869↗
- High expression of DDR1 is associated with the poor prognosis in Chinese patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s13046-015-0202-1↗
- Downregulation of RPL15 may predict poor survival and associate with tumor progression in pancreatic ductal adenocarcinoma. Co-author DOI 10.18632/oncotarget.5939↗
- High expression of WISP-1 correlates with poor prognosis in pancreatic ductal adenocarcinoma. Co-author PMID 26550461↗
- Long non-coding RNAs expressed in pancreatic ductal adenocarcinoma and lncRNA BC008363 an independent prognostic factor in PDAC. Co-author DOI 10.1016/j.pan.2014.07.013↗
- Effect of CD74 on the prognosis of patients with resectable pancreatic cancer. First author DOI 10.1016/s1499-3872(14)60011-4↗
- MicroRNAs in stool samples as potential screening biomarkers for pancreatic ductal adenocarcinoma cancer. Last author PMID 25520858↗
- Influence of perineural invasion on survival and recurrence in patients with resected pancreatic cancer. First author DOI 10.7314/apjcp.2013.14.9.5133↗
Closest collaborators
The 25 strongest of 66 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yongwei Sun · 29 shared papers, 2014–2024 · strength 2.46
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Yan‐Miao Huo · 27 shared papers, 2014–2024 · strength 2.39
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. DOI 10.1155/2021/5553425↗
- Jian‐Yu Yang · 24 shared papers, 2014–2024 · strength 2.08
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. DOI 10.1155/2021/5553425↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Minwei Yang · 19 shared papers, 2015–2024 · strength 1.71
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Xueliang Fu · 19 shared papers, 2015–2024 · strength 1.65
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Dejun Liu · 18 shared papers, 2014–2024 · strength 1.59
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Junfeng Zhang · 11 shared papers, 2016–2023 · strength 1.11
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. DOI 10.1155/2021/5553425↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Shu‐Heng Jiang · 10 shared papers, 2015–2023 · strength 0.92
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Junfeng Zhang · 10 shared papers, 2014–2024 · strength 0.82
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Paper CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreati… DOI 10.1016/j.canlet.2021.03.016↗
- Zhigang Zhang · 9 shared papers, 2015–2023 · strength 0.71
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper Integrated genomic and transcriptomic analysis reveals unique characteristics of hepatic metastases and pro-metastatic … DOI 10.1186/s13059-020-02222-w↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Jia-Hao Zheng · 4 shared papers, 2022–2024 · strength 0.62
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Hong‐Fei Yao · 4 shared papers, 2022–2024 · strength 0.62
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Wei Chiang Liu · 6 shared papers, 2013–2022 · strength 0.53
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper Molecular markers associated with perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.3892/ol.2020.11866↗
- Paper Integrated genomic and transcriptomic analysis reveals unique characteristics of hepatic metastases and pro-metastatic … DOI 10.1186/s13059-020-02222-w↗
- Ruizhe He · 5 shared papers, 2019–2022 · strength 0.48
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Wei Liu · 5 shared papers, 2014–2024 · strength 0.45
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreati… DOI 10.1016/j.canlet.2021.03.016↗
- Paper Elevated expression of CTHRC1 predicts unfavorable prognosis in patients with pancreatic ductal adenocarcinoma. PMID 27648368↗
- Zong‐Hao Duan · 2 shared papers, 2024 · strength 0.36
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Qin-Yuan Jia · 2 shared papers, 2024 · strength 0.36
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Wei Liu · 2 shared papers, 2020–2024 · strength 0.29
- Li-Peng Hu · 3 shared papers, 2019–2022 · strength 0.27
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Paper GABRP regulates chemokine signalling, macrophage recruitment and tumour progression in pancreatic cancer through tuning… DOI 10.1136/gutjnl-2018-317479↗
- Dejun Liu · 4 shared papers, 2016–2019 · strength 0.25
- Paper Decreased expression of USP9X is associated with poor prognosis in Chinese pancreatic ductal adenocarcinoma patients. DOI 10.3892/ol.2018.8452↗
- Paper Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via trans… DOI 10.1038/s41419-019-2072-5↗
- Paper Elevated expression of CTHRC1 predicts unfavorable prognosis in patients with pancreatic ductal adenocarcinoma. PMID 27648368↗
- Wei Liu · 2 shared papers, 2021–2022 · strength 0.24
- Jie Hong · 5 shared papers, 2014–2017 · strength 0.24
- Paper High Expression of FAM83B Predicts Poor Prognosis in Patients with Pancreatic Ductal Adenocarcinoma and Correlates with… DOI 10.7150/jca.20086↗
- Paper Analysis of long non-coding RNA expression profiles in pancreatic ductal adenocarcinoma. DOI 10.1038/srep33535↗
- Paper Downregulation of RPL15 may predict poor survival and associate with tumor progression in pancreatic ductal adenocarcin… DOI 10.18632/oncotarget.5939↗
- Jianyu Yang · 2 shared papers, 2015–2024 · strength 0.23
- Rongkun Li · 2 shared papers, 2019–2023 · strength 0.23
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper GABRP regulates chemokine signalling, macrophage recruitment and tumour progression in pancreatic cancer through tuning… DOI 10.1136/gutjnl-2018-317479↗
- Qin Yang · 2 shared papers, 2019–2023 · strength 0.23
- Paper The genomic, transcriptomic, and immunological profiles of perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.1007/s11427-022-2146-5↗
- Paper GABRP regulates chemokine signalling, macrophage recruitment and tumour progression in pancreatic cancer through tuning… DOI 10.1136/gutjnl-2018-317479↗
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