ResearcherShanghai Jiao Tong UniversityActive in the last 2 years
Wei Liu
Matched on OpenAlex author ID A5159649202. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 26
2020: 1 paper · 2021: none · 2022: none · 2023: none · 2024: 3 papers · 2025: none · 2026: 1 paper
Papers 5
- OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. Co-author DOI 10.7150/ijbs.125552↗
- Evaluation of a new scoring system for assessing nerve invasion in resected pancreatic cancer: A single-center retrospective analysis. Co-author DOI 10.1016/j.canlet.2024.217213↗
- A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2024.114633↗
- Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s41419-024-07110-w↗
- Endoplasmic Reticulum stress-dependent expression of ERO1L promotes aerobic glycolysis in Pancreatic Cancer. Co-author DOI 10.7150/thno.45124↗
Closest collaborators
The 25 strongest of 26 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Minwei Yang · 4 shared papers, 2020–2026 · strength 0.72
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper Evaluation of a new scoring system for assessing nerve invasion in resected pancreatic cancer: A single-center retrospe… DOI 10.1016/j.canlet.2024.217213↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Yan‐Miao Huo · 4 shared papers, 2020–2026 · strength 0.70
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Zong‐Hao Duan · 3 shared papers, 2024–2026 · strength 0.59
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Hong‐Fei Yao · 3 shared papers, 2024 · strength 0.54
- Paper Evaluation of a new scoring system for assessing nerve invasion in resected pancreatic cancer: A single-center retrospe… DOI 10.1016/j.canlet.2024.217213↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Yongwei Sun · 3 shared papers, 2020–2024 · strength 0.47
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Endoplasmic Reticulum stress-dependent expression of ERO1L promotes aerobic glycolysis in Pancreatic Cancer. DOI 10.7150/thno.45124↗
- Qin-Yuan Jia · 2 shared papers, 2024 · strength 0.35
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Jia-Hao Zheng · 2 shared papers, 2024 · strength 0.35
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Yifan Yin · 2 shared papers, 2024 · strength 0.35
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Rong Hua · 2 shared papers, 2020–2024 · strength 0.29
- Shu‐Heng Jiang · 2 shared papers, 2020–2024 · strength 0.29
- Linli Yao · 1 shared paper, 2026 · strength 0.24
- Yong-Wei Sun · 1 shared paper, 2024 · strength 0.19
- Jun-Feng Zhang · 1 shared paper, 2024 · strength 0.19
- Shan Zhang · 1 shared paper, 2024 · strength 0.19
- Rong Hua · 1 shared paper, 2024 · strength 0.19
- Yu-Heng Zhu · 1 shared paper, 2024 · strength 0.18
- Jianyu Yang · 1 shared paper, 2024 · strength 0.18
- Xueliang Fu · 1 shared paper, 2024 · strength 0.18
- Qing Li · 1 shared paper, 2024 · strength 0.18
- Jian Yang · 1 shared paper, 2024 · strength 0.18
- Li-Peng Hu · 1 shared paper, 2024 · strength 0.18
- Pei‐Xuan Ji · 1 shared paper, 2024 · strength 0.18
- Dejun Liu · 1 shared paper, 2024 · strength 0.18
- Dejun Liu · 1 shared paper, 2024 · strength 0.18
- Jian‐Yu Yang · 1 shared paper, 2020 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.