ResearcherShanghai Jiao Tong UniversityActive in the last 2 years
Yan‐Miao Huo
Matched on ORCID iD 0000-0003-4730-7184; OpenAlex author ID A5024260926, A5129606199. Sources: OpenAlex.
Their work
- Papers
- 43 (first author 2, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 84
2014: 2 papers · 2015: 4 papers · 2016: 4 papers · 2017: 2 papers · 2018: 2 papers · 2019: 3 papers · 2020: 4 papers · 2021: 5 papers · 2022: 5 papers · 2023: 3 papers · 2024: 5 papers · 2025: 1 paper · 2026: 3 papers
Papers 43
- A cholesterol-dependent LRRC8A-caveolin-1 axis sustains KRAS/EGFR signaling, ribosome biogenesis and growth in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41388-026-03864-z↗
- A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment and Drive Hepatic Metastasis, Revealing… Co-author DOI 10.1158/1078-0432.ccr-25-3359↗
- OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. Last author DOI 10.7150/ijbs.125552↗
- TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. Co-author DOI 10.7150/ijbs.104598↗
- AGFG1 increases cholesterol biosynthesis by disrupting intracellular cholesterol homeostasis to promote PDAC progression. Co-author DOI 10.1016/j.canlet.2024.217130↗
- A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2024.114633↗
- Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41419-024-07110-w↗
- The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling pathway. Last author DOI 10.1186/s12935-024-03440-z↗
- A Nutrient-Deficient Microenvironment Facilitates Ferroptosis Resistance via the FAM60A-PPAR Axis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.34133/research.0300↗
- Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreatic cancer. Co-author DOI 10.1007/s10495-023-01843-3↗
- Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. Last author DOI 10.1007/s13402-022-00741-1↗
- ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. Last author DOI 10.1016/j.hbpd.2022.04.005↗
- Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreatic cancer: CSTB orchestrates autophagy a… Co-author DOI 10.1002/ctm2.1126↗
- IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. Co-author DOI 10.1007/s13402-022-00670-z↗
- SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2022.215693↗
- Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. Co-author DOI 10.1111/cpr.13237↗
- GJB3 promotes pancreatic cancer liver metastasis by enhancing the polarization and survival of neutrophil. Co-author DOI 10.3389/fimmu.2022.983116↗
- SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. First author DOI 10.1002/1878-0261.12970↗
- CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2021.03.016↗
- Single-cell RNA sequencing reveals that targeting HSP90 suppresses PDAC progression by restraining mitochondrial bioenergetics. Co-author DOI 10.1038/s41389-021-00311-4↗
- ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. Co-author DOI 10.1155/2021/5553425↗
- Integrated genomic and transcriptomic analysis reveals unique characteristics of hepatic metastases and pro-metastatic role of complement C1q in pancreatic duc… Co-author DOI 10.1186/s13059-020-02222-w↗
- Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-19-2689↗
- Lactate-Modulated Immunosuppression of Myeloid-Derived Suppressor Cells Contributes to the Radioresistance of Pancreatic Cancer. Co-author DOI 10.1158/2326-6066.cir-20-0111↗
- Molecular markers associated with perineural invasion in pancreatic ductal adenocarcinoma. Co-author DOI 10.3892/ol.2020.11866↗
- Endoplasmic Reticulum stress-dependent expression of ERO1L promotes aerobic glycolysis in Pancreatic Cancer. Co-author DOI 10.7150/thno.45124↗
- Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via transcription of PGK1. Co-author DOI 10.1038/s41419-019-2072-5↗
- DKK2 Impairs Tumor Immunity Infiltration and Correlates with Poor Prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1155/2019/8656282↗
- SFRP4 is a prognostic marker and correlated with Treg cell infiltration in pancreatic ductal adenocarcinoma. Co-author PMID 30906634↗
- IL22RA1/STAT3 Signaling Promotes Stemness and Tumorigenicity in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-17-3131↗
- Decreased expression of USP9X is associated with poor prognosis in Chinese pancreatic ductal adenocarcinoma patients. First author DOI 10.3892/ol.2018.8452↗
- The role of Dickkopf-1 as a potential prognostic marker in pancreatic ductal adenocarcinoma. Co-author DOI 10.1080/15384101.2017.1356510↗
- High Expression of FAM83B Predicts Poor Prognosis in Patients with Pancreatic Ductal Adenocarcinoma and Correlates with Cell Cycle and Cell Proliferation. Co-author DOI 10.7150/jca.20086↗
- Analysis of long non-coding RNA expression profiles in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/srep33535↗
- Efficacy of Neo-Adjuvant Chemoradiotherapy for Resectable Pancreatic Adenocarcinoma: A PRISMA-Compliant Meta-Analysis and Systematic Review. Co-author DOI 10.1097/md.0000000000003009↗
- Overexpressed EDIL3 predicts poor prognosis and promotes anchorage-independent tumor growth in human pancreatic cancer. Co-author DOI 10.18632/oncotarget.6772↗
- Elevated expression of CTHRC1 predicts unfavorable prognosis in patients with pancreatic ductal adenocarcinoma. Co-author PMID 27648368↗
- Decreased LKB1 predicts poor prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1038/srep10575↗
- Corrigendum: Decreased LKB1 predicts poor prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1038/srep15869↗
- High expression of DDR1 is associated with the poor prognosis in Chinese patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s13046-015-0202-1↗
- High expression of WISP-1 correlates with poor prognosis in pancreatic ductal adenocarcinoma. Co-author PMID 26550461↗
- Long non-coding RNAs expressed in pancreatic ductal adenocarcinoma and lncRNA BC008363 an independent prognostic factor in PDAC. Co-author DOI 10.1016/j.pan.2014.07.013↗
- Effect of CD74 on the prognosis of patients with resectable pancreatic cancer. Co-author DOI 10.1016/s1499-3872(14)60011-4↗
Closest collaborators
The 25 strongest of 84 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yongwei Sun · 35 shared papers, 2014–2026 · strength 3.75
- Paper A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment… DOI 10.1158/1078-0432.ccr-25-3359↗
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Minwei Yang · 29 shared papers, 2015–2026 · strength 3.30
- Paper A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment… DOI 10.1158/1078-0432.ccr-25-3359↗
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Jian‐Yu Yang · 26 shared papers, 2015–2026 · strength 2.57
- Paper A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment… DOI 10.1158/1078-0432.ccr-25-3359↗
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Rong Hua · 27 shared papers, 2014–2024 · strength 2.39
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. DOI 10.1155/2021/5553425↗
- Dejun Liu · 21 shared papers, 2014–2024 · strength 2.04
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. DOI 10.1007/s13402-022-00741-1↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Xueliang Fu · 21 shared papers, 2015–2024 · strength 1.94
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Hong‐Fei Yao · 10 shared papers, 2022–2025 · strength 1.69
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Jia-Hao Zheng · 10 shared papers, 2022–2024 · strength 1.64
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. DOI 10.1007/s13402-022-00741-1↗
- Zong‐Hao Duan · 8 shared papers, 2023–2026 · strength 1.61
- Paper A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment… DOI 10.1158/1078-0432.ccr-25-3359↗
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Shu‐Heng Jiang · 13 shared papers, 2015–2026 · strength 1.56
- Paper A cholesterol-dependent LRRC8A-caveolin-1 axis sustains KRAS/EGFR signaling, ribosome biogenesis and growth in pancreat… DOI 10.1038/s41388-026-03864-z↗
- Paper Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. DOI 10.1007/s13402-022-00741-1↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Junfeng Zhang · 13 shared papers, 2016–2026 · strength 1.42
- Paper A Malignant Subpopulation of H2AFZ+ Cells Interacts with Myeloid Cells to Promote an Anti-inflammatory Microenvironment… DOI 10.1158/1078-0432.ccr-25-3359↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper ERO1L Promotes Hepatic Metastasis through Activating Epithelial-Mesenchymal Transition (EMT) in Pancreatic Cancer. DOI 10.1155/2021/5553425↗
- Qin-Yuan Jia · 7 shared papers, 2023–2025 · strength 1.26
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. DOI 10.1007/s13402-022-00741-1↗
- Li-Peng Hu · 7 shared papers, 2020–2026 · strength 1.05
- Paper A cholesterol-dependent LRRC8A-caveolin-1 axis sustains KRAS/EGFR signaling, ribosome biogenesis and growth in pancreat… DOI 10.1038/s41388-026-03864-z↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper AGFG1 increases cholesterol biosynthesis by disrupting intracellular cholesterol homeostasis to promote PDAC progressio… DOI 10.1016/j.canlet.2024.217130↗
- Ruizhe He · 8 shared papers, 2019–2023 · strength 0.95
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Yu-Heng Zhu · 5 shared papers, 2023–2025 · strength 0.91
- Paper TMEM105 modulates disulfidptosis and tumor growth in pancreatic cancer via the β-catenin-c-MYC-GLUT1 axis. DOI 10.7150/ijbs.104598↗
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper Immunosuppression, immune escape, and immunotherapy in pancreatic cancer: focused on the tumor microenvironment. DOI 10.1007/s13402-022-00741-1↗
- Zhigang Zhang · 10 shared papers, 2015–2026 · strength 0.89
- Paper A cholesterol-dependent LRRC8A-caveolin-1 axis sustains KRAS/EGFR signaling, ribosome biogenesis and growth in pancreat… DOI 10.1038/s41388-026-03864-z↗
- Paper Integrated genomic and transcriptomic analysis reveals unique characteristics of hepatic metastases and pro-metastatic … DOI 10.1186/s13059-020-02222-w↗
- Paper Single-cell RNA sequencing reveals that targeting HSP90 suppresses PDAC progression by restraining mitochondrial bioene… DOI 10.1038/s41389-021-00311-4↗
- Junfeng Zhang · 9 shared papers, 2014–2024 · strength 0.77
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Paper CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreati… DOI 10.1016/j.canlet.2021.03.016↗
- Wei Liu · 4 shared papers, 2020–2026 · strength 0.70
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Wei Chiang Liu · 7 shared papers, 2016–2022 · strength 0.66
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Paper Molecular markers associated with perineural invasion in pancreatic ductal adenocarcinoma. DOI 10.3892/ol.2020.11866↗
- Dejun Liu · 6 shared papers, 2016–2024 · strength 0.60
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper AGFG1 increases cholesterol biosynthesis by disrupting intracellular cholesterol homeostasis to promote PDAC progressio… DOI 10.1016/j.canlet.2024.217130↗
- Paper Decreased expression of USP9X is associated with poor prognosis in Chinese pancreatic ductal adenocarcinoma patients. DOI 10.3892/ol.2018.8452↗
- Jian Yang · 4 shared papers, 2022–2024 · strength 0.58
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Yifan Yin · 3 shared papers, 2024 · strength 0.53
- Paper A CLIC1 network coordinates matrix stiffness and the Warburg effect to promote tumor growth in pancreatic cancer. DOI 10.1016/j.celrep.2024.114633↗
- Paper Oncogenic GALNT5 confers FOLFIRINOX resistance via activating the MYH9/ NOTCH/ DDR axis in pancreatic ductal adenocarci… DOI 10.1038/s41419-024-07110-w↗
- Paper AGFG1 increases cholesterol biosynthesis by disrupting intracellular cholesterol homeostasis to promote PDAC progressio… DOI 10.1016/j.canlet.2024.217130↗
- Linli Yao · 3 shared papers, 2021–2026 · strength 0.51
- Paper OSBPL10 Drives Lipophagy-Mediated Lipid Mobilization to Promote Pancreatic Ductal Adenocarcinoma Progression. DOI 10.7150/ijbs.125552↗
- Paper AGFG1 increases cholesterol biosynthesis by disrupting intracellular cholesterol homeostasis to promote PDAC progressio… DOI 10.1016/j.canlet.2024.217130↗
- Paper Single-cell RNA sequencing reveals that targeting HSP90 suppresses PDAC progression by restraining mitochondrial bioene… DOI 10.1038/s41389-021-00311-4↗
- Wei Liu · 5 shared papers, 2014–2024 · strength 0.45
- Paper The lncRNA LINC01605 promotes the progression of pancreatic ductal adenocarcinoma by activating the mTOR signaling path… DOI 10.1186/s12935-024-03440-z↗
- Paper CD74 promotes perineural invasion of cancer cells and mediates neuroplasticity via the AKT/EGR-1/GDNF axis in pancreati… DOI 10.1016/j.canlet.2021.03.016↗
- Paper Elevated expression of CTHRC1 predicts unfavorable prognosis in patients with pancreatic ductal adenocarcinoma. PMID 27648368↗
- Dapeng Xu · 3 shared papers, 2022–2023 · strength 0.45
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Increased SPON1 promotes pancreatic ductal adenocarcinoma progression by enhancing IL-6 trans-signalling. DOI 10.1111/cpr.13237↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.