ResearcherShanghai Jiao Tong UniversityActive in the last 2 years
Ruizhe He
Matched on ORCID iD 0000-0001-5355-3889; OpenAlex author ID A5074421421. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 48
2018: 1 paper · 2019: 3 papers · 2020: 1 paper · 2021: 1 paper · 2022: 2 papers · 2023: 2 papers · 2024: none · 2025: none · 2026: 1 paper
Papers 11
- A conserved re-epithelialization program underlies malignancy in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.ccell.2026.03.021↗
- Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreatic cancer. Co-author DOI 10.1007/s10495-023-01843-3↗
- ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. Co-author DOI 10.1016/j.hbpd.2022.04.005↗
- IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. Co-author DOI 10.1007/s13402-022-00670-z↗
- SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2022.215693↗
- SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. Co-author DOI 10.1002/1878-0261.12970↗
- Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-19-2689↗
- CD90 highly expressed population harbors a stemness signature and creates an immunosuppressive niche in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2019.03.051↗
- Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via transcription of PGK1. First author DOI 10.1038/s41419-019-2072-5↗
- DKK2 Impairs Tumor Immunity Infiltration and Correlates with Poor Prognosis in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1155/2019/8656282↗
- High expression of WNT7A predicts poor prognosis and promote tumor metastasis in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41598-018-34094-3↗
Closest collaborators
The 25 strongest of 48 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Minwei Yang · 10 shared papers, 2018–2023 · strength 1.13
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Yan‐Miao Huo · 8 shared papers, 2019–2023 · strength 0.95
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Yongwei Sun · 8 shared papers, 2019–2023 · strength 0.89
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Xueliang Fu · 8 shared papers, 2018–2023 · strength 0.84
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Jian‐Yu Yang · 7 shared papers, 2019–2023 · strength 0.76
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Dejun Liu · 5 shared papers, 2019–2023 · strength 0.60
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Rong Hua · 5 shared papers, 2019–2022 · strength 0.48
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Jia-Hao Zheng · 3 shared papers, 2022–2023 · strength 0.47
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Hong‐Fei Yao · 3 shared papers, 2022–2023 · strength 0.47
- Paper ADAMTS12 promotes migration and epithelial-mesenchymal transition and predicts poor prognosis for pancreatic cancer. DOI 10.1016/j.hbpd.2022.04.005↗
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Jian Yang · 3 shared papers, 2022–2023 · strength 0.40
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Shu‐Heng Jiang · 3 shared papers, 2020–2022 · strength 0.32
- Paper IRAK2-NF-κB signaling promotes glycolysis-dependent tumor growth in pancreatic cancer. DOI 10.1007/s13402-022-00670-z↗
- Paper SF3B1 mutation in pancreatic cancer contributes to aerobic glycolysis and tumor growth through a PP2A-c-Myc axis. DOI 10.1002/1878-0261.12970↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Dapeng Xu · 2 shared papers, 2022–2023 · strength 0.32
- Ping Lu · 3 shared papers, 2019–2022 · strength 0.30
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Paper CD90 highly expressed population harbors a stemness signature and creates an immunosuppressive niche in pancreatic canc… DOI 10.1016/j.canlet.2019.03.051↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Ningning Niu · 3 shared papers, 2019–2022 · strength 0.30
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Paper CD90 highly expressed population harbors a stemness signature and creates an immunosuppressive niche in pancreatic canc… DOI 10.1016/j.canlet.2019.03.051↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Jing Xue · 3 shared papers, 2019–2022 · strength 0.30
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Paper CD90 highly expressed population harbors a stemness signature and creates an immunosuppressive niche in pancreatic canc… DOI 10.1016/j.canlet.2019.03.051↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Yongsheng Jiang · 3 shared papers, 2019–2022 · strength 0.28
- Paper SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer. DOI 10.1016/j.canlet.2022.215693↗
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Paper Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via trans… DOI 10.1038/s41419-019-2072-5↗
- Wei Liu · 2 shared papers, 2021–2022 · strength 0.24
- Zhigang Zhang · 3 shared papers, 2019–2020 · strength 0.24
- Paper Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarc… DOI 10.1158/0008-5472.can-19-2689↗
- Paper DKK2 Impairs Tumor Immunity Infiltration and Correlates with Poor Prognosis in Pancreatic Ductal Adenocarcinoma. DOI 10.1155/2019/8656282↗
- Paper Transcription factor NFAT5 contributes to the glycolytic phenotype rewiring and pancreatic cancer progression via trans… DOI 10.1038/s41419-019-2072-5↗
- Junfeng Zhang · 2 shared papers, 2019–2023 · strength 0.23
- Paper Analysis of cuproptosis-related lncRNA signature for predicting prognosis and tumor immune microenvironment in pancreat… DOI 10.1007/s10495-023-01843-3↗
- Paper DKK2 Impairs Tumor Immunity Infiltration and Correlates with Poor Prognosis in Pancreatic Ductal Adenocarcinoma. DOI 10.1155/2019/8656282↗
- Meilian Zhuo · 1 shared paper, 2026 · strength 0.21
- Xianlin Han · 1 shared paper, 2026 · strength 0.21
- Mingyang Liu · 1 shared paper, 2026 · strength 0.21
- Mo Chen · 1 shared paper, 2026 · strength 0.21
- Junya Peng · 1 shared paper, 2026 · strength 0.21
- Menghua Dai · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.