ResearcherShanghai Jiao Tong UniversityNo recent work listed
Cheng Xiong
Matched on OpenAlex author ID A5024473312. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 21
2021: 1 paper · 2022: 1 paper · 2023: 3 papers
Papers 5
- Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired immunity infiltration in pancreatic ductal… Co-author DOI 10.1007/s00262-023-03438-y↗
- Schwann cells regulate tumor cells and cancer-associated fibroblasts in the pancreatic ductal adenocarcinoma microenvironment. Co-author DOI 10.1038/s41467-023-40314-w↗
- LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability. Co-author DOI 10.1186/s12967-023-04702-6↗
- Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreatic cancer: CSTB orchestrates autophagy a… Co-author DOI 10.1002/ctm2.1126↗
- Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal transition. Co-author DOI 10.18632/aging.202264↗
Closest collaborators
The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Lingxi Jiang · 5 shared papers, 2021–2023 · strength 0.73
- Paper LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintainin… DOI 10.1186/s12967-023-04702-6↗
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal… DOI 10.18632/aging.202264↗
- Youwei Zhu · 5 shared papers, 2021–2023 · strength 0.73
- Paper LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintainin… DOI 10.1186/s12967-023-04702-6↗
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal… DOI 10.18632/aging.202264↗
- Meilin Xue · 5 shared papers, 2021–2023 · strength 0.73
- Paper LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintainin… DOI 10.1186/s12967-023-04702-6↗
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal… DOI 10.18632/aging.202264↗
- Hao Chen · 4 shared papers, 2021–2023 · strength 0.60
- Paper LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintainin… DOI 10.1186/s12967-023-04702-6↗
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal… DOI 10.18632/aging.202264↗
- Yongsheng Jiang · 3 shared papers, 2021–2023 · strength 0.43
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Tumor-associated macrophages promote pancreatic ductal adenocarcinoma progression by inducing epithelial-to-mesenchymal… DOI 10.18632/aging.202264↗
- Paper Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreat… DOI 10.1002/ctm2.1126↗
- Lijie Han · 3 shared papers, 2022–2023 · strength 0.43
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreat… DOI 10.1002/ctm2.1126↗
- Paper Schwann cells regulate tumor cells and cancer-associated fibroblasts in the pancreatic ductal adenocarcinoma microenvir… DOI 10.1038/s41467-023-40314-w↗
- Baiyong Shen · 2 shared papers, 2023 · strength 0.30
- Paper LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintainin… DOI 10.1186/s12967-023-04702-6↗
- Paper Schwann cells regulate tumor cells and cancer-associated fibroblasts in the pancreatic ductal adenocarcinoma microenvir… DOI 10.1038/s41467-023-40314-w↗
- Baiyong Shen · 2 shared papers, 2022–2023 · strength 0.29
- Paper Identification of a novel immune checkpoint molecule V-set immunoglobulin domain-containing 4 that leads to impaired im… DOI 10.1007/s00262-023-03438-y↗
- Paper Cystatin B increases autophagic flux by sustaining proteolytic activity of cathepsin B and fuels glycolysis in pancreat… DOI 10.1002/ctm2.1126↗
- Ting Wang · 1 shared paper, 2023 · strength 0.16
- Zhichong Wu · 1 shared paper, 2023 · strength 0.16
- Xiaozhu Lin · 1 shared paper, 2023 · strength 0.16
- Lijie Han · 1 shared paper, 2023 · strength 0.16
- Jiän Zhang · 1 shared paper, 2023 · strength 0.16
- Minwei Yang · 1 shared paper, 2023 · strength 0.16
- Yan‐Miao Huo · 1 shared paper, 2022 · strength 0.14
- Hongzhe Li · 1 shared paper, 2022 · strength 0.14
- Yizhi Cao · 1 shared paper, 2023 · strength 0.13
- Shulin Zhao · 1 shared paper, 2023 · strength 0.13
- Hao Chen · 1 shared paper, 2023 · strength 0.13
- Chenghong Peng · 1 shared paper, 2023 · strength 0.13
- Xiaxing Deng · 1 shared paper, 2023 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.