ResearcherUniversity Hospital OlomoucNo recent work listed
R Havlík
Matched on OpenAlex author ID A5032028505. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 23
2013: 1 paper · 2014: none · 2015: 2 papers · 2016: 3 papers · 2017: 2 papers · 2018: none · 2019: 1 paper
Papers 9
- Genetic analysis of subsequent second primary malignant neoplasms in long-term pancreatic cancer survivors suggests new potential hereditary genetic alteration… Co-author DOI 10.2147/cmar.s185352↗
- Different clinical presentations of metachronous pulmonary metastases after resection of pancreatic ductal adenocarcinoma: Retrospective study and review of th… Co-author DOI 10.3748/wjg.v23.i35.6420↗
- Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. Co-author DOI 10.3892/ol.2017.6946↗
- Long-term survival after resections for pancreatic ductal adenocarcinoma. Single centre study. Last author DOI 10.5507/bp.2016.011↗
- [Current status regarding surgical treatment of pancreatic cancer in the Czech Republic]. Co-author PMID 27226268↗
- [The survival of patients with radically non-resectable pancreatic cancer]. Co-author PMID 27410755↗
- [Determination of CEA, EGFR and hTERT expression in peritoneal lavage in patients with pancreatic adenocarcinoma using RT - PCR method]. Co-author PMID 26766154↗
- [Surgical therapy of pancreatic cancer - 5 years survival]. Last author PMID 26766155↗
- Occult tumour cells in peritoneal lavage are a negative prognostic factor in pancreatic cancer. Co-author DOI 10.5507/bp.2012.061↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Martin Loveček · 9 shared papers, 2013–2019 · strength 0.48
- Paper Genetic analysis of subsequent second primary malignant neoplasms in long-term pancreatic cancer survivors suggests new… DOI 10.2147/cmar.s185352↗
- Paper [The survival of patients with radically non-resectable pancreatic cancer]. PMID 27410755↗
- Paper Different clinical presentations of metachronous pulmonary metastases after resection of pancreatic ductal adenocarcino… DOI 10.3748/wjg.v23.i35.6420↗
- Č Neoral · 7 shared papers, 2013–2019 · strength 0.36
- Paper Genetic analysis of subsequent second primary malignant neoplasms in long-term pancreatic cancer survivors suggests new… DOI 10.2147/cmar.s185352↗
- Paper [The survival of patients with radically non-resectable pancreatic cancer]. PMID 27410755↗
- Paper Long-term survival after resections for pancreatic ductal adenocarcinoma. Single centre study. DOI 10.5507/bp.2016.011↗
- Dušan Klos · 6 shared papers, 2013–2017 · strength 0.31
- Paper [The survival of patients with radically non-resectable pancreatic cancer]. PMID 27410755↗
- Paper Different clinical presentations of metachronous pulmonary metastases after resection of pancreatic ductal adenocarcino… DOI 10.3748/wjg.v23.i35.6420↗
- Paper Long-term survival after resections for pancreatic ductal adenocarcinoma. Single centre study. DOI 10.5507/bp.2016.011↗
- Bohuslav Melichar · 3 shared papers, 2017–2019 · strength 0.19
- Paper Genetic analysis of subsequent second primary malignant neoplasms in long-term pancreatic cancer survivors suggests new… DOI 10.2147/cmar.s185352↗
- Paper Different clinical presentations of metachronous pulmonary metastases after resection of pancreatic ductal adenocarcino… DOI 10.3748/wjg.v23.i35.6420↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Beatrice Mohelníková-Duchoňová · 3 shared papers, 2017–2019 · strength 0.19
- Paper Genetic analysis of subsequent second primary malignant neoplasms in long-term pancreatic cancer survivors suggests new… DOI 10.2147/cmar.s185352↗
- Paper Different clinical presentations of metachronous pulmonary metastases after resection of pancreatic ductal adenocarcino… DOI 10.3748/wjg.v23.i35.6420↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Linda Bebarova · 3 shared papers, 2015–2016 · strength 0.16
- Paper [The survival of patients with radically non-resectable pancreatic cancer]. PMID 27410755↗
- Paper Long-term survival after resections for pancreatic ductal adenocarcinoma. Single centre study. DOI 10.5507/bp.2016.011↗
- Paper [Determination of CEA, EGFR and hTERT expression in peritoneal lavage in patients with pancreatic adenocarcinoma using … PMID 26766154↗
- Viktor Hlaváč · 2 shared papers, 2017–2019 · strength 0.13
- Pavel Souček · 2 shared papers, 2017–2019 · strength 0.13
- Mohamed Ghothim · 3 shared papers, 2013–2015 · strength 0.13
- Paper [Surgical therapy of pancreatic cancer - 5 years survival]. PMID 26766155↗
- Paper [Determination of CEA, EGFR and hTERT expression in peritoneal lavage in patients with pancreatic adenocarcinoma using … PMID 26766154↗
- Paper Occult tumour cells in peritoneal lavage are a negative prognostic factor in pancreatic cancer. DOI 10.5507/bp.2012.061↗
- Radmila Lemstrová · 2 shared papers, 2017 · strength 0.11
- Jan Hlavsa · 2 shared papers, 2016–2017 · strength 0.10
- Marián Hajdúch · 2 shared papers, 2013–2015 · strength 0.08
- Marianna Borecká · 1 shared paper, 2019 · strength 0.08
- Tomáš Zemánek · 1 shared paper, 2019 · strength 0.08
- Zdeněk Kleibl · 1 shared paper, 2019 · strength 0.08
- Markéta Janatová · 1 shared paper, 2019 · strength 0.08
- Martin Stašek · 1 shared paper, 2016 · strength 0.05
- Martin Oliverius · 1 shared paper, 2017 · strength 0.05
- David J. Hughes · 1 shared paper, 2017 · strength 0.05
- Zdeněk Kala · 1 shared paper, 2017 · strength 0.05
- R Gürlich · 1 shared paper, 2016 · strength 0.04
- Z Krška · 1 shared paper, 2016 · strength 0.04
- Miroslav Ryska · 1 shared paper, 2016 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.