ResearcherUniversity College DublinActive in the last 2 years
David J. Hughes
Matched on ORCID iD 0000-0003-1668-8770; OpenAlex author ID A5039013705, A5134049873. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 45
2017: 3 papers · 2018: none · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: 2 papers · 2025: 2 papers · 2026: 1 paper
Papers 8
- Non-coding RNAs as biomarkers and therapeutic targets in pancreatic cancer: Clinical implications and translational perspectives. Co-author DOI 10.1016/j.biopha.2026.119394↗
- The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. Last author DOI 10.1016/j.semcancer.2025.03.002↗
- The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC. Co-author DOI 10.1016/j.semcancer.2025.05.004↗
- Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic ductal adenocarcinoma: a Mendelian randomisation study. Co-author DOI 10.1038/s41598-024-77431-5↗
- Publisher Correction: Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic ductal adenocarcinoma: a Mendelian randomi… Co-author DOI 10.1038/s41598-024-83618-7↗
- SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. Co-author DOI 10.1038/srep43812↗
- Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. Co-author DOI 10.1038/tpj.2016.55↗
- Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. Co-author DOI 10.3892/ol.2017.6946↗
Closest collaborators
The 25 strongest of 45 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Daniele Campa · 6 shared papers, 2017–2026 · strength 1.13
- Paper The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. DOI 10.1016/j.semcancer.2025.03.002↗
- Paper Non-coding RNAs as biomarkers and therapeutic targets in pancreatic cancer: Clinical implications and translational per… DOI 10.1016/j.biopha.2026.119394↗
- Paper The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC. DOI 10.1016/j.semcancer.2025.05.004↗
- Neil Daniel · 5 shared papers, 2024–2026 · strength 1.09
- Paper The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. DOI 10.1016/j.semcancer.2025.03.002↗
- Paper Non-coding RNAs as biomarkers and therapeutic targets in pancreatic cancer: Clinical implications and translational per… DOI 10.1016/j.biopha.2026.119394↗
- Paper The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC. DOI 10.1016/j.semcancer.2025.05.004↗
- Cosmeri Rizzato · 5 shared papers, 2017–2025 · strength 0.90
- Paper The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. DOI 10.1016/j.semcancer.2025.03.002↗
- Paper The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC. DOI 10.1016/j.semcancer.2025.05.004↗
- Paper Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic ductal adenocarcinoma: a… DOI 10.1038/s41598-024-77431-5↗
- Riccardo Farinella · 4 shared papers, 2024–2025 · strength 0.86
- Paper The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. DOI 10.1016/j.semcancer.2025.03.002↗
- Paper The exposome and pancreatic cancer, lifestyle and environmental risk factors for PDAC. DOI 10.1016/j.semcancer.2025.05.004↗
- Paper Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic ductal adenocarcinoma: a… DOI 10.1038/s41598-024-77431-5↗
- Federico Canzian · 3 shared papers, 2017–2024 · strength 0.43
- Paper Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic ductal adenocarcinoma: a… DOI 10.1038/s41598-024-77431-5↗
- Paper Publisher Correction: Genetically predicted gut bacteria, circulating bacteria-associated metabolites and pancreatic du… DOI 10.1038/s41598-024-83618-7↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Pinar Uysal Onganer · 2 shared papers, 2025–2026 · strength 0.43
- Juan Sáinz · 2 shared papers, 2025–2026 · strength 0.43
- Pavel Souček · 4 shared papers, 2017–2025 · strength 0.42
- Paper The relationship of the microbiome, associated metabolites and the gut barrier with pancreatic cancer. DOI 10.1016/j.semcancer.2025.03.002↗
- Paper Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. DOI 10.1038/tpj.2016.55↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Ľudmila Vodičková · 2 shared papers, 2017–2025 · strength 0.24
- Pavel Vodička · 2 shared papers, 2017–2025 · strength 0.24
- Stefano Landi · 2 shared papers, 2017–2025 · strength 0.24
- María Carretero-Fernández · 1 shared paper, 2026 · strength 0.23
- Annalisa Comandatore · 1 shared paper, 2025 · strength 0.20
- Ruggero Ponz de Leon Pisani · 1 shared paper, 2025 · strength 0.20
- Lívia Archibugi · 1 shared paper, 2025 · strength 0.20
- Giulia Peduzzi · 1 shared paper, 2025 · strength 0.20
- Radmila Lemstrová · 3 shared papers, 2017 · strength 0.15
- Paper Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. DOI 10.1038/tpj.2016.55↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Martin Oliverius · 3 shared papers, 2017 · strength 0.15
- Paper Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. DOI 10.1038/tpj.2016.55↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Zdeněk Kala · 3 shared papers, 2017 · strength 0.15
- Paper Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. DOI 10.1038/tpj.2016.55↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Beatrice Mohelníková-Duchoňová · 3 shared papers, 2017 · strength 0.15
- Paper Hedgehog pathway overexpression in pancreatic cancer is abrogated by new-generation taxoid SB-T-1216. DOI 10.1038/tpj.2016.55↗
- Paper Dysregulation of KRAS signaling in pancreatic cancer is not associated with KRAS mutations and outcome. DOI 10.3892/ol.2017.6946↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Jan Hlavsa · 2 shared papers, 2017 · strength 0.11
- R Havlík · 1 shared paper, 2017 · strength 0.05
- Martin Loveček · 1 shared paper, 2017 · strength 0.05
- Bohuslav Melichar · 1 shared paper, 2017 · strength 0.05
- Viktor Hlaváč · 1 shared paper, 2017 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.