ResearcherGuangdong Academy of Medical SciencesActive in the last 2 years
Liangtang Zeng
Matched on OpenAlex author ID A5091797627. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 13
2023: 2 papers · 2024: none · 2025: 4 papers
Papers 6
- Life's Essential 8, genetic susceptibility, and risk of incident pancreatic cancer: A prospective cohort study. First author DOI 10.1002/ijc.35184↗
- Combining genetic and non-genetic factors to predict the risk of pancreatic cancer in patients with new-onset diabetes mellitus. Co-author DOI 10.1186/s12916-025-04048-4↗
- EIF4E-mediated biogenesis of circPHF14 promotes the growth and metastasis of pancreatic ductal adenocarcinoma via Wnt/β-catenin pathway. Co-author DOI 10.1186/s12943-025-02262-5↗
- RBMS1-mediates the biogenesis of circNFIB promotes perineural invasion of pancreatic ductal adenocarcinoma via the L1CAM/MAPK pathway. Co-author DOI 10.7150/thno.112753↗
- Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort study in the UK Biobank. Co-author DOI 10.1186/s12916-023-03202-0↗
- Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis and Immunotherapy Response. First author DOI 10.3390/cancers15051442↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Zhuo Kui Wu · 5 shared papers, 2023–2025 · strength 1.40
- Paper Combining genetic and non-genetic factors to predict the risk of pancreatic cancer in patients with new-onset diabetes … DOI 10.1186/s12916-025-04048-4↗
- Paper EIF4E-mediated biogenesis of circPHF14 promotes the growth and metastasis of pancreatic ductal adenocarcinoma via Wnt/β… DOI 10.1186/s12943-025-02262-5↗
- Paper RBMS1-mediates the biogenesis of circNFIB promotes perineural invasion of pancreatic ductal adenocarcinoma via the L1CA… DOI 10.7150/thno.112753↗
- Rufu Chen · 4 shared papers, 2023–2025 · strength 1.10
- Paper Combining genetic and non-genetic factors to predict the risk of pancreatic cancer in patients with new-onset diabetes … DOI 10.1186/s12916-025-04048-4↗
- Paper Life's Essential 8, genetic susceptibility, and risk of incident pancreatic cancer: A prospective cohort study. DOI 10.1002/ijc.35184↗
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0↗
- Yu Zhou · 3 shared papers, 2023–2025 · strength 0.80
- Paper Combining genetic and non-genetic factors to predict the risk of pancreatic cancer in patients with new-onset diabetes … DOI 10.1186/s12916-025-04048-4↗
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442↗
- Zhou Fang · 2 shared papers, 2025 · strength 0.61
- Jiabin Yang · 2 shared papers, 2023 · strength 0.43
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442↗
- Yu Zhou · 1 shared paper, 2025 · strength 0.30
- Rufu Chen · 1 shared paper, 2025 · strength 0.30
- Chao Yu · 1 shared paper, 2025 · strength 0.30
- Zhuo Wu · 1 shared paper, 2025 · strength 0.30
- Dingwen Zhang · 1 shared paper, 2025 · strength 0.30
- Rufu Chen · 1 shared paper, 2025 · strength 0.30
- Leyi Huang · 1 shared paper, 2023 · strength 0.19
- Min Yu · 1 shared paper, 2023 · strength 0.19
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.