ResearcherGuangdong Academy of Medical SciencesActive in the last 2 years

Jiabin Yang

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0009-0001-9493-9064; OpenAlex author ID A5101480342. Sources: OpenAlex.

Their work

Papers: 7 (first author 1, last author 0)
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 19

Papers, trials and grants by year

2022: 1 paper · 2023: 3 papers · 2024: 2 papers · 2025: none · 2026: 1 paper

Papers 7

Machine learning-based clinical prediction model and multi-omics integration for assessing pancreatic cancer risk in new-onset diabetes. Co-author DOI 10.1186/s12967-026-07767-1 ↗
Endosomal Trafficking Bypassed by the RAB5B-CD109 Interplay Promotes Axonogenesis in KRAS-Mutant Pancreatic Cancer. Co-author DOI 10.1002/advs.202405092 ↗
Nudt21-mediated alternative polyadenylation of MZT1 3'UTR contributes to pancreatic cancer progression. First author DOI 10.1016/j.isci.2024.108822 ↗
Mutant KRAS Mediates circARFGEF2 Biogenesis to Promote Lymphatic Metastasis of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-22-3997 ↗
Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort study in the UK Biobank. Co-author DOI 10.1186/s12916-023-03202-0 ↗
Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis and Immunotherapy Response. Co-author DOI 10.3390/cancers15051442 ↗
KRAS mutant-driven SUMOylation controls extracellular vesicle transmission to trigger lymphangiogenesis in pancreatic cancer. Co-author DOI 10.1172/jci157644 ↗

Closest collaborators

The 19 strongest of 19 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Zhuo Kui Wu · 3 shared papers, 2023–2024 · strength 0.62
Rufu Chen · 3 shared papers, 2022–2024 · strength 0.54
Changhao Chen · 3 shared papers, 2022–2024 · strength 0.49
Yuming Luo · 3 shared papers, 2022–2024 · strength 0.49
Dingwen Zhang · 3 shared papers, 2022–2024 · strength 0.49
Leyi Huang · 2 shared papers, 2023–2024 · strength 0.43
- Paper Nudt21-mediated alternative polyadenylation of MZT1 3'UTR contributes to pancreatic cancer progression. DOI 10.1016/j.isci.2024.108822 ↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442 ↗
Liangtang Zeng · 2 shared papers, 2023 · strength 0.43
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0 ↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442 ↗
Rufu Chen · 2 shared papers, 2023 · strength 0.43
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0 ↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442 ↗
Yu Zhou · 2 shared papers, 2023 · strength 0.43
- Paper Association of genetic risk and lifestyle with pancreatic cancer and their age dependency: a large prospective cohort s… DOI 10.1186/s12916-023-03202-0 ↗
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442 ↗
Min Yu · 2 shared papers, 2023–2024 · strength 0.38
- Paper Leveraging Tumor Microenvironment Infiltration in Pancreatic Cancer to Identify Gene Signatures Related to Prognosis an… DOI 10.3390/cancers15051442 ↗
- Paper Endosomal Trafficking Bypassed by the RAB5B-CD109 Interplay Promotes Axonogenesis in KRAS-Mutant Pancreatic Cancer. DOI 10.1002/advs.202405092 ↗
Yao Kong · 2 shared papers, 2022–2023 · strength 0.30
- Paper Mutant KRAS Mediates circARFGEF2 Biogenesis to Promote Lymphatic Metastasis of Pancreatic Ductal Adenocarcinoma. DOI 10.1158/0008-5472.can-22-3997 ↗
- Paper KRAS mutant-driven SUMOylation controls extracellular vesicle transmission to trigger lymphangiogenesis in pancreatic c… DOI 10.1172/jci157644 ↗
Rufu Chen · 1 shared paper, 2026 · strength 0.26
- Paper Machine learning-based clinical prediction model and multi-omics integration for assessing pancreatic cancer risk in ne… DOI 10.1186/s12967-026-07767-1 ↗
Yu Zhou · 1 shared paper, 2024 · strength 0.24
- Paper Nudt21-mediated alternative polyadenylation of MZT1 3'UTR contributes to pancreatic cancer progression. DOI 10.1016/j.isci.2024.108822 ↗
Quanbo Zhou · 1 shared paper, 2024 · strength 0.24
- Paper Nudt21-mediated alternative polyadenylation of MZT1 3'UTR contributes to pancreatic cancer progression. DOI 10.1016/j.isci.2024.108822 ↗
Rufu Chen · 1 shared paper, 2024 · strength 0.19
- Paper Endosomal Trafficking Bypassed by the RAB5B-CD109 Interplay Promotes Axonogenesis in KRAS-Mutant Pancreatic Cancer. DOI 10.1002/advs.202405092 ↗
Chao Yu · 1 shared paper, 2024 · strength 0.19
- Paper Endosomal Trafficking Bypassed by the RAB5B-CD109 Interplay Promotes Axonogenesis in KRAS-Mutant Pancreatic Cancer. DOI 10.1002/advs.202405092 ↗
Zhou Fang · 1 shared paper, 2024 · strength 0.19
- Paper Endosomal Trafficking Bypassed by the RAB5B-CD109 Interplay Promotes Axonogenesis in KRAS-Mutant Pancreatic Cancer. DOI 10.1002/advs.202405092 ↗
Shangyou Zheng · 1 shared paper, 2023 · strength 0.16
- Paper Mutant KRAS Mediates circARFGEF2 Biogenesis to Promote Lymphatic Metastasis of Pancreatic Ductal Adenocarcinoma. DOI 10.1158/0008-5472.can-22-3997 ↗
Zhihua Li · 1 shared paper, 2022 · strength 0.14
- Paper KRAS mutant-driven SUMOylation controls extracellular vesicle transmission to trigger lymphangiogenesis in pancreatic c… DOI 10.1172/jci157644 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.