Pancreatic Cancer Research

ResearcherShanghai UniversityActive in the last 2 years

Yanyi Xiao

Matched on OpenAlex author ID A5103950557. Sources: OpenAlex.

Their work

Papers
13 (first author 7, last author 2)
Trials
0
Grants
0
Labs led
1
Collaborators
14
Papers, trials and grants by year

2020: 1 paper · 2021: 5 papers · 2022: 3 papers · 2023: none · 2024: 3 papers · 2025: 1 paper

Papers 13

  1. Correction: Chloroxine inhibits pancreatic cancer progression through targeted antagonization of the PI3K/AKT/mTOR signaling pathway. First author DOI 10.1007/s12094-025-03997-9↗
  2. Chloroxine inhibits pancreatic cancer progression through targeted antagonization of the PI3K/AKT/mTOR signaling pathway. First author DOI 10.1007/s12094-023-03328-w↗
  3. Lymphocyte antigen 6 family member E suppresses apoptosis and promotes pancreatic cancer growth and migration via Wnt/β-catenin pathway activation. Last author DOI 10.1038/s41598-024-70764-1↗
  4. Author Correction: Lymphocyte antigen 6 family member E suppresses apoptosis and promotes pancreatic cancer growth and migration via Wnt/β-catenin pathway acti… Last author DOI 10.1038/s41598-024-73144-x↗
  5. The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, reduce immune evasion ability and suppress p… Co-author DOI 10.1038/s41419-022-04573-7↗
  6. Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, reduce immune evasion ability an… Co-author DOI 10.1038/s41419-022-04705-z↗
  7. Inhibition of STAT3Y705 phosphorylation by Stattic suppresses proliferation and induces mitochondrial-dependent apoptosis in pancreatic cancer cells. First author DOI 10.1038/s41420-022-00922-9↗
  8. Quercetin suppresses pancreatic ductal adenocarcinoma progression via inhibition of SHH and TGF-β/Smad signaling pathways. Co-author DOI 10.1007/s10565-020-09562-0↗
  9. The anti-dysenteric drug fraxetin enhances anti-tumor efficacy of gemcitabine and suppresses pancreatic cancer development by antagonizing STAT3 activation. First author DOI 10.18632/aging.203301↗
  10. Fisetin inhibits the proliferation, migration and invasion of pancreatic cancer by targeting PI3K/AKT/mTOR signaling. Co-author DOI 10.18632/aging.203713↗
  11. The isoflavone puerarin exerts anti-tumor activity in pancreatic ductal adenocarcinoma by suppressing mTOR-mediated glucose metabolism. First author DOI 10.18632/aging.203725↗
  12. Pharmacological Inhibition of STAT3 Attenuates Pancreatic Cancer Cell Invasion and Migration By Interacting with TGF-Β/Smad Signaling First author DOI 10.21203/rs.3.rs-1055773/v1↗
  13. The Anti-Dysenteric Drug Fraxetin Enhances Anti-Tumor Efficacy of Gemcitabine and Suppresses Pancreatic Cancer Development by Antagonizing STAT3 Activation First author DOI 10.21203/rs.3.rs-125318/v1↗

Closest collaborators

The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Qiyu Zhang · 8 shared papers, 2020–2025 · strength 1.31
  2. Hangcheng Guo · 8 shared papers, 2020–2022 · strength 1.05
  3. Yongheng Bai · 8 shared papers, 2020–2022 · strength 1.05
  4. Miaomiao Lin · 4 shared papers, 2022–2025 · strength 0.83
  5. Hengyue Zhu · 6 shared papers, 2020–2022 · strength 0.79
  6. Yangyang Guo · 6 shared papers, 2020–2022 · strength 0.79
  7. Kenan Cen · 2 shared papers, 2024 · strength 0.56
  8. Zhe Chen · 2 shared papers, 2024–2025 · strength 0.52
  9. Jiawei Chen · 2 shared papers, 2021–2022 · strength 0.26
  10. Qinbo Chen · 2 shared papers, 2021–2022 · strength 0.26
  11. Junjie Deng · 2 shared papers, 2020–2021 · strength 0.23
  12. Linxiao Sun · 1 shared paper, 2021 · strength 0.14
  13. Xiangyang Lin · 1 shared paper, 2021 · strength 0.13
  14. Yunfeng Shan · 1 shared paper, 2021 · strength 0.13

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.