ResearcherWenzhou Medical UniversityNo recent work listed
Yangyang Guo
Matched on ORCID iD 0009-0002-0238-6958; OpenAlex author ID A5045485102. Sources: OpenAlex.
Their work
- Papers
- 13
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 23
2019: 1 paper · 2020: 4 papers · 2021: 5 papers · 2022: 2 papers · 2023: 1 paper
Papers 13
- STAT3 inhibition enhances gemcitabine sensitivity in pancreatic cancer by suppressing EMT, immune escape and inducing oxidative stress damage. Co-author DOI 10.1016/j.intimp.2023.110709↗
- The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, reduce immune evasion ability and suppress p… Co-author DOI 10.1038/s41419-022-04573-7↗
- Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, reduce immune evasion ability an… Co-author DOI 10.1038/s41419-022-04705-z↗
- Quercetin suppresses pancreatic ductal adenocarcinoma progression via inhibition of SHH and TGF-β/Smad signaling pathways. Co-author DOI 10.1007/s10565-020-09562-0↗
- Correction to: Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic cancer. Co-author DOI 10.1186/s12906-021-03254-w↗
- NUDCD1 knockdown inhibits the proliferation, migration, and invasion of pancreatic cancer via the EMT process. Co-author DOI 10.18632/aging.203276↗
- The anti-dysenteric drug fraxetin enhances anti-tumor efficacy of gemcitabine and suppresses pancreatic cancer development by antagonizing STAT3 activation. Co-author DOI 10.18632/aging.203301↗
- The isoflavone puerarin exerts anti-tumor activity in pancreatic ductal adenocarcinoma by suppressing mTOR-mediated glucose metabolism. Co-author DOI 10.18632/aging.203725↗
- Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic cancer. Co-author DOI 10.1186/s12906-020-02976-7↗
- The Anti-Dysenteric Drug Fraxetin Enhances Anti-Tumor Efficacy of Gemcitabine and Suppresses Pancreatic Cancer Development by Antagonizing STAT3 Activation Co-author DOI 10.21203/rs.3.rs-125318/v1↗
- CC-223, NSC781406, and BGT226 Exerts a Cytotoxic Effect Against Pancreatic Cancer Cells via mTOR Signaling. Co-author DOI 10.3389/fphar.2020.580407↗
- Molecular Mechanism of Pancreatic Stellate Cells Activation in Chronic Pancreatitis and Pancreatic Cancer. Co-author DOI 10.7150/jca.38616↗
- The DNA methylation profile of non-coding RNAs improves prognosis prediction for pancreatic adenocarcinoma. Co-author DOI 10.1186/s12935-019-0828-8↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hengyue Zhu · 10 shared papers, 2020–2022 · strength 1.38
- Paper Correction to: Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic c… DOI 10.1186/s12906-021-03254-w↗
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Yongheng Bai · 8 shared papers, 2020–2023 · strength 1.11
- Paper STAT3 inhibition enhances gemcitabine sensitivity in pancreatic cancer by suppressing EMT, immune escape and inducing o… DOI 10.1016/j.intimp.2023.110709↗
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Hangcheng Guo · 7 shared papers, 2020–2023 · strength 0.97
- Paper STAT3 inhibition enhances gemcitabine sensitivity in pancreatic cancer by suppressing EMT, immune escape and inducing o… DOI 10.1016/j.intimp.2023.110709↗
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Yanyi Xiao · 6 shared papers, 2020–2022 · strength 0.79
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Paper The isoflavone puerarin exerts anti-tumor activity in pancreatic ductal adenocarcinoma by suppressing mTOR-mediated glu… DOI 10.18632/aging.203725↗
- Qiyu Zhang · 4 shared papers, 2020–2022 · strength 0.53
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Paper The anti-dysenteric drug fraxetin enhances anti-tumor efficacy of gemcitabine and suppresses pancreatic cancer developm… DOI 10.18632/aging.203301↗
- Linxiao Sun · 3 shared papers, 2020–2021 · strength 0.45
- Paper Correction to: Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic c… DOI 10.1186/s12906-021-03254-w↗
- Paper Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic cancer. DOI 10.1186/s12906-020-02976-7↗
- Paper CC-223, NSC781406, and BGT226 Exerts a Cytotoxic Effect Against Pancreatic Cancer Cells via mTOR Signaling. DOI 10.3389/fphar.2020.580407↗
- Cheng Wang · 3 shared papers, 2020–2021 · strength 0.45
- Paper Correction to: Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic c… DOI 10.1186/s12906-021-03254-w↗
- Paper Chemopreventive effect of Betulinic acid via mTOR -Caspases/Bcl2/Bax apoptotic signaling in pancreatic cancer. DOI 10.1186/s12906-020-02976-7↗
- Paper CC-223, NSC781406, and BGT226 Exerts a Cytotoxic Effect Against Pancreatic Cancer Cells via mTOR Signaling. DOI 10.3389/fphar.2020.580407↗
- Miaomiao Lin · 2 shared papers, 2022 · strength 0.30
- Paper The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine activity, redu… DOI 10.1038/s41419-022-04573-7↗
- Paper Correction: The anthelmintic drug niclosamide induces GSK-β-mediated β-catenin degradation to potentiate gemcitabine ac… DOI 10.1038/s41419-022-04705-z↗
- Bicheng Chen · 2 shared papers, 2019–2020 · strength 0.24
- Junjie Deng · 2 shared papers, 2020–2021 · strength 0.23
- Paper The anti-dysenteric drug fraxetin enhances anti-tumor efficacy of gemcitabine and suppresses pancreatic cancer developm… DOI 10.18632/aging.203301↗
- Paper The Anti-Dysenteric Drug Fraxetin Enhances Anti-Tumor Efficacy of Gemcitabine and Suppresses Pancreatic Cancer Developm… DOI 10.21203/rs.3.rs-125318/v1↗
- Jiawei Chen · 1 shared paper, 2023 · strength 0.18
- Guihua Jin · 1 shared paper, 2020 · strength 0.15
- Zhensheng Cao · 1 shared paper, 2021 · strength 0.14
- Chunling Shi · 1 shared paper, 2021 · strength 0.14
- Hewei Zhang · 1 shared paper, 2020 · strength 0.13
- Xiangyang Lin · 1 shared paper, 2021 · strength 0.13
- Yunfeng Shan · 1 shared paper, 2021 · strength 0.13
- Jie Zhang · 1 shared paper, 2019 · strength 0.09
- Wanqing Weng · 1 shared paper, 2019 · strength 0.09
- Weiguo Huang · 1 shared paper, 2019 · strength 0.09
- Keqing Shi · 1 shared paper, 2019 · strength 0.09
- Yukai Xiang · 1 shared paper, 2019 · strength 0.09
- Mengtao Zhou · 1 shared paper, 2019 · strength 0.09
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.