ResearcherUniversity of Rome Tor VergataNo recent work listed
Gianfranco Delle Fave
Matched on OpenAlex author ID A5112256412. Sources: OpenAlex.
Their work
- Papers
- 16 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 62
2013: 3 papers · 2014: none · 2015: 2 papers · 2016: 3 papers · 2017: 4 papers · 2018: 2 papers · 2019: 2 papers
Papers 16
- Genetic determinants of telomere length and risk of pancreatic cancer: A PANDoRA study. Co-author DOI 10.1002/ijc.31928↗
- Germline BRCA2 K3326X and CHEK2 I157T mutations increase risk for sporadic pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.32127↗
- Results of surveillance in individuals at high-risk of pancreatic cancer: A systematic review and meta-analysis. Co-author DOI 10.1177/2050640617752182↗
- Co-treatment with gemcitabine and nab-paclitaxel exerts additive effects on pancreatic cancer cell death. Co-author DOI 10.3892/or.2018.6233↗
- Alternative polyadenylation of ZEB1 promotes its translation during genotoxic stress in pancreatic cancer cells. Co-author DOI 10.1038/cddis.2017.562↗
- Exclusive and Combined Use of Statins and Aspirin and the Risk of Pancreatic Cancer: a Case-Control Study. Co-author DOI 10.1038/s41598-017-13430-z↗
- SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. Co-author DOI 10.1038/srep43812↗
- The Neutrophil/Lymphocyte Ratio at Diagnosis Is Significantly Associated with Survival in Metastatic Pancreatic Cancer Patients. Co-author DOI 10.3390/ijms18040730↗
- Statin Use and Survival in Resectable Pancreatic Cancer: Confounders and Mechanisms. Last author DOI 10.1038/ajg.2015.412↗
- Modulation of PKM alternative splicing by PTBP1 promotes gemcitabine resistance in pancreatic cancer cells. Co-author DOI 10.1038/onc.2015.270↗
- Functional single nucleotide polymorphisms within the cyclin-dependent kinase inhibitor 2A/2B region affect pancreatic cancer risk. Co-author DOI 10.18632/oncotarget.10935↗
- TERT gene harbors multiple variants associated with pancreatic cancer susceptibility. Co-author DOI 10.1002/ijc.29590↗
- Early onset pancreatic cancer: risk factors, presentation and outcome. Last author DOI 10.1016/j.pan.2015.01.013↗
- Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDoRA) consortium. Co-author DOI 10.1016/j.dld.2012.09.014↗
- Gemcitabine triggers a pro-survival response in pancreatic cancer cells through activation of the MNK2/eIF4E pathway. Co-author DOI 10.1038/onc.2012.306↗
- Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. Co-author DOI 10.1158/1055-9965.epi-12-1182↗
Closest collaborators
The 25 strongest of 62 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Gabriele Capurso · 12 shared papers, 2013–2018 · strength 0.73
- Paper Statin Use and Survival in Resectable Pancreatic Cancer: Confounders and Mechanisms. DOI 10.1038/ajg.2015.412↗
- Paper Results of surveillance in individuals at high-risk of pancreatic cancer: A systematic review and meta-analysis. DOI 10.1177/2050640617752182↗
- Paper Co-treatment with gemcitabine and nab-paclitaxel exerts additive effects on pancreatic cancer cell death. DOI 10.3892/or.2018.6233↗
- Lívia Archibugi · 4 shared papers, 2015–2017 · strength 0.28
- Paper Statin Use and Survival in Resectable Pancreatic Cancer: Confounders and Mechanisms. DOI 10.1038/ajg.2015.412↗
- Paper The Neutrophil/Lymphocyte Ratio at Diagnosis Is Significantly Associated with Survival in Metastatic Pancreatic Cancer … DOI 10.3390/ijms18040730↗
- Paper Exclusive and Combined Use of Statins and Aspirin and the Risk of Pancreatic Cancer: a Case-Control Study. DOI 10.1038/s41598-017-13430-z↗
- Claudio Sette · 4 shared papers, 2013–2018 · strength 0.27
- Paper Co-treatment with gemcitabine and nab-paclitaxel exerts additive effects on pancreatic cancer cell death. DOI 10.3892/or.2018.6233↗
- Paper Alternative polyadenylation of ZEB1 promotes its translation during genotoxic stress in pancreatic cancer cells. DOI 10.1038/cddis.2017.562↗
- Paper Modulation of PKM alternative splicing by PTBP1 promotes gemcitabine resistance in pancreatic cancer cells. DOI 10.1038/onc.2015.270↗
- Ilaria Passacantilli · 3 shared papers, 2016–2018 · strength 0.23
- Paper Co-treatment with gemcitabine and nab-paclitaxel exerts additive effects on pancreatic cancer cell death. DOI 10.3892/or.2018.6233↗
- Paper Alternative polyadenylation of ZEB1 promotes its translation during genotoxic stress in pancreatic cancer cells. DOI 10.1038/cddis.2017.562↗
- Paper Modulation of PKM alternative splicing by PTBP1 promotes gemcitabine resistance in pancreatic cancer cells. DOI 10.1038/onc.2015.270↗
- Roberto Valente · 4 shared papers, 2015–2017 · strength 0.22
- Paper The Neutrophil/Lymphocyte Ratio at Diagnosis Is Significantly Associated with Survival in Metastatic Pancreatic Cancer … DOI 10.3390/ijms18040730↗
- Paper Exclusive and Combined Use of Statins and Aspirin and the Risk of Pancreatic Cancer: a Case-Control Study. DOI 10.1038/s41598-017-13430-z↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Marianna Signoretti · 3 shared papers, 2015–2018 · strength 0.21
- Paper Results of surveillance in individuals at high-risk of pancreatic cancer: A systematic review and meta-analysis. DOI 10.1177/2050640617752182↗
- Paper The Neutrophil/Lymphocyte Ratio at Diagnosis Is Significantly Associated with Survival in Metastatic Pancreatic Cancer … DOI 10.3390/ijms18040730↗
- Paper Early onset pancreatic cancer: risk factors, presentation and outcome. DOI 10.1016/j.pan.2015.01.013↗
- Matteo Piciucchi · 3 shared papers, 2015–2017 · strength 0.18
- Paper The Neutrophil/Lymphocyte Ratio at Diagnosis Is Significantly Associated with Survival in Metastatic Pancreatic Cancer … DOI 10.3390/ijms18040730↗
- Paper Exclusive and Combined Use of Statins and Aspirin and the Risk of Pancreatic Cancer: a Case-Control Study. DOI 10.1038/s41598-017-13430-z↗
- Paper Early onset pancreatic cancer: risk factors, presentation and outcome. DOI 10.1016/j.pan.2015.01.013↗
- Valentina Panzeri · 2 shared papers, 2017–2018 · strength 0.17
- Sara Calabretta · 2 shared papers, 2013–2016 · strength 0.10
- Marco J. Bruno · 1 shared paper, 2018 · strength 0.10
- Nathalia A. Giese · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Maria Gazouli · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Federico Canzian · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Ľudmila Vodičková · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Pavel Vodička · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Renata Talar‐Wojnarowska · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Daniele Campa · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Stefano Landi · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Pavel Souček · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Raffaele Pezzilli · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Cosmeri Rizzato · 3 shared papers, 2013–2017 · strength 0.09
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Lack of replication of seven pancreatic cancer susceptibility loci identified in two Asian populations. DOI 10.1158/1055-9965.epi-12-1182↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Beatrice Mohelníková-Duchoňová · 2 shared papers, 2013–2017 · strength 0.07
- Patrick Maisonneuve · 1 shared paper, 2017 · strength 0.07
- Michèle Milella · 1 shared paper, 2017 · strength 0.07
- Paola Fogar · 2 shared papers, 2013–2017 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.