ResearcherUniversity of PaduaNo recent work listed
Paola Fogar
Matched on OpenAlex author ID A5051237706. Sources: OpenAlex.
Their work
- Papers
- 13 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 50
2011: 2 papers · 2012: none · 2013: 3 papers · 2014: 1 paper · 2015: 1 paper · 2016: 1 paper · 2017: 2 papers · 2018: 2 papers · 2019: 1 paper
Papers 13
- Germline BRCA2 K3326X and CHEK2 I157T mutations increase risk for sporadic pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.32127↗
- Do pancreatic cancer and chronic pancreatitis share the same genetic risk factors? A PANcreatic Disease ReseArch (PANDoRA) consortium investigation. Co-author DOI 10.1002/ijc.31047↗
- Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neoplasms. Co-author DOI 10.1093/carcin/bgx150↗
- SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. Co-author DOI 10.1038/srep43812↗
- PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related axis. Co-author DOI 10.18632/oncotarget.20863↗
- Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and prognosis of PDAC-associated diabetes mell… Co-author DOI 10.1016/j.cca.2016.02.018↗
- TERT gene harbors multiple variants associated with pancreatic cancer susceptibility. Co-author DOI 10.1002/ijc.29590↗
- Inflammation and pancreatic cancer: molecular and functional interactions between S100A8, S100A9, NT-S100A8 and TGFβ1. Co-author DOI 10.1186/1478-811x-12-20↗
- Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDoRA) consortium. Co-author DOI 10.1016/j.dld.2012.09.014↗
- Usefulness of MALDI-TOF/MS identification of low-MW fragments in sera for the differential diagnosis of pancreatic cancer. Co-author DOI 10.1097/mpa.0b013e318273096c↗
- The S100A8/A9 complex reduces CTLA4 expression by immature myeloid cells: Implications for pancreatic cancer-driven immunosuppression. First author DOI 10.4161/onci.24441↗
- Altered intracellular calcium fluxes in pancreatic cancer induced diabetes mellitus: Relevance of the S100A8 N-terminal peptide (NT-S100A8). Co-author DOI 10.1002/jcp.22355↗
- Pancreatic cancer alters human CD4+ T lymphocyte function: a piece in the immune evasion puzzle. Co-author DOI 10.1097/mpa.0b013e31822077b8↗
Closest collaborators
The 25 strongest of 50 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Daniela Basso · 9 shared papers, 2011–2018 · strength 0.35
- Paper The S100A8/A9 complex reduces CTLA4 expression by immature myeloid cells: Implications for pancreatic cancer-driven imm… DOI 10.4161/onci.24441↗
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related ax… DOI 10.18632/oncotarget.20863↗
- Mario Plebani · 8 shared papers, 2011–2017 · strength 0.30
- Paper The S100A8/A9 complex reduces CTLA4 expression by immature myeloid cells: Implications for pancreatic cancer-driven imm… DOI 10.4161/onci.24441↗
- Paper PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related ax… DOI 10.18632/oncotarget.20863↗
- Paper Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and pr… DOI 10.1016/j.cca.2016.02.018↗
- Sergio Pedrazzoli · 8 shared papers, 2011–2017 · strength 0.28
- Paper PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related ax… DOI 10.18632/oncotarget.20863↗
- Paper Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and pr… DOI 10.1016/j.cca.2016.02.018↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Andrea Padoan · 6 shared papers, 2011–2017 · strength 0.21
- Paper PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related ax… DOI 10.18632/oncotarget.20863↗
- Paper Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and pr… DOI 10.1016/j.cca.2016.02.018↗
- Paper Inflammation and pancreatic cancer: molecular and functional interactions between S100A8, S100A9, NT-S100A8 and TGFβ1. DOI 10.1186/1478-811x-12-20↗
- Stefania Moz · 5 shared papers, 2011–2017 · strength 0.19
- Paper PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related ax… DOI 10.18632/oncotarget.20863↗
- Paper Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and pr… DOI 10.1016/j.cca.2016.02.018↗
- Paper Inflammation and pancreatic cancer: molecular and functional interactions between S100A8, S100A9, NT-S100A8 and TGFβ1. DOI 10.1186/1478-811x-12-20↗
- Cosimo Sperti · 3 shared papers, 2013–2017 · strength 0.12
- Paper Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and pr… DOI 10.1016/j.cca.2016.02.018↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Usefulness of MALDI-TOF/MS identification of low-MW fragments in sera for the differential diagnosis of pancreatic canc… DOI 10.1097/mpa.0b013e318273096c↗
- Maria Gazouli · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Federico Canzian · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Niccola Funel · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Renata Talar‐Wojnarowska · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Ewa I. Malecka-Panas · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Gabriele Capurso · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Daniele Campa · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Stefano Landi · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Francesca Tavano · 3 shared papers, 2013–2018 · strength 0.12
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper SLC22A3 polymorphisms do not modify pancreatic cancer risk, but may influence overall patient survival. DOI 10.1038/srep43812↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Thilo Hackert · 2 shared papers, 2017–2018 · strength 0.10
- Rita Teresa Lawlor · 2 shared papers, 2017–2018 · strength 0.10
- Oliver Strobel · 2 shared papers, 2017–2018 · strength 0.10
- Angelo Andriulli · 2 shared papers, 2013–2018 · strength 0.08
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- John P. Neoptolemos · 2 shared papers, 2013–2018 · strength 0.08
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- William Greenhalf · 2 shared papers, 2013–2018 · strength 0.08
- Paper Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neo… DOI 10.1093/carcin/bgx150↗
- Paper Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDo… DOI 10.1016/j.dld.2012.09.014↗
- Gianfranco Delle Fave · 2 shared papers, 2013–2017 · strength 0.07
- Nathalia A. Giese · 2 shared papers, 2013–2017 · strength 0.07
- Andrea Mambrini · 2 shared papers, 2013–2017 · strength 0.07
- Maurizio Cantore · 2 shared papers, 2013–2017 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.