Pancreatic Cancer Research

ResearcherUniversity of PaduaActive in the last 2 years

Daniela Basso

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0000-0001-8745-6171; OpenAlex author ID A5037249662. Sources: OpenAlex.

Their work

Papers
35 (first author 3, last author 3)
Trials
0
Grants
0
Labs led
1
Collaborators
71
Papers, trials and grants by year

2011: 2 papers · 2012: none · 2013: 3 papers · 2014: 2 papers · 2015: none · 2016: 4 papers · 2017: 2 papers · 2018: 1 paper · 2019: 3 papers · 2020: 1 paper · 2021: 6 papers · 2022: 1 paper · 2023: 6 papers · 2024: 2 papers · 2025: 2 papers

Papers 35

  1. Polymorphisms within autophagy-related genes as susceptibility biomarkers for pancreatic cancer: A meta-analysis of three large European cohorts and functional… Co-author DOI 10.1002/ijc.35196↗
  2. A pleiotropy scan to discover new susceptibility loci for pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/mutage/geae012↗
  3. Potential association between PSCA rs2976395 functional variant and pancreatic cancer risk. Co-author DOI 10.1002/ijc.35046↗
  4. Polymorphisms in transcription factor binding sites and enhancer regions and pancreatic ductal adenocarcinoma risk. Co-author DOI 10.1186/s40246-024-00576-x↗
  5. Association between a polymorphic variant in the CDKN2B-AS1/ANRIL gene and pancreatic cancer risk. Co-author DOI 10.1002/ijc.34383↗
  6. The PANcreatic Disease ReseArch (PANDoRA) consortium: Ten years' experience of association studies to understand the genetic architecture of pancreatic cancer. Co-author DOI 10.1016/j.critrevonc.2023.104020↗
  7. Genetic and non-genetic risk factors for early-onset pancreatic cancer. Co-author DOI 10.1016/j.dld.2023.02.023↗
  8. A scan of all coding region variants of the human genome, identifies 13q12.2-rs9579139 and 15q24.1-rs2277598 as novel risk loci for pancreatic ductal adenocarc… Co-author DOI 10.1093/carcin/bgad056↗
  9. Polymorphic variants involved in methylation regulation: a strategy to discover risk loci for pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/jmg-2022-108910↗
  10. Exploring the Neandertal legacy of pancreatic ductal adenocarcinoma risk in Eurasians. Co-author DOI 10.1186/s40659-023-00457-y↗
  11. Common variability in oestrogen-related genes and pancreatic ductal adenocarcinoma risk in women. Co-author DOI 10.1038/s41598-022-22973-9↗
  12. Associations between pancreatic expression quantitative traits and risk of pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/carcin/bgab057↗
  13. Polygenic and multifactorial scores for pancreatic ductal adenocarcinoma risk prediction. Co-author DOI 10.1136/jmedgenet-2020-106961↗
  14. Genetic Polymorphisms Involved in Mitochondrial Metabolism and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-21-0353↗
  15. Association of Genetic Variants Affecting microRNAs and Pancreatic Cancer Risk. Co-author DOI 10.3389/fgene.2021.693933↗
  16. Identification of Recessively Inherited Genetic Variants Potentially Linked to Pancreatic Cancer Risk. Co-author DOI 10.3389/fonc.2021.771312↗
  17. Serum miRNA Profiling for Early PDAC Diagnosis and Prognosis: A Retrospective Study. Last author DOI 10.3390/biomedicines9070845↗
  18. Vitamin D Prevents Pancreatic Cancer-Induced Apoptosis Signaling of Inflammatory Cells. Last author DOI 10.3390/biom10071055↗
  19. Genetic determinants of telomere length and risk of pancreatic cancer: A PANDoRA study. Co-author DOI 10.1002/ijc.31928↗
  20. Germline BRCA2 K3326X and CHEK2 I157T mutations increase risk for sporadic pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.32127↗
  21. Inflammation and Pancreatic Cancer: Focus on Metabolism, Cytokines, and Immunity. Last author DOI 10.3390/ijms20030676↗
  22. Common genetic variants associated with pancreatic adenocarcinoma may also modify risk of pancreatic neuroendocrine neoplasms. Co-author DOI 10.1093/carcin/bgx150↗
  23. Lack of Association for Reported Endocrine Pancreatic Cancer Risk Loci in the PANDoRA Consortium. Co-author DOI 10.1158/1055-9965.epi-17-0075↗
  24. PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related axis. Co-author DOI 10.18632/oncotarget.20863↗
  25. Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 supports diagnosis and prognosis of PDAC-associated diabetes mell… First author DOI 10.1016/j.cca.2016.02.018↗
  26. Functional single nucleotide polymorphisms within the cyclin-dependent kinase inhibitor 2A/2B region affect pancreatic cancer risk. Co-author DOI 10.18632/oncotarget.10935↗
  27. Three new pancreatic cancer susceptibility signals identified on chromosomes 1q32.1, 5p15.33 and 8q24.21. Co-author DOI 10.18632/oncotarget.11041↗
  28. SMAD4 loss enables EGF, TGFβ1 and S100A8/A9 induced activation of critical pathways to invasion in human pancreatic adenocarcinoma cells. First author DOI 10.18632/oncotarget.12068↗
  29. Genome-wide association study identifies multiple susceptibility loci for pancreatic cancer. Co-author DOI 10.1038/ng.3052↗
  30. Inflammation and pancreatic cancer: molecular and functional interactions between S100A8, S100A9, NT-S100A8 and TGFβ1. Co-author DOI 10.1186/1478-811x-12-20↗
  31. Genetic susceptibility to pancreatic cancer and its functional characterisation: the PANcreatic Disease ReseArch (PANDoRA) consortium. Co-author DOI 10.1016/j.dld.2012.09.014↗
  32. Usefulness of MALDI-TOF/MS identification of low-MW fragments in sera for the differential diagnosis of pancreatic cancer. Co-author DOI 10.1097/mpa.0b013e318273096c↗
  33. The S100A8/A9 complex reduces CTLA4 expression by immature myeloid cells: Implications for pancreatic cancer-driven immunosuppression. Co-author DOI 10.4161/onci.24441↗
  34. Altered intracellular calcium fluxes in pancreatic cancer induced diabetes mellitus: Relevance of the S100A8 N-terminal peptide (NT-S100A8). Co-author DOI 10.1002/jcp.22355↗
  35. Pancreatic cancer alters human CD4+ T lymphocyte function: a piece in the immune evasion puzzle. First author DOI 10.1097/mpa.0b013e31822077b8↗

Closest collaborators

The 25 strongest of 71 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Mario Plebani · 12 shared papers, 2011–2021 · strength 0.79
  2. Andrea Padoan · 8 shared papers, 2011–2019 · strength 0.42
  3. Stefania Moz · 7 shared papers, 2011–2020 · strength 0.38
  4. Paola Fogar · 9 shared papers, 2011–2018 · strength 0.35
  5. Federico Canzian · 5 shared papers, 2013–2023 · strength 0.33
  6. John P. Neoptolemos · 5 shared papers, 2013–2023 · strength 0.33
  7. Gabriele Capurso · 5 shared papers, 2013–2023 · strength 0.33
  8. Daniele Campa · 5 shared papers, 2013–2023 · strength 0.33
  9. Francesca Tavano · 5 shared papers, 2013–2023 · strength 0.33
  10. Rita Teresa Lawlor · 4 shared papers, 2017–2023 · strength 0.31
  11. Sergio Pedrazzoli · 8 shared papers, 2011–2017 · strength 0.28
  12. Lívia Archibugi · 3 shared papers, 2018–2023 · strength 0.26
  13. Stefano Landi · 4 shared papers, 2013–2023 · strength 0.23
  14. Cosimo Sperti · 3 shared papers, 2013–2021 · strength 0.23
  15. Pavel Souček · 3 shared papers, 2013–2023 · strength 0.23
  16. Raffaele Pezzilli · 3 shared papers, 2013–2023 · strength 0.23
  17. Renata Talar‐Wojnarowska · 4 shared papers, 2013–2022 · strength 0.22
  18. Thilo Hackert · 3 shared papers, 2017–2023 · strength 0.21
  19. Giulia Martina Cavestro · 2 shared papers, 2022–2023 · strength 0.20
  20. Giuseppe Vanella · 2 shared papers, 2022–2023 · strength 0.20
  21. Luca Morelli · 2 shared papers, 2022–2023 · strength 0.20
  22. Manuel Gentiluomo · 2 shared papers, 2022–2023 · strength 0.20
  23. Jakob Robert Izbicki · 2 shared papers, 2022–2023 · strength 0.20
  24. Casper H.J. van Eijck · 2 shared papers, 2022–2023 · strength 0.20
  25. Hermann Brenner · 2 shared papers, 2022–2023 · strength 0.20

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.