ResearcherQueen Mary University of LondonActive in the last 2 years
Petra Jagušt
Matched on ORCID iD 0000-0001-5291-251X; OpenAlex author ID A5006255380. Sources: OpenAlex.
Their work
- Papers
- 6
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 13
2015: 1 paper · 2016: none · 2017: none · 2018: none · 2019: none · 2020: 1 paper · 2021: 2 papers · 2022: none · 2023: none · 2024: 1 paper · 2025: 1 paper
Papers 6
- PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3475↗
- Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. Co-author DOI 10.1186/s12967-024-05598-6↗
- PPAR-delta acts as a metabolic master checkpoint for metastasis in pancreatic cancer Co-author DOI 10.1101/2021.11.15.468579↗
- Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invasive Potential. Co-author DOI 10.3390/cancers13040698↗
- Glutathione metabolism is essential for self-renewal and chemoresistance of pancreatic cancer stem cells. Co-author DOI 10.4252/wjsc.v12.i11.1410↗
- MYC/PGC-1α Balance Determines the Metabolic Phenotype and Plasticity of Pancreatic Cancer Stem Cells. Co-author DOI 10.1016/j.cmet.2015.08.015↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Patricia Sancho · 6 shared papers, 2015–2025 · strength 0.78
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Glutathione metabolism is essential for self-renewal and chemoresistance of pancreatic cancer stem cells. DOI 10.4252/wjsc.v12.i11.1410↗
- Christopher Heeschen · 6 shared papers, 2015–2025 · strength 0.78
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Glutathione metabolism is essential for self-renewal and chemoresistance of pancreatic cancer stem cells. DOI 10.4252/wjsc.v12.i11.1410↗
- Sarah Courtois · 4 shared papers, 2021–2025 · strength 0.59
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invas… DOI 10.3390/cancers13040698↗
- Bruno Sáinz · 5 shared papers, 2015–2025 · strength 0.58
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Glutathione metabolism is essential for self-renewal and chemoresistance of pancreatic cancer stem cells. DOI 10.4252/wjsc.v12.i11.1410↗
- Paper Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invas… DOI 10.3390/cancers13040698↗
- Pilar Espiau-Romera · 3 shared papers, 2021–2025 · strength 0.47
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper PPAR-delta acts as a metabolic master checkpoint for metastasis in pancreatic cancer DOI 10.1101/2021.11.15.468579↗
- Beatriz Parejo-Alonso · 3 shared papers, 2021–2025 · strength 0.39
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invas… DOI 10.3390/cancers13040698↗
- Paper PPAR-delta acts as a metabolic master checkpoint for metastasis in pancreatic cancer DOI 10.1101/2021.11.15.468579↗
- Sara Maria David Trabulo · 2 shared papers, 2021–2025 · strength 0.27
- Quan Zheng · 2 shared papers, 2021–2025 · strength 0.27
- Sonia Alcalá · 2 shared papers, 2020–2021 · strength 0.27
- Marta Mascaraque · 1 shared paper, 2024 · strength 0.20
- Matthias M. Gaida · 1 shared paper, 2025 · strength 0.18
- Andrés Gordo-Ortiz · 1 shared paper, 2025 · strength 0.18
- Jiajia Tang · 1 shared paper, 2021 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.