ResearcherInstituto de Investigación Sanitaria AragónActive in the last 2 years
Sarah Courtois
Matched on ORCID iD 0000-0001-8572-2499; OpenAlex author ID A5073891040. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 16
2020: 1 paper · 2021: 2 papers · 2022: none · 2023: 1 paper · 2024: 1 paper · 2025: 1 paper
Papers 6
- PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3475↗
- Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. First author DOI 10.1186/s12967-024-05598-6↗
- Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.biopha.2022.114162↗
- PPAR-delta acts as a metabolic master checkpoint for metastasis in pancreatic cancer Co-author DOI 10.1101/2021.11.15.468579↗
- Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invasive Potential. Co-author DOI 10.3390/cancers13040698↗
- Molecular and Metabolic Subtypes Correspondence for Pancreatic Ductal Adenocarcinoma Classification. First author DOI 10.3390/jcm9124128↗
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Patricia Sancho Andrés · 6 shared papers, 2020–2025 · strength 0.93
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ducta… DOI 10.1016/j.biopha.2022.114162↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Pilar Espiau-Romera · 5 shared papers, 2020–2025 · strength 0.81
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ducta… DOI 10.1016/j.biopha.2022.114162↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Christopher Heeschen · 5 shared papers, 2021–2025 · strength 0.77
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ducta… DOI 10.1016/j.biopha.2022.114162↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Beatriz Parejo-Alonso · 5 shared papers, 2020–2025 · strength 0.73
- Paper Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ducta… DOI 10.1016/j.biopha.2022.114162↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Molecular and Metabolic Subtypes Correspondence for Pancreatic Ductal Adenocarcinoma Classification. DOI 10.3390/jcm9124128↗
- Petra Jagušt · 4 shared papers, 2021–2025 · strength 0.59
- Paper Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells. DOI 10.1186/s12967-024-05598-6↗
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invas… DOI 10.3390/cancers13040698↗
- Bruno Sáinz · 3 shared papers, 2021–2025 · strength 0.39
- Paper PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3475↗
- Paper Inhibition of Mitochondrial Dynamics Preferentially Targets Pancreatic Cancer Cells with Enhanced Tumorigenic and Invas… DOI 10.3390/cancers13040698↗
- Paper PPAR-delta acts as a metabolic master checkpoint for metastasis in pancreatic cancer DOI 10.1101/2021.11.15.468579↗
- Sara Maria David Trabulo · 2 shared papers, 2021–2025 · strength 0.27
- Quan Zheng · 2 shared papers, 2021–2025 · strength 0.27
- Marta Mascaraque Checa · 1 shared paper, 2024 · strength 0.20
- Álvaro Curiel‐García · 1 shared paper, 2023 · strength 0.18
- Sladjana Zagorac · 1 shared paper, 2023 · strength 0.18
- Kenneth P. Olive · 1 shared paper, 2023 · strength 0.18
- Matthias M. Gaida · 1 shared paper, 2025 · strength 0.18
- Andrés Gordo-Ortiz · 1 shared paper, 2025 · strength 0.18
- Sonia Alcalá · 1 shared paper, 2021 · strength 0.12
- Jiajia Tang · 1 shared paper, 2021 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.