ResearcherSoochow UniversityActive in the last 2 years
Xuemin Chen
Matched on ORCID iD 0000-0001-7387-0662; OpenAlex author ID A5101957486. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 1, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 16
2016: 1 paper · 2017: 1 paper · 2018: 3 papers · 2019: none · 2020: none · 2021: none · 2022: 1 paper · 2023: none · 2024: none · 2025: none · 2026: 1 paper
Papers 7
- SUZ12 promotes pancreatic ductal adenocarcinoma progression and chemoresistance by epigenetically silencing NCOA4-induced ferroptosis. Last author DOI 10.3724/abbs.2025215↗
- NDUFA4 promotes cell proliferation by enhancing oxidative phosphorylation in pancreatic adenocarcinoma. Co-author DOI 10.1007/s10863-022-09949-0↗
- circZMYM2 Competed Endogenously with miR-335-5p to Regulate JMJD2C in Pancreatic Cancer. Co-author DOI 10.1159/000495868↗
- LncRNA DLX6-AS1 promoted cancer cell proliferation and invasion by attenuating the endogenous function of miR-181b in pancreatic cancer. First author DOI 10.1186/s12935-018-0643-7↗
- MiR-216b inhibits pancreatic cancer cell progression and promotes apoptosis by down-regulating KRAS. Co-author DOI 10.5114/aoms.2018.72564↗
- LncRNA HOTAIR acts a competing endogenous RNA to control the expression of notch3 via sponging miR-613 in pancreatic cancer. Co-author DOI 10.18632/oncotarget.16462↗
- Epigenetic inhibition of miR-663b by long non-coding RNA HOTAIR promotes pancreatic cancer cell proliferation via up-regulation of insulin-like growth factor 2. Co-author DOI 10.18632/oncotarget.13490↗
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Huihua Cai · 6 shared papers, 2016–2022 · strength 0.54
- Paper NDUFA4 promotes cell proliferation by enhancing oxidative phosphorylation in pancreatic adenocarcinoma. DOI 10.1007/s10863-022-09949-0↗
- Paper LncRNA DLX6-AS1 promoted cancer cell proliferation and invasion by attenuating the endogenous function of miR-181b in p… DOI 10.1186/s12935-018-0643-7↗
- Paper circZMYM2 Competed Endogenously with miR-335-5p to Regulate JMJD2C in Pancreatic Cancer. DOI 10.1159/000495868↗
- Yong An · 5 shared papers, 2016–2018 · strength 0.38
- Paper LncRNA DLX6-AS1 promoted cancer cell proliferation and invasion by attenuating the endogenous function of miR-181b in p… DOI 10.1186/s12935-018-0643-7↗
- Paper circZMYM2 Competed Endogenously with miR-335-5p to Regulate JMJD2C in Pancreatic Cancer. DOI 10.1159/000495868↗
- Paper MiR-216b inhibits pancreatic cancer cell progression and promotes apoptosis by down-regulating KRAS. DOI 10.5114/aoms.2018.72564↗
- Xiaowu Xu · 1 shared paper, 2026 · strength 0.27
- Borui Li · 1 shared paper, 2026 · strength 0.27
- Zheng Li · 1 shared paper, 2026 · strength 0.27
- Lei Yin · 1 shared paper, 2026 · strength 0.27
- Xinquan Wu · 2 shared papers, 2018–2022 · strength 0.24
- Di Wu · 2 shared papers, 2017–2022 · strength 0.22
- Yue Zhang · 1 shared paper, 2022 · strength 0.16
- Yong Yang · 2 shared papers, 2017–2018 · strength 0.16
- Paper LncRNA DLX6-AS1 promoted cancer cell proliferation and invasion by attenuating the endogenous function of miR-181b in p… DOI 10.1186/s12935-018-0643-7↗
- Paper LncRNA HOTAIR acts a competing endogenous RNA to control the expression of notch3 via sponging miR-613 in pancreatic ca… DOI 10.18632/oncotarget.16462↗
- Yong Jiang · 2 shared papers, 2017–2018 · strength 0.15
- Xiaozhou He · 2 shared papers, 2016–2017 · strength 0.12
- Paper LncRNA HOTAIR acts a competing endogenous RNA to control the expression of notch3 via sponging miR-613 in pancreatic ca… DOI 10.18632/oncotarget.16462↗
- Paper Epigenetic inhibition of miR-663b by long non-coding RNA HOTAIR promotes pancreatic cancer cell proliferation via up-re… DOI 10.18632/oncotarget.13490↗
- Xiaozhou He · 1 shared paper, 2018 · strength 0.09
- Baoqiang Wu · 1 shared paper, 2018 · strength 0.08
- B Y Luo · 1 shared paper, 2017 · strength 0.06
- Jie J. Yao · 1 shared paper, 2017 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.