Pancreatic Cancer Research

ResearcherSoochow UniversityNo recent work listed

Huihua Cai

Matched on ORCID iD 0000-0003-2558-7170; OpenAlex author ID A5101506088. Sources: OpenAlex.

Their work

Papers
11 (first author 2, last author 2)
Trials
0
Grants
0
Labs led
1
Collaborators
25
Papers, trials and grants by year

2011: 1 paper · 2012: none · 2013: none · 2014: none · 2015: 1 paper · 2016: 1 paper · 2017: 1 paper · 2018: 4 papers · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper

Papers 11

  1. NDUFA4 promotes cell proliferation by enhancing oxidative phosphorylation in pancreatic adenocarcinoma. Co-author DOI 10.1007/s10863-022-09949-0↗
  2. MicroRNA-519 inhibits hypoxia-induced tumorigenesis of pancreatic cancer by regulating immune checkpoint PD-L1. Last author DOI 10.3892/ol.2019.11234↗
  3. LncRNA SNHG14 potentiates pancreatic cancer progression via modulation of annexin A2 expression by acting as a competing endogenous RNA for miR-613. Co-author DOI 10.1111/jcmm.14467↗
  4. Identification of ENTPD8 and cytidine in pancreatic cancer by metabolomic and transcriptomic conjoint analysis. First author DOI 10.1111/cas.13733↗
  5. circZMYM2 Competed Endogenously with miR-335-5p to Regulate JMJD2C in Pancreatic Cancer. First author DOI 10.1159/000495868↗
  6. LncRNA DLX6-AS1 promoted cancer cell proliferation and invasion by attenuating the endogenous function of miR-181b in pancreatic cancer. Last author DOI 10.1186/s12935-018-0643-7↗
  7. MiR-216b inhibits pancreatic cancer cell progression and promotes apoptosis by down-regulating KRAS. Co-author DOI 10.5114/aoms.2018.72564↗
  8. LncRNA HOTAIR acts a competing endogenous RNA to control the expression of notch3 via sponging miR-613 in pancreatic cancer. Co-author DOI 10.18632/oncotarget.16462↗
  9. Epigenetic inhibition of miR-663b by long non-coding RNA HOTAIR promotes pancreatic cancer cell proliferation via up-regulation of insulin-like growth factor 2. Co-author DOI 10.18632/oncotarget.13490↗
  10. miR-615-5p is epigenetically inactivated and functions as a tumor suppressor in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/onc.2014.101↗
  11. Aberrant methylation frequency of TNFRSF10C promoter in pancreatic cancer cell lines. Co-author DOI 10.1016/s1499-3872(11)60014-3↗

Closest collaborators

The 25 strongest of 25 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Xuemin Chen · 6 shared papers, 2016–2022 · strength 0.54
  2. Yong An · 6 shared papers, 2016–2018 · strength 0.46
  3. Xinquan Wu · 4 shared papers, 2018–2022 · strength 0.41
  4. Yong Yang · 3 shared papers, 2017–2019 · strength 0.25
  5. Di Wu · 2 shared papers, 2017–2022 · strength 0.22
  6. Yue Zhang · 1 shared paper, 2022 · strength 0.16
  7. Yong Jiang · 2 shared papers, 2017–2018 · strength 0.15
  8. Kate Nong · 1 shared paper, 2020 · strength 0.13
  9. Xiaozhou He · 2 shared papers, 2016–2017 · strength 0.12
  10. Jie J. Yao · 2 shared papers, 2011–2017 · strength 0.09
  11. Peng‐cheng Deng · 1 shared paper, 2019 · strength 0.09
  12. Xiaozhou He · 1 shared paper, 2018 · strength 0.09
  13. Baoqiang Wu · 1 shared paper, 2018 · strength 0.08
  14. Xuemin Chen · 1 shared paper, 2018 · strength 0.08
  15. Yue Zhang · 1 shared paper, 2018 · strength 0.08
  16. Yi Miao · 2 shared papers, 2011–2015 · strength 0.07
  17. B Y Luo · 1 shared paper, 2017 · strength 0.06
  18. Yulan Zhu · 1 shared paper, 2015 · strength 0.04
  19. Keiko Shinjo · 1 shared paper, 2015 · strength 0.04
  20. Wen Gao · 1 shared paper, 2015 · strength 0.04
  21. Yumei Gu · 1 shared paper, 2015 · strength 0.04
  22. Min Tu · 1 shared paper, 2015 · strength 0.04
  23. Yutaka Kondo · 1 shared paper, 2015 · strength 0.04
  24. Han-Lin Zhao · 1 shared paper, 2011 · strength 0.03
  25. Wentao Gao · 1 shared paper, 2011 · strength 0.03

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.