ResearcherHenry Ford Health + Michigan State University Health SciencesActive in the last 2 years
Debora Barbosa Vendramini Costa
Matched on ORCID iD 0000-0001-7617-4945; OpenAlex author ID A5005941184, A5108352639; NIH RePORTER profile ID 16046441. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 2, last author 1)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 56
2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 4 papers · 2025: 1 paper · 2026: 3 papers, 1 grant
Papers 11
- ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppression. Co-author DOI 10.1038/s43018-026-01265-5↗
- Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Immune Microenvironments. Co-author DOI 10.1158/0008-5472.can-25-2748↗
- HMGA2 Expression in Pancreatic Ductal Adenocarcinoma Controls Tumor Microenvironment and Immunosuppression via IL1α Co-author DOI 10.64898/2026.10.02.754697↗
- Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. Co-author DOI 10.1158/1078-0432.ccr-24-2183↗
- Rethinking the Roles of Cancer-Associated Fibroblasts in Pancreatic Cancer. Last author DOI 10.1016/j.jcmgh.2024.01.022↗
- Pulsed low-dose-rate radiation (PLDR) reduces the tumor-promoting responses induced by conventional chemoradiation in pancreatic cancer-associated fibroblasts Co-author DOI 10.1101/2024.01.13.575510↗
- Netrin G1 Ligand is a new stromal immunomodulator that promotes pancreatic cancer Co-author DOI 10.1101/2024.05.15.594354↗
- Pancreatic Cancer-Associated Fibroblasts: Where Do We Go from Here? First author DOI 10.1158/0008-5472.can-24-2860↗
- Netrin-1 feedforward mechanism promotes pancreatic cancer liver metastasis via hepatic stellate cell activation, retinoid, and ELF3 signaling. Co-author DOI 10.1016/j.celrep.2023.113369↗
- NetrinG1+ cancer-associated fibroblasts generate unique extracellular vesicles that support the survival of pancreatic cancer cells under nutritional stress. Co-author DOI 10.1158/2767-9764.crc-21-0147↗
- Palladin isoforms 3 and 4 regulate cancer-associated fibroblast pro-tumor functions in pancreatic ductal adenocarcinoma. First author DOI 10.1038/s41598-021-82937-3↗
Grants 1
Closest collaborators
The 25 strongest of 56 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Ralph A. Francescone III · 9 shared papers, 2021–2026 · strength 1.77
- Paper Rethinking the Roles of Cancer-Associated Fibroblasts in Pancreatic Cancer. DOI 10.1016/j.jcmgh.2024.01.022↗
- Paper HMGA2 Expression in Pancreatic Ductal Adenocarcinoma Controls Tumor Microenvironment and Immunosuppression via IL1α DOI 10.64898/2026.10.02.754697↗
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Edna Cukierman · 6 shared papers, 2021–2026 · strength 1.03
- Paper Pancreatic Cancer-Associated Fibroblasts: Where Do We Go from Here? DOI 10.1158/0008-5472.can-24-2860↗
- Paper Pulsed low-dose-rate radiation (PLDR) reduces the tumor-promoting responses induced by conventional chemoradiation in p… DOI 10.1101/2024.01.13.575510↗
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Howard C. Crawford · 4 shared papers, 2024–2026 · strength 0.96
- Paper Rethinking the Roles of Cancer-Associated Fibroblasts in Pancreatic Cancer. DOI 10.1016/j.jcmgh.2024.01.022↗
- Paper HMGA2 Expression in Pancreatic Ductal Adenocarcinoma Controls Tumor Microenvironment and Immunosuppression via IL1α DOI 10.64898/2026.10.02.754697↗
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Janusz Franco‐Barraza · 5 shared papers, 2021–2026 · strength 0.81
- Paper Pulsed low-dose-rate radiation (PLDR) reduces the tumor-promoting responses induced by conventional chemoradiation in p… DOI 10.1101/2024.01.13.575510↗
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Netrin G1 Ligand is a new stromal immunomodulator that promotes pancreatic cancer DOI 10.1101/2024.05.15.594354↗
- Nina G. Steele · 4 shared papers, 2024–2026 · strength 0.76
- Paper HMGA2 Expression in Pancreatic Ductal Adenocarcinoma Controls Tumor Microenvironment and Immunosuppression via IL1α DOI 10.64898/2026.10.02.754697↗
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Tiffany Luong · 4 shared papers, 2022–2026 · strength 0.68
- Paper Pulsed low-dose-rate radiation (PLDR) reduces the tumor-promoting responses induced by conventional chemoradiation in p… DOI 10.1101/2024.01.13.575510↗
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Netrin G1 Ligand is a new stromal immunomodulator that promotes pancreatic cancer DOI 10.1101/2024.05.15.594354↗
- Charline Ogier · 4 shared papers, 2022–2026 · strength 0.60
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Netrin G1 Ligand is a new stromal immunomodulator that promotes pancreatic cancer DOI 10.1101/2024.05.15.594354↗
- Paper Netrin-1 feedforward mechanism promotes pancreatic cancer liver metastasis via hepatic stellate cell activation, retino… DOI 10.1016/j.celrep.2023.113369↗
- Ian M. Loveless · 3 shared papers, 2025–2026 · strength 0.60
- Paper HMGA2 Expression in Pancreatic Ductal Adenocarcinoma Controls Tumor Microenvironment and Immunosuppression via IL1α DOI 10.64898/2026.10.02.754697↗
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Kristopher S. Raghavan · 3 shared papers, 2021–2024 · strength 0.47
- Paper Pulsed low-dose-rate radiation (PLDR) reduces the tumor-promoting responses induced by conventional chemoradiation in p… DOI 10.1101/2024.01.13.575510↗
- Paper NetrinG1+ cancer-associated fibroblasts generate unique extracellular vesicles that support the survival of pancreatic … DOI 10.1158/2767-9764.crc-21-0147↗
- Paper Palladin isoforms 3 and 4 regulate cancer-associated fibroblast pro-tumor functions in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-021-82937-3↗
- Daniel James Salas-Escabillas · 2 shared papers, 2025–2026 · strength 0.38
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Ling Huang · 2 shared papers, 2025–2026 · strength 0.38
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- David Kwon · 2 shared papers, 2025–2026 · strength 0.38
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Yuesong Wu · 2 shared papers, 2025–2026 · strength 0.38
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Kailee M. Hartway · 2 shared papers, 2025–2026 · strength 0.38
- Paper Spatial Transcriptomics on a Diverse Pancreatic Cancer Cohort Reveals Race-Related Differences in Tumor Subtypes and Im… DOI 10.1158/0008-5472.can-25-2748↗
- Paper Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. DOI 10.1158/1078-0432.ccr-24-2183↗
- Igor A. Astsaturov · 2 shared papers, 2023–2026 · strength 0.32
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Netrin-1 feedforward mechanism promotes pancreatic cancer liver metastasis via hepatic stellate cell activation, retino… DOI 10.1016/j.celrep.2023.113369↗
- Eileen S. Carpenter · 1 shared paper, 2024 · strength 0.23
- Marie C. Hasselluhn · 1 shared paper, 2024 · strength 0.23
- Marina Pasca di Magliano · 1 shared paper, 2024 · strength 0.23
- Mara H. Sherman · 1 shared paper, 2024 · strength 0.23
- Anirban Maitra · 1 shared paper, 2024 · strength 0.23
- Kenneth P. Olive · 1 shared paper, 2024 · strength 0.23
- Naomi Yamamoto · 1 shared paper, 2026 · strength 0.22
- Stephanie Dobersch · 1 shared paper, 2026 · strength 0.22
- Joana E. Aggrey-Fynn · 1 shared paper, 2026 · strength 0.22
- Sita Kugel · 1 shared paper, 2026 · strength 0.22
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.