ResearcherColumbia UniversityActive in the last 2 years
Ryota Takahashi
Matched on ORCID iD 0000-0001-7886-8481; OpenAlex author ID A5100724725. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 45
2013: 1 paper · 2014: none · 2015: none · 2016: 1 paper · 2017: none · 2018: 1 paper · 2019: 1 paper · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper · 2023: none · 2024: none · 2025: 1 paper
Papers 9
- WWP1 inhibition suppresses the proliferation of pancreatic cancer cells by regulating the PI3K-AKT pathway. Co-author DOI 10.1007/s00535-024-02192-x↗
- The Role of Neural Signaling in the Pancreatic Cancer Microenvironment. Co-author DOI 10.3390/cancers14174269↗
- Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppression. Co-author DOI 10.1136/gutjnl-2019-319912↗
- Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancreatic cancer. Co-author DOI 10.1038/s41598-020-78320-3↗
- A DNA Hypomethylating Drug Alters the Tumor Microenvironment and Improves the Effectiveness of Immune Checkpoint Inhibitors in a Mouse Model of Pancreatic Canc… Co-author DOI 10.1158/0008-5472.can-20-0285↗
- Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal adenocarcinoma through reduced cell invas… Co-author DOI 10.1038/s41389-018-0117-8↗
- β2 Adrenergic-Neurotrophin Feedforward Loop Promotes Pancreatic Cancer. First author DOI 10.1016/j.ccell.2017.11.007↗
- Stromal remodeling by the BET bromodomain inhibitor JQ1 suppresses the progression of human pancreatic cancer. Co-author DOI 10.18632/oncotarget.11129↗
- Therapeutic effect of c-Jun N-terminal kinase inhibition on pancreatic cancer. Co-author DOI 10.1111/cas.12080↗
Closest collaborators
The 25 strongest of 45 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hideaki Ijichi · 6 shared papers, 2013–2025 · strength 0.74
- Paper The Role of Neural Signaling in the Pancreatic Cancer Microenvironment. DOI 10.3390/cancers14174269↗
- Paper WWP1 inhibition suppresses the proliferation of pancreatic cancer cells by regulating the PI3K-AKT pathway. DOI 10.1007/s00535-024-02192-x↗
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Mitsuhiro Fujishiro · 2 shared papers, 2022–2025 · strength 0.50
- Koji Miyabayashi · 4 shared papers, 2016–2025 · strength 0.45
- Paper WWP1 inhibition suppresses the proliferation of pancreatic cancer cells by regulating the PI3K-AKT pathway. DOI 10.1007/s00535-024-02192-x↗
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal a… DOI 10.1038/s41389-018-0117-8↗
- Keisuke Yamamoto · 3 shared papers, 2016–2025 · strength 0.37
- Paper WWP1 inhibition suppresses the proliferation of pancreatic cancer cells by regulating the PI3K-AKT pathway. DOI 10.1007/s00535-024-02192-x↗
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Stromal remodeling by the BET bromodomain inhibitor JQ1 suppresses the progression of human pancreatic cancer. DOI 10.18632/oncotarget.11129↗
- Kenneth P. Olive · 3 shared papers, 2018–2021 · strength 0.26
- Paper Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppress… DOI 10.1136/gutjnl-2019-319912↗
- Paper A DNA Hypomethylating Drug Alters the Tumor Microenvironment and Improves the Effectiveness of Immune Checkpoint Inhibi… DOI 10.1158/0008-5472.can-20-0285↗
- Paper β2 Adrenergic-Neurotrophin Feedforward Loop Promotes Pancreatic Cancer. DOI 10.1016/j.ccell.2017.11.007↗
- Timothy Cragin Wang · 3 shared papers, 2018–2021 · strength 0.26
- Paper Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppress… DOI 10.1136/gutjnl-2019-319912↗
- Paper A DNA Hypomethylating Drug Alters the Tumor Microenvironment and Improves the Effectiveness of Immune Checkpoint Inhibi… DOI 10.1158/0008-5472.can-20-0285↗
- Paper β2 Adrenergic-Neurotrophin Feedforward Loop Promotes Pancreatic Cancer. DOI 10.1016/j.ccell.2017.11.007↗
- Kazuhiko Koike · 4 shared papers, 2013–2020 · strength 0.24
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal a… DOI 10.1038/s41389-018-0117-8↗
- Paper Stromal remodeling by the BET bromodomain inhibitor JQ1 suppresses the progression of human pancreatic cancer. DOI 10.18632/oncotarget.11129↗
- Takuma Iwata · 1 shared paper, 2025 · strength 0.24
- Takahiro Kishikawa · 1 shared paper, 2025 · strength 0.24
- Takahiro Seimiya · 1 shared paper, 2025 · strength 0.24
- Genso Notoya · 1 shared paper, 2025 · strength 0.24
- Motoyuki Otsuka · 1 shared paper, 2025 · strength 0.24
- Hiroyuki Isayama · 3 shared papers, 2016–2020 · strength 0.21
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal a… DOI 10.1038/s41389-018-0117-8↗
- Paper Stromal remodeling by the BET bromodomain inhibitor JQ1 suppresses the progression of human pancreatic cancer. DOI 10.18632/oncotarget.11129↗
- Tamas A. Gonda · 2 shared papers, 2020–2021 · strength 0.21
- Paper Interleukin-1β-induced pancreatitis promotes pancreatic ductal adenocarcinoma via B lymphocyte-mediated immune suppress… DOI 10.1136/gutjnl-2019-319912↗
- Paper A DNA Hypomethylating Drug Alters the Tumor Microenvironment and Improves the Effectiveness of Immune Checkpoint Inhibi… DOI 10.1158/0008-5472.can-20-0285↗
- Harold L. Moses · 3 shared papers, 2013–2020 · strength 0.20
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal a… DOI 10.1038/s41389-018-0117-8↗
- Paper Therapeutic effect of c-Jun N-terminal kinase inhibition on pancreatic cancer. DOI 10.1111/cas.12080↗
- Makoto Sano · 2 shared papers, 2019–2020 · strength 0.17
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Blocking CXCLs-CXCR2 axis in tumor-stromal interactions contributes to survival in a mouse model of pancreatic ductal a… DOI 10.1038/s41389-018-0117-8↗
- Alina Cornelia Iuga · 2 shared papers, 2018–2021 · strength 0.16
- Takayuki Tanaka · 2 shared papers, 2018–2021 · strength 0.16
- Marina Macchini · 2 shared papers, 2018–2021 · strength 0.16
- Bernhard Willibald Renz · 2 shared papers, 2018–2021 · strength 0.16
- Christoph Benedikt Westphalen · 2 shared papers, 2018–2021 · strength 0.16
- Yousuke Nakai · 2 shared papers, 2016–2020 · strength 0.13
- Paper Soluble VCAM-1 promotes gemcitabine resistance via macrophage infiltration and predicts therapeutic response in pancrea… DOI 10.1038/s41598-020-78320-3↗
- Paper Stromal remodeling by the BET bromodomain inhibitor JQ1 suppresses the progression of human pancreatic cancer. DOI 10.18632/oncotarget.11129↗
- Ruth Aroon White · 1 shared paper, 2021 · strength 0.11
- Masaki Sunagawa · 1 shared paper, 2021 · strength 0.11
- Jeanine M. Genkinger · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.