ResearcherChinese Academy of Medical Sciences & Peking Union Medical CollegeNo recent work listed
Xianglan Jin
Matched on ORCID iD 0000-0001-9934-3641; OpenAlex author ID A5064594549. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 9
2013: 1 paper · 2014: none · 2015: 3 papers · 2016: 1 paper
Papers 5
- KRAS-related noncoding RNAs in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.cdtm.2016.11.012↗
- MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. Co-author DOI 10.1371/journal.pone.0119783↗
- Deregulation of the MiR-193b-KRAS Axis Contributes to Impaired Cell Growth in Pancreatic Cancer. Co-author DOI 10.1371/journal.pone.0125515↗
- Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. Co-author DOI 10.1371/journal.pone.0128257↗
- [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. First author DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
Closest collaborators
The 9 strongest of 9 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shuangni Yu · 4 shared papers, 2015–2016 · strength 0.23
- Paper KRAS-related noncoding RNAs in pancreatic ductal adenocarcinoma. DOI 10.1016/j.cdtm.2016.11.012↗
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Yang Sun · 4 shared papers, 2013–2015 · strength 0.21
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper Deregulation of the MiR-193b-KRAS Axis Contributes to Impaired Cell Growth in Pancreatic Cancer. DOI 10.1371/journal.pone.0125515↗
- Congwei Jia · 3 shared papers, 2013–2015 · strength 0.16
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
- Jie Chen · 3 shared papers, 2013–2015 · strength 0.16
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
- Tingting Zhang · 3 shared papers, 2013–2015 · strength 0.16
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
- Jie Chen · 2 shared papers, 2015–2016 · strength 0.12
- Cuiping Wang · 2 shared papers, 2015 · strength 0.11
- Pingping Liu · 1 shared paper, 2016 · strength 0.07
- Yihui Ma · 1 shared paper, 2016 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.