ResearcherQingdao UniversityNo recent work listed
Yang Sun
Matched on ORCID iD 0000-0002-7505-0534; OpenAlex author ID A5101646091. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 3, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 20
2013: 2 papers · 2014: 1 paper · 2015: 4 papers · 2016: none · 2017: 1 paper · 2018: none · 2019: none · 2020: none · 2021: none · 2022: 1 paper
Papers 9
- Protein tyrosine phosphatase 1 protects human pancreatic cancer from erastin-induced ferroptosis. Co-author DOI 10.1016/j.asjsur.2021.11.048↗
- RUNX1 promote invasiveness in pancreatic ductal adenocarcinoma through regulating miR-93. Co-author DOI 10.18632/oncotarget.20433↗
- Elevated miR-483-3p expression is an early event and indicates poor prognosis in pancreatic ductal adenocarcinoma. First author DOI 10.1007/s13277-015-3690-x↗
- MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. Co-author DOI 10.1371/journal.pone.0119783↗
- Deregulation of the MiR-193b-KRAS Axis Contributes to Impaired Cell Growth in Pancreatic Cancer. First author DOI 10.1371/journal.pone.0125515↗
- Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. Co-author DOI 10.1371/journal.pone.0128257↗
- Retinoic acid inhibits pancreatic cancer cell migration and EMT through the downregulation of IL-6 in cancer associated fibroblast cells. Co-author DOI 10.1016/j.canlet.2013.12.006↗
- [Target prediction and verification of miR-27a in pancreatic cancer]. First author DOI 10.3760/cma.j.issn.0529-5807.2013.06.008↗
- [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. Co-author DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
Closest collaborators
The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shuangni Yu · 6 shared papers, 2013–2017 · strength 0.33
- Paper RUNX1 promote invasiveness in pancreatic ductal adenocarcinoma through regulating miR-93. DOI 10.18632/oncotarget.20433↗
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Jie Chen · 5 shared papers, 2013–2017 · strength 0.28
- Paper RUNX1 promote invasiveness in pancreatic ductal adenocarcinoma through regulating miR-93. DOI 10.18632/oncotarget.20433↗
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Congwei Jia · 5 shared papers, 2013–2015 · strength 0.24
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
- Xianglan Jin · 4 shared papers, 2013–2015 · strength 0.21
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper Deregulation of the MiR-193b-KRAS Axis Contributes to Impaired Cell Growth in Pancreatic Cancer. DOI 10.1371/journal.pone.0125515↗
- Tingting Zhang · 4 shared papers, 2013–2015 · strength 0.20
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper [MiR-150-5p inhibits the proliferation and promoted apoptosis of pancreatic cancer cells]. DOI 10.3760/cma.j.issn.0529-5807.2013.07.007↗
- Xiao-Dong Huang · 1 shared paper, 2022 · strength 0.19
- Fengjun Xiao · 1 shared paper, 2022 · strength 0.19
- Xian-Jie Shi · 1 shared paper, 2022 · strength 0.19
- Cuiping Wang · 3 shared papers, 2015 · strength 0.16
- Paper MiRNA-615-5p functions as a tumor suppressor in pancreatic ductal adenocarcinoma by targeting AKT2. DOI 10.1371/journal.pone.0119783↗
- Paper Correction: MiRNA-615-5p Functions as a Tumor Suppressor in Pancreatic Ductal Adenocarcinoma by Targeting AKT2. DOI 10.1371/journal.pone.0128257↗
- Paper Elevated miR-483-3p expression is an early event and indicates poor prognosis in pancreatic ductal adenocarcinoma. DOI 10.1007/s13277-015-3690-x↗
- Yin Cheng · 2 shared papers, 2014–2017 · strength 0.12
- Jie Chen · 2 shared papers, 2014–2015 · strength 0.09
- Paper Elevated miR-483-3p expression is an early event and indicates poor prognosis in pancreatic ductal adenocarcinoma. DOI 10.1007/s13277-015-3690-x↗
- Paper Retinoic acid inhibits pancreatic cancer cell migration and EMT through the downregulation of IL-6 in cancer associated… DOI 10.1016/j.canlet.2013.12.006↗
- Jie Chen · 1 shared paper, 2015 · strength 0.05
- Pingping Liu · 1 shared paper, 2015 · strength 0.05
- Huanwen Wu · 1 shared paper, 2015 · strength 0.05
- Yunxiao Meng · 1 shared paper, 2015 · strength 0.05
- Mingyang Liu · 1 shared paper, 2015 · strength 0.05
- Li Zhang · 1 shared paper, 2015 · strength 0.05
- Hui Zhang · 1 shared paper, 2014 · strength 0.04
- Yumei Gu · 1 shared paper, 2014 · strength 0.04
- Jian Guan · 1 shared paper, 2014 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.