ResearcherUniversity of Nebraska Medical CenterNo recent work listed
Srustidhar Das
Matched on ORCID iD 0000-0002-0833-588X; OpenAlex author ID A5068629595. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 23
2011: 1 paper · 2012: none · 2013: none · 2014: none · 2015: 4 papers
Papers 5
- NCOA3-mediated upregulation of mucin expression via transcriptional and post-translational changes during the development of pancreatic cancer. First author DOI 10.1038/onc.2014.409↗
- Amyloid precursor-like protein 2 (APLP2) affects the actin cytoskeleton and increases pancreatic cancer growth and metastasis. Co-author DOI 10.18632/oncotarget.2990↗
- Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.3308↗
- Pancreatic cancer metastasis: are we being pre-EMTed? Co-author DOI 10.2174/1381612821666141211115234↗
- Pathobiological implications of MUC16 expression in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0026839↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Surinder K. Batra · 5 shared papers, 2011–2015 · strength 0.27
- Paper Pancreatic cancer metastasis: are we being pre-EMTed? DOI 10.2174/1381612821666141211115234↗
- Paper Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to… DOI 10.18632/oncotarget.3308↗
- Paper NCOA3-mediated upregulation of mucin expression via transcriptional and post-translational changes during the developme… DOI 10.1038/onc.2014.409↗
- Satyanarayana Rachagani · 4 shared papers, 2011–2015 · strength 0.16
- Paper Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to… DOI 10.18632/oncotarget.3308↗
- Paper NCOA3-mediated upregulation of mucin expression via transcriptional and post-translational changes during the developme… DOI 10.1038/onc.2014.409↗
- Paper Amyloid precursor-like protein 2 (APLP2) affects the actin cytoskeleton and increases pancreatic cancer growth and meta… DOI 10.18632/oncotarget.2990↗
- Sukhwinder Kaur · 2 shared papers, 2015 · strength 0.09
- Paper NCOA3-mediated upregulation of mucin expression via transcriptional and post-translational changes during the developme… DOI 10.1038/onc.2014.409↗
- Paper Amyloid precursor-like protein 2 (APLP2) affects the actin cytoskeleton and increases pancreatic cancer growth and meta… DOI 10.18632/oncotarget.2990↗
- Imayavaramban Lakshmanan · 2 shared papers, 2011–2015 · strength 0.08
- Moorthy Palanimuthu Ponnusamy · 2 shared papers, 2011–2015 · strength 0.07
- Sushil Kumar K · 2 shared papers, 2011–2015 · strength 0.07
- Dhanya Haridas · 2 shared papers, 2011–2015 · strength 0.06
- Michael Anthony Hollingsworth · 2 shared papers, 2011–2015 · strength 0.06
- María P. Torres · 1 shared paper, 2015 · strength 0.05
- Lynette M. Smith · 1 shared paper, 2015 · strength 0.05
- Kay‐Uwe Wagner · 1 shared paper, 2015 · strength 0.05
- Maneesh Jain · 1 shared paper, 2015 · strength 0.05
- S Joshi · 1 shared paper, 2015 · strength 0.05
- Richard G. MacDonald · 1 shared paper, 2015 · strength 0.04
- Jane L. Meza · 1 shared paper, 2015 · strength 0.04
- Poomy Pandey · 1 shared paper, 2015 · strength 0.04
- Haley L. Peters · 1 shared paper, 2015 · strength 0.04
- Parthasarathy Seshacharyulu · 1 shared paper, 2015 · strength 0.04
- Prakash Radhakrishnan · 1 shared paper, 2015 · strength 0.04
- Joyce C. Solheim · 1 shared paper, 2015 · strength 0.04
- Subodh M. Lele · 1 shared paper, 2011 · strength 0.02
- Subhankar Chakraborty · 1 shared paper, 2011 · strength 0.02
- Uwe A. Wittel · 1 shared paper, 2011 · strength 0.02
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.