ResearcherNebraska Medical CenterActive in the last 2 years
Kay‐Uwe Wagner
Matched on ORCID iD 0000-0002-5081-0226; OpenAlex author ID A5054645034; NIH RePORTER profile ID 6798938. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 0, last author 4)
- Trials
- 0
- Grants
- 2
- Labs led
- 2
- Collaborators
- 41
2012: 1 paper, 1 grant · 2013: 1 paper · 2014: 1 paper · 2015: 2 papers · 2016: none · 2017: none · 2018: none · 2019: none · 2020: none · 2021: 2 papers, 1 grant · 2022: none · 2023: 1 paper · 2024: 2 papers
Papers 10
- RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. Co-author DOI 10.1016/j.canlet.2024.217275↗
- The Janus kinase 1 is critical for pancreatic cancer initiation and progression. Last author DOI 10.1016/j.celrep.2024.114202↗
- Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor progression via the c-Myc-RCC1-Ran axis Co-author DOI 10.1101/2023.12.18.572102↗
- Models of pancreatic ductal adenocarcinoma. Last author DOI 10.1007/s10555-021-09989-9↗
- Exosomal microRNA in Pancreatic Cancer Diagnosis, Prognosis, and Treatment: From Bench to Bedside. Co-author DOI 10.3390/cancers13112777↗
- Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets. Co-author DOI 10.1038/ncomms7744↗
- Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.3308↗
- Cancer cell dormancy in novel mouse models for reversible pancreatic cancer: a lingering challenge in the development of targeted therapies. Last author DOI 10.1158/0008-5472.can-13-3437↗
- Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. Last author DOI 10.1158/0008-5472.can-12-2067↗
- Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diagnosis and therapy. Co-author DOI 10.1186/1756-8722-5-68↗
Grants 2
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Md. Hafiz Uddin · 3 shared papers, 2021–2024 · strength 0.48
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Paper Exosomal microRNA in Pancreatic Cancer Diagnosis, Prognosis, and Treatment: From Bench to Bedside. DOI 10.3390/cancers13112777↗
- Asfar Sohail Azmi · 3 shared papers, 2021–2024 · strength 0.48
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Paper Exosomal microRNA in Pancreatic Cancer Diagnosis, Prognosis, and Treatment: From Bench to Bedside. DOI 10.3390/cancers13112777↗
- Hridaya Shrestha · 2 shared papers, 2021–2024 · strength 0.41
- Rayane Dennaoui · 2 shared papers, 2021–2024 · strength 0.41
- Sahar F. Bannoura · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Boris Pasche · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Husain Yar Khan · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Eliza Wright Beal · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Amro Aboukameel · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Ramzi M. Mohammad · 2 shared papers, 2021–2024 · strength 0.32
- Philip A. Philip · 2 shared papers, 2021–2024 · strength 0.32
- Nirakar Rajbhandari · 3 shared papers, 2013–2024 · strength 0.28
- Paper The Janus kinase 1 is critical for pancreatic cancer initiation and progression. DOI 10.1016/j.celrep.2024.114202↗
- Paper Cancer cell dormancy in novel mouse models for reversible pancreatic cancer: a lingering challenge in the development o… DOI 10.1158/0008-5472.can-13-3437↗
- Paper Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. DOI 10.1158/0008-5472.can-12-2067↗
- Dieter Saur · 1 shared paper, 2024 · strength 0.18
- Muhammad Wasif Saif · 1 shared paper, 2024 · strength 0.17
- Bassel F. El‐Rayes · 1 shared paper, 2024 · strength 0.17
- Wan-Chi Lin · 3 shared papers, 2013–2015 · strength 0.14
- Paper Cancer cell dormancy in novel mouse models for reversible pancreatic cancer: a lingering challenge in the development o… DOI 10.1158/0008-5472.can-13-3437↗
- Paper Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets. DOI 10.1038/ncomms7744↗
- Paper Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. DOI 10.1158/0008-5472.can-12-2067↗
- Surinder K. Batra · 3 shared papers, 2012–2015 · strength 0.11
- Paper Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to… DOI 10.18632/oncotarget.3308↗
- Paper Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. DOI 10.1158/0008-5472.can-12-2067↗
- Paper Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diag… DOI 10.1186/1756-8722-5-68↗
- María P. Torres · 2 shared papers, 2012–2015 · strength 0.08
- Paper Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to… DOI 10.18632/oncotarget.3308↗
- Paper Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diag… DOI 10.1186/1756-8722-5-68↗
- Satyanarayana Rachagani · 2 shared papers, 2012–2015 · strength 0.08
- Paper Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to… DOI 10.18632/oncotarget.3308↗
- Paper Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diag… DOI 10.1186/1756-8722-5-68↗
- Agnieszka Katarzyna Witkiewicz · 2 shared papers, 2013–2015 · strength 0.07
- Srustidhar Das · 1 shared paper, 2015 · strength 0.05
- Lynette M. Smith · 1 shared paper, 2015 · strength 0.05
- Imayavaramban Lakshmanan · 1 shared paper, 2015 · strength 0.05
- GuemHee Baek · 1 shared paper, 2015 · strength 0.04
- Adam Charles Yopp · 1 shared paper, 2015 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.