ResearcherUniversity of MichiganActive in the last 2 years
Amro Aboukameel
Matched on ORCID iD 0009-0009-9912-2887; OpenAlex author ID A5038465132. Sources: OpenAlex.
Their work
- Papers
- 28 (first author 4, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 40
2011: 1 paper · 2012: 2 papers · 2013: 2 papers · 2014: 5 papers · 2015: none · 2016: none · 2017: 2 papers · 2018: 1 paper · 2019: 2 papers · 2020: 2 papers · 2021: none · 2022: 3 papers · 2023: 3 papers · 2024: 1 paper · 2025: 3 papers · 2026: 1 paper
Papers 28
- XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2026.218804↗
- Pharmacological advances in cannabinoid-based therapies for pancreatic cancer: preclinical efficacy of CCL-106 and its epimers. Co-author DOI 10.1016/j.esmogo.2025.100211↗
- Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2025.11.21.689857↗
- PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer Co-author DOI 10.64898/2025.12.06.692635↗
- RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. First author DOI 10.1016/j.canlet.2024.217275↗
- Molecular analysis of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in KPC pancreatic cancer mouse model. Co-author DOI 10.1002/ctm2.1513↗
- Targeting Cellular Metabolism With CPI-613 Sensitizes Pancreatic Cancer Cells to Radiation Therapy. Co-author DOI 10.1016/j.adro.2022.101122↗
- Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor progression via the c-Myc-RCC1-Ran axis First author DOI 10.1101/2023.12.18.572102↗
- Connecting the Human Microbiome and Pancreatic Cancer. Co-author DOI 10.1007/s10555-022-10022-w↗
- Correction to: Connecting the human microbiome and pancreatic cancer. Co-author DOI 10.1007/s10555-022-10037-3↗
- Molecular profiling of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in cellular and LSL-Kras G12D/+; Trp53 fl/+; Pdx1-Cre (KPC) pancreatic cancer … First author DOI 10.1101/2022.03.08.481108↗
- Preclinical Assessment with Clinical Validation of Selinexor with Gemcitabine and Nab-Paclitaxel for the Treatment of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-19-1728↗
- Calcium Release-Activated Calcium (CRAC) Channel Inhibition Suppresses Pancreatic Ductal Adenocarcinoma Cell Proliferation and Patient-Derived Tumor Growth. Co-author DOI 10.3390/cancers12030750↗
- Targeting Rho GTPase effector p21 activated kinase 4 (PAK4) suppresses p-Bad-microRNA drug resistance axis leading to inhibition of pancreatic ductal adenocarc… Co-author DOI 10.1080/21541248.2017.1329694↗
- DNA-Methylation-Caused Downregulation of miR-30 Contributes to the High Expression of XPO1 and the Aggressive Growth of Tumors in Pancreatic Ductal Adenocarcin… Co-author DOI 10.3390/cancers11081101↗
- Retraction notice to "Increased Ras GTPase activity is regulated by miRNAs that can be attenuated by CDF treatment in pancreatic cancer cells" [Cancer Lett. 31… Co-author DOI 10.1016/j.canlet.2017.11.030↗
- Novel p21-Activated Kinase 4 (PAK4) Allosteric Modulators Overcome Drug Resistance and Stemness in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1535-7163.mct-16-0205↗
- Exportin 1 (XPO1) inhibition leads to restoration of tumor suppressor miR-145 and consequent suppression of pancreatic cancer cell proliferation and migration. Co-author DOI 10.18632/oncotarget.19285↗
- Deregulation of miR-146a expression in a mouse model of pancreatic cancer affecting EGFR signaling. Co-author DOI 10.1016/j.canlet.2014.05.013↗
- Pancreatic cancer stem-like cells display aggressive behavior mediated via activation of FoxQ1. Co-author DOI 10.1074/jbc.m113.532887↗
- Differentially expressed miRNAs in cancer-stem-like cells: markers for tumor cell aggressiveness of pancreatic cancer. Co-author DOI 10.1089/scd.2013.0551↗
- A novel small-molecule inhibitor of mcl-1 blocks pancreatic cancer growth in vitro and in vivo. Co-author DOI 10.1158/1535-7163.mct-12-0767↗
- Nuclear retention of Fbw7 by specific inhibitors of nuclear export leads to Notch1 degradation in pancreatic cancer. Co-author DOI 10.18632/oncotarget.1813↗
- Synthesis, characterization and anti-tumor activity of novel thymoquinone analogs against pancreatic cancer. Co-author DOI 10.1016/j.bmcl.2013.03.003↗
- Selective inhibitors of nuclear export block pancreatic cancer cell proliferation and reduce tumor growth in mice. First author DOI 10.1053/j.gastro.2012.10.036↗
- RETRACTED: Increased Ras GTPase activity is regulated by miRNAs that can be attenuated by CDF treatment in pancreatic cancer cells. Co-author DOI 10.1016/j.canlet.2012.01.013↗
- Hypoxia-induced aggressiveness of pancreatic cancer cells is due to increased expression of VEGF, IL-6 and miR-21, which can be attenuated by CDF treatment. Co-author DOI 10.1371/journal.pone.0050165↗
- Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0017850↗
Closest collaborators
The 25 strongest of 40 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Asfar Sohail Azmi · 23 shared papers, 2012–2026 · strength 2.57
- Paper Pharmacological advances in cannabinoid-based therapies for pancreatic cancer: preclinical efficacy of CCL-106 and its … DOI 10.1016/j.esmogo.2025.100211↗
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Husain Yar Khan · 13 shared papers, 2020–2026 · strength 2.00
- Paper Pharmacological advances in cannabinoid-based therapies for pancreatic cancer: preclinical efficacy of CCL-106 and its … DOI 10.1016/j.esmogo.2025.100211↗
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Sahar F. Bannoura · 10 shared papers, 2022–2026 · strength 1.59
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Philip A. Philip · 15 shared papers, 2011–2025 · strength 1.42
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Targeting Cellular Metabolism With CPI-613 Sensitizes Pancreatic Cancer Cells to Radiation Therapy. DOI 10.1016/j.adro.2022.101122↗
- Md. Hafiz Uddin · 9 shared papers, 2022–2026 · strength 1.42
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Ramzi M. Mohammad · 14 shared papers, 2013–2026 · strength 1.41
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Mohammed Najeeb Al Hallak · 8 shared papers, 2020–2026 · strength 1.23
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Yiwei Li · 10 shared papers, 2012–2023 · strength 1.05
- Paper Targeting Cellular Metabolism With CPI-613 Sensitizes Pancreatic Cancer Cells to Radiation Therapy. DOI 10.1016/j.adro.2022.101122↗
- Paper Molecular analysis of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in KPC pancreatic cancer mouse model. DOI 10.1002/ctm2.1513↗
- Paper Connecting the Human Microbiome and Pancreatic Cancer. DOI 10.1007/s10555-022-10022-w↗
- Bassel F. El‐Rayes · 6 shared papers, 2022–2026 · strength 0.99
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Bin Bao · 11 shared papers, 2011–2026 · strength 0.89
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Boris Pasche · 5 shared papers, 2023–2026 · strength 0.88
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Muhammad Wasif Saif · 4 shared papers, 2024–2026 · strength 0.72
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper PCNA Inhibition Enhances the Antitumor Activity of KRAS-Targeted Therapies in Pancreatic Cancer DOI 10.64898/2025.12.06.692635↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Eliza Wright Beal · 4 shared papers, 2023–2026 · strength 0.70
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Anthony Frank Shields · 4 shared papers, 2022–2026 · strength 0.64
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Paper Molecular analysis of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in KPC pancreatic cancer mouse model. DOI 10.1002/ctm2.1513↗
- Steve H. Kim · 5 shared papers, 2020–2025 · strength 0.63
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Paper Molecular analysis of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in KPC pancreatic cancer mouse model. DOI 10.1002/ctm2.1513↗
- Paper Molecular profiling of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in cellular and LSL-Kras G12D/+; Trp53… DOI 10.1101/2022.03.08.481108↗
- Rachel E. Sexton · 4 shared papers, 2020–2022 · strength 0.50
- Paper Connecting the Human Microbiome and Pancreatic Cancer. DOI 10.1007/s10555-022-10022-w↗
- Paper Correction to: Connecting the human microbiome and pancreatic cancer. DOI 10.1007/s10555-022-10037-3↗
- Paper Molecular profiling of XPO1 inhibitor and gemcitabine-nab-paclitaxel combination in cellular and LSL-Kras G12D/+; Trp53… DOI 10.1101/2022.03.08.481108↗
- Irfana Muqbil · 5 shared papers, 2017–2020 · strength 0.44
- Paper DNA-Methylation-Caused Downregulation of miR-30 Contributes to the High Expression of XPO1 and the Aggressive Growth of… DOI 10.3390/cancers11081101↗
- Paper Targeting Rho GTPase effector p21 activated kinase 4 (PAK4) suppresses p-Bad-microRNA drug resistance axis leading to i… DOI 10.1080/21541248.2017.1329694↗
- Paper Preclinical Assessment with Clinical Validation of Selinexor with Gemcitabine and Nab-Paclitaxel for the Treatment of P… DOI 10.1158/1078-0432.ccr-19-1728↗
- Fazlul H. Sarkar · 9 shared papers, 2011–2018 · strength 0.38
- Paper Retraction notice to "Increased Ras GTPase activity is regulated by miRNAs that can be attenuated by CDF treatment in p… DOI 10.1016/j.canlet.2017.11.030↗
- Paper Deregulation of miR-146a expression in a mouse model of pancreatic cancer affecting EGFR signaling. DOI 10.1016/j.canlet.2014.05.013↗
- Paper Differentially expressed miRNAs in cancer-stem-like cells: markers for tumor cell aggressiveness of pancreatic cancer. DOI 10.1089/scd.2013.0551↗
- P. A. Philip · 2 shared papers, 2025–2026 · strength 0.37
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Azeddine Atfi · 2 shared papers, 2025–2026 · strength 0.37
- Paper XPO1 inhibition enhances the efficacy and durability of RAS-targeted therapy in preclinical models of KRASG12D mutant p… DOI 10.1016/j.canlet.2026.218804↗
- Paper Co-targeting KRAS and Exportin1 as an effective therapeutic strategy for KRASG12D mutant pancreatic ductal adenocarcino… DOI 10.1101/2025.11.21.689857↗
- Mandana Kamgar · 3 shared papers, 2020–2023 · strength 0.35
- Paper Targeting Cellular Metabolism With CPI-613 Sensitizes Pancreatic Cancer Cells to Radiation Therapy. DOI 10.1016/j.adro.2022.101122↗
- Paper Preclinical Assessment with Clinical Validation of Selinexor with Gemcitabine and Nab-Paclitaxel for the Treatment of P… DOI 10.1158/1078-0432.ccr-19-1728↗
- Paper Calcium Release-Activated Calcium (CRAC) Channel Inhibition Suppresses Pancreatic Ductal Adenocarcinoma Cell Proliferat… DOI 10.3390/cancers12030750↗
- Kay‐Uwe Wagner · 2 shared papers, 2023–2024 · strength 0.33
- Paper RCC1 regulation of subcellular protein localization via Ran GTPase drives pancreatic ductal adenocarcinoma growth. DOI 10.1016/j.canlet.2024.217275↗
- Paper Regulator of Chromosome Condensation (RCC1) a novel therapeutic target in pancreatic ductal adenocarcinoma drives tumor… DOI 10.1101/2023.12.18.572102↗
- Shadan S. Ali · 7 shared papers, 2011–2018 · strength 0.30
- Paper Retraction notice to "Increased Ras GTPase activity is regulated by miRNAs that can be attenuated by CDF treatment in p… DOI 10.1016/j.canlet.2017.11.030↗
- Paper Deregulation of miR-146a expression in a mouse model of pancreatic cancer affecting EGFR signaling. DOI 10.1016/j.canlet.2014.05.013↗
- Paper Differentially expressed miRNAs in cancer-stem-like cells: markers for tumor cell aggressiveness of pancreatic cancer. DOI 10.1089/scd.2013.0551↗
- Bayan A. Al-Share · 2 shared papers, 2022 · strength 0.27
- K.P. Ray · 1 shared paper, 2025 · strength 0.22
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.